Lisa M Guay-Woodford

The polycystic kidney disease proteins, polycystin-1, polycystin-2, polaris, and cystin, are co-localized in renal cilia.

L. Guay-Woodford, B. Yoder, X. Hou, 2002, Journal of the American Society of Nephrology : JASN.

Volume progression in polycystic kidney disease.

Bernard F King, K. Bae, S. Klahr, 2006, The New England journal of medicine.

Magnetic resonance imaging evaluation of hepatic cysts in early autosomal-dominant polycystic kidney disease: the Consortium for Radiologic Imaging Studies of Polycystic Kidney Disease cohort.

Paul A Thompson, Bernard F King, Marijn E Brummer, 2005, Clinical journal of the American Society of Nephrology : CJASN.

CureGN Study Rationale, Design, and Methods: Establishing a Large Prospective Observational Study of Glomerular Disease.

Krzysztof Kiryluk, Matthias Kretzler, Brenda W Gillespie, 2019, American journal of kidney diseases : the official journal of the National Kidney Foundation.

Milder presentation of recessive polycystic kidney disease requires presence of amino acid substitution mutations.

Carsten Bergmann, Klaus Zerres, Ali Gharavi, 2003, Journal of the American Society of Nephrology : JASN.

Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells.

L. Guay-Woodford, B. Yoder, P. Komlósi, 2006, American journal of physiology. Renal physiology.

Renal structure in early autosomal-dominant polycystic kidney disease (ADPKD): The Consortium for Radiologic Imaging Studies of Polycystic Kidney Disease (CRISP) cohort.

Paul A Thompson, James F Glockner, Bernard F King, 2003, Kidney international.