Kasia Goleniewska

发表

Testosterone Attenuates Group 2 Innate Lymphoid Cell-Mediated Airway Inflammation.

K. Boyd, R. Peebles, Melissa H. Bloodworth, 2017, Cell reports.

Host-derived interleukin-5 promotes adenocarcinoma-induced malignant pleural effusion.

B. Fingleton, C. Roussos, R. Peebles, 2010, American journal of respiratory and critical care medicine.

Differential regulation of GM1 and asialo-GM1 expression by T cells and natural killer (NK) cells in respiratory syncytial virus infection.

R. Peebles, J. Durbin, M. Moore, 2008, Viral immunology.

Cutting Edge: Oseltamivir Decreases T Cell GM1 Expression and Inhibits Clearance of Respiratory Syncytial Virus: Potential Role of Endogenous Sialidase in Antiviral Immunity1

R. Peebles, J. Higginbotham, M. Moore, 2007, The Journal of Immunology.

Endogenous Sialidase in Antiviral Immunity Respiratory Syncytial Virus: Potential Role of GM1 Expression and Inhibits Clearance of Cutting Edge: Oseltamivir Decreases T Cell

J. Higginbotham, M. Moore, Weisong Zhou, 2007 .

Syncytial Virus Infection Il-4 Expression Induced by Respiratory Stat1 Negatively Regulates Lung Basophil Stat1 Negatively Regulates Lung Basophil Il-4 Expression Induced by Respiratory Syncytial Virus Infection Il-4 Contributes to Immunopathology Induced in Mice by Primary Respiratory Syncytial Vir

Vrajesh V. Parekh, L. Kaer, D. Mitchell, 2022 .

STAT1 Negatively Regulates Lung Basophil IL-4 Expression Induced by Respiratory Syncytial Virus Infection1

Vrajesh V. Parekh, D. Mitchell, J. Boyce, 2009, The Journal of Immunology.

STAT 6-Independent Allergic Lung CD 4 T Cell Activation , and Proinflammatory Chemokine Expression , Suppresses 2 Prostaglandin

Pingsheng Wu, M. Boothby, R. Peebles, 2016 .

Prostaglandin I2 Suppresses Proinflammatory Chemokine Expression, CD4 T Cell Activation, and STAT6-Independent Allergic Lung Inflammation

Pingsheng Wu, M. Boothby, R. Peebles, 2016, The Journal of Immunology.

Dietary supplementation of omega-3 fatty acid-containing fish oil suppresses F2-isoprostanes but enhances inflammatory cytokine response in a mouse model of ovalbumin-induced allergic lung inflammation.

J. Morrow, H. Yin, R. Peebles, 2009, Free radical biology & medicine.

PGI synthase overexpression protects against bleomycin-induced mortality and is associated with increased Nqo 1 expression.

J. Morrow, S. Biswal, Pingsheng Wu, 2011, American journal of physiology. Lung cellular and molecular physiology.

Signaling through the Prostaglandin I2 Receptor IP Protects against Respiratory Syncytial Virus-Induced Illness

J. Morrow, G. FitzGerald, B. Graham, 2004, Journal of Virology.

Cell Stimulatory Function of Dendritic Cells Proinflammatory Cytokine Production and T Prostaglandin I2 Analogs Inhibit

K. Hashimoto, M. Geraci, T. Blackwell, 2013 .

Prostaglandin I2 Analogs Inhibit Proinflammatory Cytokine Production and T Cell Stimulatory Function of Dendritic Cells1

G. FitzGerald, K. Hashimoto, R. Peebles, 2007, The Journal of Immunology.

Cell Stimulatory Function of Dendritic Cells Proinflammatory Cytokine Production and T Analogs Inhibit 2 Prostaglandin

G. FitzGerald, K. Hashimoto, R. Peebles, 2006 .

IL-17A inhibits airway reactivity induced by respiratory syncytial virus infection during allergic airway inflammation

J. Kolls, R. Peebles, D. Newcomb, 2013, Thorax.

Estrogen and progesterone decrease Let-7f miRNA expression and increase IL-23/IL-23R signaling and IL-17A production in severe asthma

Weisong, J. Fahrenholz, J. Kolls, 2015 .

Allergen-induced airway hyperresponsiveness mediated by cyclooxygenase inhibition is not dependent on 5-lipoxygenase or IL-5, but is IL-13 dependent.

J. Morrow, B. Graham, K. Hashimoto, 2006, Journal of immunology.

Allergen-Induced Airway Hyperresponsiveness Mediated by Cyclooxygenase Inhibition Is Not Dependent on 5-Lipoxygenase or IL-5, but Is IL-13 Dependent1

J. Morrow, B. Graham, K. Hashimoto, 2005, The Journal of Immunology.

Dependent on 5-Lipoxygenase or IL-5 , but Is IL-13 Cyclooxygenase Inhibition Is Not Dependent Hyperresponsiveness Mediated by Allergen-Induced Airway

J. Morrow, B. Graham, K. Hashimoto, 2005 .

Cyclooxygenase Inhibition Augments Allergic Inflammation through CD4-Dependent, STAT6-Independent Mechanisms1

J. Morrow, B. Graham, K. Hashimoto, 2005, The Journal of Immunology.

The PGI2 Analog Cicaprost Inhibits IL-33–Induced Th2 Responses, IL-2 Production, and CD25 Expression in Mouse CD4+ T Cells

R. Peebles, Melissa H. Bloodworth, D. Newcomb, 2018, The Journal of Immunology.

Endogenous PGI2 signaling through IP inhibits neutrophilic lung inflammation in LPS-induced acute lung injury mice model.

R. Peebles, D. Newcomb, W. Lawson, 2018, Prostaglandins & other lipid mediators.

Glucagon‐like peptide 1 signaling inhibits allergen‐induced lung IL‐33 release and reduces group 2 innate lymphoid cell cytokine production in vivo

A. Galli, K. Boyd, R. Peebles, 2018, The Journal of allergy and clinical immunology.

Cyclooxygenase inhibition abrogates aeroallergen-induced immune tolerance by suppressing prostaglandin I2 receptor signaling.

G. FitzGerald, Pingsheng Wu, G. Milne, 2014, The Journal of allergy and clinical immunology.

PGI2 signaling inhibits antigen uptake and increases migration of immature dendritic cells

G. FitzGerald, R. Peebles, D. Newcomb, 2013, Journal of leukocyte biology.

Cyclooxygenase Inhibition during Allergic Sensitization Increases STAT6-Independent Primary and Memory Th2 Responses1

R. Peebles, D. Newcomb, M. Moore, 2008, The Journal of Immunology.

Running Title: Allergic inflammation decreases lung Klebsiella burden

K. Boyd, J. Kolls, R. Peebles, 2014 .

STAT6-Independent Mechanisms Inflammation through CD4-Dependent, Cyclooxygenase Inhibition Augments Allergic

J. Morrow, K. Hashimoto, J. Sheller, 2013 .

IL-33 promotes the egress of group 2 innate lymphoid cells from the bone marrow

R. Peebles, Mark Rusznak, D. Newcomb, 2018, The Journal of experimental medicine.

STAT1 Represses Cytokine-Producing Group 2 and Group 3 Innate Lymphoid Cells during Viral Infection

K. Boyd, J. Kolls, R. Peebles, 2017, The Journal of Immunology.

Respiratory syncytial virus infection activates IL-13–producing group 2 innate lymphoid cells through thymic stromal lymphopoietin

K. Boyd, T. Hartert, R. Peebles, 2016, The Journal of allergy and clinical immunology.

Respiratory syncytial virus in allergic lung inflammation increases Muc5ac and gob-5.

B. Graham, K. Hashimoto, K. Adler, 2004, American journal of respiratory and critical care medicine.

IL-17A induces signal transducers and activators of transcription-6-independent airway mucous cell metaplasia.

S. Brody, K. Boyd, J. Kolls, 2013, American journal of respiratory cell and molecular biology.

Differential Pathogenesis of Respiratory Syncytial Virus Clinical Isolates in BALB/c Mice

R. Peebles, D. Newcomb, M. Moore, 2011, Journal of Virology.

A Chimeric A2 Strain of Respiratory Syncytial Virus (RSV) with the Fusion Protein of RSV Strain Line 19 Exhibits Enhanced Viral Load, Mucus, and Airway Dysfunction

J. Crowe, R. Peebles, D. Newcomb, 2009, Journal of Virology.

IL-13 is associated with reduced illness and replication in primary respiratory syncytial virus infection in the mouse

B. Graham, K. Hashimoto, J. Durbin, 2006, Microbes and Infection.

Natural Killer (NK) Cells, Not T Cells, Produce the Cytokine IL-4 in Response to Primary Respiratory Syncytial Virus Infection

L. Kaer, R. Peebles, J. Durbin, 2007 .

STAT6 Signaling Attenuates Interleukin-17-Producing (cid:2)(cid:3) T Cells during Acute Klebsiella pneumoniae Infection

J. Kolls, R. Peebles, Melissa H. Bloodworth, 2016 .

COX Inhibition Increases Alternaria-Induced Pulmonary Group 2 Innate Lymphoid Cell Responses and IL-33 Release in Mice

Pingsheng Wu, H. Kita, K. Boyd, 2020, The Journal of Immunology.

Human TH17 cells express a functional IL-13 receptor and IL-13 attenuates IL-17A production.

J. Kolls, R. Peebles, D. Newcomb, 2011, The Journal of allergy and clinical immunology.

R. Peebles, D. Newcomb, Joyce Cheung-Flynn, 2016, American journal of respiratory cell and molecular biology.

Human IgE mAbs define variability in commercial Aspergillus extract allergen composition.

A. Pomés, R. Peebles, Scott A. Smith, 2018, JCI insight.

A Functional IL-13 Receptor is Expressed on Polarized Murine CD4 + Th17 Cells and IL-13 Signaling Negatively Regulates Th17 Cytokine Production

J. Kolls, R. Peebles, D. Newcomb, 2009 .

A Functional IL-13 Receptor Is Expressed on Polarized Murine CD4+ Th17 Cells and IL-13 Signaling Attenuates Th17 Cytokine Production1

Martin L. Moore, Jay K. Kolls, J. Kolls, 2009, The Journal of Immunology.

Glucagon-like peptide 1 receptor signaling attenuates respiratory syncytial virus-induced type 2 responses and immunopathology.

L. Bastarache, K. Boyd, T. Hartert, 2018, The Journal of allergy and clinical immunology.

Prostaglandin I2 analogs inhibit Th1 and Th2 effector cytokine production by CD4 T cells

G. FitzGerald, R. Breyer, R. Peebles, 2007, Journal of leukocyte biology.

IL-13 Regulates Th17 Secretion of IL-17A in an IL-10–Dependent Manner

J. Kolls, R. Peebles, D. Newcomb, 2012, The Journal of Immunology.

Testosterone Decreases House Dust Mite–Induced Type 2 and IL-17A–Mediated Airway Inflammation

R. Peebles, D. Newcomb, Hubaida Fuseini, 2018, The Journal of Immunology.

Respiratory syncytial virus infection in the absence of STAT 1 results in airway dysfunction, airway mucus, and augmented IL-17 levels.

B. Graham, K. Hashimoto, J. Kolls, 2005, The Journal of allergy and clinical immunology.

STAT6 Signaling Attenuates Interleukin-17-Producing γδ T Cells during Acute Klebsiella pneumoniae Infection

J. Kolls, R. Peebles, Melissa H. Bloodworth, 2016, Infection and Immunity.

Production IL-13 Signaling Attenuates Th 17 Cytokine Th 17 Cells and + Polarized Murine CD 4 A Functional IL-13 Receptor Is Expressed

J. Kolls, R. Peebles, D. Newcomb, 2009 .

Allergic Airway Inflammation Decreases Lung Bacterial Burden following Acute Klebsiella pneumoniae Infection in a Neutrophil- and CCL8-Dependent Manner

K. Boyd, J. Kolls, R. Peebles, 2014, Infection and Immunity.

IL-33 Is a Cell-Intrinsic Regulator of Fitness during Early B Cell Development

R. Mitra, C. M. Eischen, R. Peebles, 2019, The Journal of Immunology.

TSLP and IL‐33 reciprocally promote each other's lung protein expression and ILC2 receptor expression to enhance innate type‐2 airway inflammation

Kelli L Boyd, H. Kita, K. Boyd, 2020, Allergy.

Deficiency of gp91phox inhibits allergic airway inflammation.

K. Boyd, R. Peebles, D. Newcomb, 2013, American journal of respiratory cell and molecular biology.

Estrogen and progesterone decrease let-7f microRNA expression and increase IL-23/IL-23 receptor signaling and IL-17A production in patients with severe asthma.

J. Fahrenholz, J. Kolls, R. Peebles, 2015, The Journal of allergy and clinical immunology.

Attenuation of chronic pulmonary inflammation in A2B adenosine receptor knockout mice.

D. Mitchell, H. Yin, R. Peebles, 2010, American journal of respiratory cell and molecular biology.

The histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activation

K. Boyd, R. Peebles, Melissa H. Bloodworth, 2016, Thorax.

Prostaglandin I2 Signaling Drives Th17 Differentiation and Exacerbates Experimental Autoimmune Encephalomyelitis

G. FitzGerald, Pingsheng Wu, R. Breyer, 2012, PloS one.

Respiratory Syncytial Virus Induces IL-25 and IL-33 Production In The Lungs

R. Peebles, M. Stier, Kasia Goleniewska, 2014 .