Ivy E. Dick

发表

Mechanism of Local and Global Ca2+ Sensing by Calmodulin in Complex with a Ca2+ Channel

David T. Yue, D. T. Yue, M. Tadross, 2008, Cell.

CaV1.2 channelopathic mutations evoke diverse pathophysiological mechanisms

Manu Ben-Johny, Ivy E. Dick, K. Herold, 2022, bioRxiv.

Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels.

Manu Ben-Johny, D. T. Yue, Worawan B Limpitikul, 2015, Current molecular pharmacology.

Infanticide vs. inherited cardiac arrhythmias

R. Redon, P. Schwartz, M. Nyegaard, 2020, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.

Discovery of potent and use-dependent sodium channel blockers for treatment of chronic pain.

Jun Liang, J. W. Tarpley, M. Fisher, 2005, Bioorganic & medicinal chemistry letters.

Impaired CaV1.2 inactivation reduces the efficacy of calcium channel blockers in the treatment of LQT8.

D. Disilvestre, Ivy E. Dick, Moradeke A. Bamgboye, 2022, Journal of molecular and cellular cardiology.

Discovery of potent and use-dependent sodium channel blockers for treatment of chronic pain.

Jun Liang, J. W. Tarpley, M. Fisher, 2005, Bioorganic & medicinal chemistry letters.

Fibroblast growth factor homologous factors serve as a molecular rheostat in tuning arrhythmogenic cardiac late sodium current

G. Tomaselli, S. Marx, Manu Ben-Johny, 2022, Nature Cardiovascular Research.

David T. Yue: In Memoriam

H. Colecraft, Ivy E. Dick, 2015, Neuron.

A rendezvous with the queen of ion channels: Three decades of ion channel research by David T Yue and his Calcium Signals Laboratory

G. Tomaselli, Manu Ben-Johny, Worawan B Limpitikul, 2016, Channels.

Novel fluorescence resonance energy transfer‐based reporter reveals differential calcineurin activation in neonatal and adult cardiomyocytes

Hojjat Bazzazi, Rosy Joshi-Mukherjee, D. T. Yue, 2015, The Journal of physiology.

Nanodomain Ca2+ of Ca2+ channels detected by a tethered genetically encoded Ca2+ sensor

M. Mank, O. Griesbeck, D. T. Yue, 2012, Nature Communications.

nanodomain Ca 2 + of Ca 2 + channels detected by a tethered genetically encoded Ca 2 + sensor

M. Mank, O. Griesbeck, D. T. Yue, 2012 .

A disubstituted succinamide is a potent sodium channel blocker with efficacy in a rat pain model.

V. Warren, Y. S. Tang, K. Wafford, 2004, Biochemistry.

An autism-associated mutation in CaV1.3 channels has opposing effects on voltage- and Ca2+-dependent regulation

Manu Ben-Johny, D. T. Yue, Worawan B Limpitikul, 2016, Scientific Reports.

Protein kinase A modulation of CaV1.4 calcium channels

D. T. Yue, Ivy E. Dick, L. Sang, 2016, Nature Communications.

A modular switch for spatial Ca2+ selectivity in the calmodulin regulation of CaV channels

D. T. Yue, M. Tadross, Ivy E. Dick, 2008, Nature.

Calmodulin Mutations in Human Disease

Worawan B Limpitikul, Ivy E. Dick, John W. Hussey, 2023, Channels.

Sodium channel blockade may contribute to the analgesic efficacy of antidepressants.

G. Kaczorowski, B. Priest, W. Martin, 2007, The journal of pain : official journal of the American Pain Society.

Block of Peripheral Nerve Sodium Channels Selectively Inhibits Features of Neuropathic Pain in Rats

O. McManus, J. W. Tarpley, Z. Ali, 2006, Molecular Pharmacology.

Arrhythmogenesis in Timothy Syndrome is associated with defects in Ca2+-dependent inactivation

D. T. Yue, Ivy E. Dick, Rosy Joshi-Mukherjee, 2016, Nature Communications.

Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca(2+) currents and promote proarrhythmic behavior in ventricular myocytes.

Rosy Joshi-Mukherjee, Alfred L George, M. Overgaard, 2014, Journal of molecular and cellular cardiology.

Allosteric regulators selectively prevent Ca2+-feedback of CaV and NaV channels

G. Tomaselli, Manu Ben-Johny, D. T. Yue, 2018, eLife.

A Precision Medicine Approach to the Rescue of Function on Malignant Calmodulinopathic Long-QT Syndrome

Michael J Ackerman, Deborah DiSilvestre, D. Tester, 2017, Circulation research.

The molecular basis of the inhibition of CaV1 calcium-dependent inactivation by the distal carboxy tail

Manu Ben-Johny, D. T. Yue, Ivy E. Dick, 2021, The Journal of biological chemistry.

A bilobal model of Ca2+-dependent inactivation to probe the physiology of L-type Ca2+ channels

R. Winslow, J. Greenstein, D. T. Yue, 2018, The Journal of general physiology.

Apocalmodulin Itself Promotes Ion Channel Opening and Ca2+ Regulation

Manu Ben-Johny, D. T. Yue, Takanari Inoue, 2014, Cell.

Following Optogenetic Dimerizers and Quantitative Prospects.

Takanari Inoue, Ivy E. Dick, Manu Ben Johny, 2016, Biophysical journal.

Biophysical Perspective Following Optogenetic Dimerizers and Quantitative Prospects

Takanari Inoue, Ivy E. Dick, Jacqueline Niu, 2016 .

Structural and functional plasticity in long-term cultures of adult ventricular myocytes.

B. O’Rourke, Ting Liu, D. T. Yue, 2013, Journal of molecular and cellular cardiology.

Abstract 17783: Calmodulin Mutants Associated With Long QT Syndrome Suppress Inactivation of Cardiac L-type Ca2+ Currents and Prolong Action Potentials in Guinea-Pig Ventricular Myocytes

A. George, D. T. Yue, Worawan B Limpitikul, 2013 .

Biophysical and pharmacological properties of the voltage‐gated potassium current of human pancreatic β‐cells

O. McManus, J. Herrington, B. Priest, 2005, The Journal of physiology.

Functional assay of voltage-gated sodium channels using membrane potential-sensitive dyes.

V. Warren, G. Kaczorowski, B. Priest, 2004, Assay and drug development technologies.

Mechanism of Spatial Ca2+ Selectivity of a Ca2+ Sensor in Complex with a Ca2+ Source

D. T. Yue, M. Tadross, Ivy E. Dick, 2014 .

CACNA1C-Related Channelopathies.

Ivy E. Dick, K. Herold, John W. Hussey, 2023, Handbook of experimental pharmacology.

Resolving the molecular fingerprint of the distal carboxy tail in modulating CaV1 calcium dependent inactivation

Manu Ben-Johny, D. T. Yue, Ivy E. Dick, 2021, bioRxiv.

A high-capacity membrane potential FRET-based assay for NaV1.8 channels.

G. Kaczorowski, B. Priest, H. Knaus, 2006, Assay and drug development technologies.