S. Khare

发表

T-cell co-stimulation through B7RP-1 and ICOS

T. Mak, T. Horan, G. Duncan, 1999, Nature.

HLA-B27 and other predisposing factors in spondyloarthropathies.

H. Luthra, C. David, S. Khare, 1998, Current opinion in rheumatology.

HLA-B 27 Heavy Chains Contribute to Spontaneous Inflammatory Disease in B 27 / human b 2-Microglobulin ( b 2 m ) Double Transgenic Mice with Disrupted Mouse b 2 m

H. Luthra, C. David, S. Khare, 2013 .

Peptide binding α1α2 domain of HLA-B27 contributes to the disease pathogenesis in transgenic mice

H. Luthra, C. David, S. Khare, 1999 .

Spontaneous inflammatory disease in HLA-B27 transgenic mice is independent of MHC class II molecules: a direct role for B27 heavy chains and not B27-derived peptides.

H. Luthra, C. David, S. Khare, 1998, Journal of immunology.

Animal models of human leukocyte antigen B27-linked arthritides.

H. Luthra, C. David, S. Khare, 1998, Rheumatic diseases clinics of North America.

Not B 27-Derived Peptides Molecules : A Direct Role for B 27 Heavy Chains Transgenic Mice Is Independent of MHC Class II Spontaneous Inflammatory Disease in HLA-B 27 Luthra and

H. Luthra, C. David, S. Khare, 1997 .

HLA-B27 heavy chains contribute to spontaneous inflammatory disease in B27/human beta2-microglobulin (beta2m) double transgenic mice with disrupted mouse beta2m.

H. Luthra, C. David, S. Khare, 1996, The Journal of clinical investigation.

Role of HLA-B27 in spondyloarthropathies.

H. Luthra, C. David, S. Khare, 1996, Current topics in microbiology and immunology.

Pharmacodynamic effects of the murine p75-Fc fusion protein in mice.

T. Kohno, S. Khare, Helen Y. Kim, 2007, The journal of investigative dermatology. Symposium proceedings.

HLA-B27 transgenic mice are susceptible to collagen-induced arthritis: type II collagen as a potential target in human disease.

H. Luthra, C. David, S. Khare, 2000, Human immunology.

Oral administration of an immunodominant human collagen peptide modulates collagen-induced arthritis.

H. Luthra, C. David, C. Krco, 1995, Journal of immunology.

Spontaneous inflammatory disease in HLA-B27 transgenic mice does not require transporter of antigenic peptides.

H. Luthra, C. David, S. Khare, 2001, Clinical immunology.

A novel modality of BAFF-specific inhibitor AMG623 peptibody reduces B-cell number and improves outcomes in murine models of autoimmune disease.

T. Boone, N. Hawkins, M. Stolina, 2012, Clinical and experimental rheumatology.

Spontaneous inflammatory arthritis in HLA-B27 transgenic mice lacking beta 2-microglobulin: a model of human spondyloarthropathies

H. Luthra, C. David, S. Khare, 1995, The Journal of experimental medicine.

H2-A polymorphism contributes to H2-Eβ-mediated protection in collagen-induced arthritis

M. González-Gay, H. Luthra, C. David, 2007, Immunogenetics.

H2-A polymorphism contributes to H2-Eβ-mediated protection in collagen-induced arthritis

M. González-Gay, H. Luthra, E. Zanelli, 1996, Immunogenetics.

Human leukocyte antigen-DRB1*1502 (DR2Dw12) transgene reduces incidence and severity of arthritis in mice.

H. Inoko, M. González-Gay, H. Luthra, 1996, Human immunology.

Characterization of a new human B7-related protein: B7RP-1 is the ligand to the co-stimulatory protein ICOS.

T. Boone, T. Horan, J. Delaney, 2000, International immunology.

Taci Is a Traf-Interacting Receptor for Tall-1, a Tumor Necrosis Factor Family Member Involved in B Cell Regulation

J. Treanor, T. Boone, Gang Yu, 2000, The Journal of experimental medicine.

The role of TALL-1 and APRIL in immune regulation.

S. Khare, H. Hsu, 2001, Trends in immunology.

Severe B cell hyperplasia and autoimmune disease in TALL-1 transgenic mice.

D. Lacey, I. Sarosi, J. Delaney, 2000, Proceedings of the National Academy of Sciences of the United States of America.