Rosane B. de Oliveira

Therapeutical targeting of nucleic acid-sensing Toll-like receptors prevents experimental cerebral malaria

Douglas T. Golenbock, F. Gusovsky, D. Golenbock, 2011, Proceedings of the National Academy of Sciences.

Malaria primes the innate immune response due to interferon-γ induced enhancement of toll-like receptor expression and function

D. Golenbock, R. Gazzinelli, C. Ropert, 2009, Proceedings of the National Academy of Sciences.

Heme induces programmed necrosis on macrophages through autocrine TNF and ROS production.

D. Golenbock, M. Kelliher, F. Chan, 2012, Blood.

Increased Survival in B-Cell-Deficient Mice during Experimental Cerebral Malaria Suggests a Role for Circulating Immune Complexes

S. Ram, D. Golenbock, R. Gazzinelli, 2014, mBio.

Hemolysis-induced lethality involves inflammasome activation by heme

D. Golenbock, M. Bozza, D. Zamboni, 2014, Proceedings of the National Academy of Sciences.

DNA-Containing Immunocomplexes Promote Inflammasome Assembly and Release of Pyrogenic Cytokines by CD14+ CD16+ CD64high CD32low Inflammatory Monocytes from Malaria Patients

B. Diamond, S. Ram, D. Golenbock, 2015, mBio.

Interferon-γ and Granulocyte/Monocyte Colony-stimulating Factor Production by Natural Killer Cells Involves Different Signaling Pathways and the Adaptor Stimulator of Interferon Genes (STING)

J. Cavaillon, F. Souza-Fonseca-Guimaraes, D. Golenbock, 2013, The Journal of Biological Chemistry.

Gonococcal lipooligosaccharide sialylation: virulence factor and target for novel immunotherapeutics.

S. Ram, S. Gulati, P. Rice, 2017, Pathogens and disease.