M. Baas

发表

DNA amplification of HIV genome in hemophiliacs and in newborns from seropositive mothers

J. Cazenave, P. Lutz, B. Guy, 1991, Annals of Hematology.

Biosynthesis and intracellular post-translational processing of normal and mutant platelet glycoprotein GPIb-IX.

J. Salamero, S. Chasserot-Golaz, J. Cazenave, 2001, The Biochemical journal.

Biosynthesis and intracellular post-translational processing of normal and mutant platelet glycoprotein GPIb-IX

J. Salamero, S. Chasserot-Golaz, J. Cazenave, 2001 .

Synthesis of GPIbβ with novel transmembrane and cytoplasmic sequences in a Bernard–Soulier patient resulting in GPIb‐defective signaling in CHO cells

T. David, C. Gachet, C. Vinciguerra, 2006, Journal of thrombosis and haemostasis : JTH.

Biosynthesis of platelet glycoprotein V expressed as a single subunit or in association with GPIb-IX.

J. Cazenave, F. Lanza, M. Baas, 2004, European journal of biochemistry.

A three‐base deletion removing a leucine residue in a leucine‐rich repeat of platelet glycoprotein Ibα associated with a variant of Bernard‐Soulier syndrome (Nancy I)

C. Gachet, D. Hanau, M. Briquel, 1995, British journal of haematology.

A Leu7Pro mutation in the signal peptide of platelet glycoprotein (GP)IX in a case of Bernard–Soulier syndrome abolishes surface expression of the GPIb–V–IX complex

J. Cazenave, F. Lanza, M. Baas, 2002, British journal of haematology.

High prevalence of the natural Asn89Asp mutation in the GP1BB gene associated with Bernard–Soulier syndrome in French patients from the genetic isolate of Reunion Island

Renhao Li, C. Gachet, M. Jacquemont, 2020, British journal of haematology.

Detection by PCR and HphI restriction analysis of a splice site mutation at the 5' end of intron 15 of the platelet GPIIb (alpha IIb integrin) gene responsible for Glanzmann's thrombasthenia type I in Gypsies originating from the Strasbourg area.

J. Cazenave, M. Baas, F. Lanza, 1995, Thrombosis and haemostasis.

Common intragenic and extragenic polymorphisms of blood coagulation factors VIII and IX are different in Chinese and Caucasian populations

A. Hanauer, J. Cazenave, C. Ruan, 1990, Clinical genetics.

A novel platelet-type von Willebrand disease mutation (GP1BA p.Met255Ile) associated with type 2B “Malmö/New York” von Willebrand disease

Renhao Li, C. Gachet, É. Fressinaud, 2016, Thrombosis and Haemostasis.

Carrier detection in hemophilia using pedigree analysis coagulation tests and DNA probes.

J. Cazenave, A. Blanco, M. Baas, 1989, Nouvelle revue francaise d'hematologie.

Role of the intracellular domains of GPIb in controlling the adhesive properties of the platelet GPIb/V/IX complex.

J. Cazenave, S. Jackson, S. Cranmer, 2003, Blood.

A Novel Monoclonal Antibody against the Extracellular Domain of GPIbβ Modulates vWF Mediated Platelet Adhesion

M. Freund, E. Rubinstein, C. Ravanat, 2001, Thrombosis and Haemostasis.

Role of the Leucine-Rich Domain of Platelet GPIbα in Correct Post-translational Processing – The Nancy I Bernard-Soulier Mutation Expressed on CHO Cells

M. Briquel, S. Jackson, S. Cranmer, 2000, Thrombosis and Haemostasis.

A novel missense mutation in a leucine‐rich repeat of GPIbα in a Bernard‐Soulier variant reduces shear‐dependent adherence on von Willebrand factor

M. Bryckaert, P. Nurden, F. Adam, 2019, British journal of haematology.

A Deletion Located in the 3′ Non Translated Part of the Factor IX Gene Responsible for Mild Haemophilia B

M. Baas, M. Wiesel, L. Grunebaum, 1993, Thrombosis and Haemostasis.