M. Nassar

发表

Sodium channels and mammalian sensory mechanotransduction

E. Hummler, Fan Wang, François Rugiero, 2012, Molecular pain.

Tamoxifen‐inducible NaV1.8‐CreERT2 recombinase activity in nociceptive neurons of dorsal root ganglia

J. Wood, M. Nassar, Jing Zhao, 2006, Genesis.

Ion channel activities implicated in pathological pain.

L. Drew, J. Wood, B. Abrahamsen, 2004, Novartis Foundation symposium.

Sensory Axon-Derived Neuregulin-1 Is Required for Axoglial Signaling and Normal Sensory Function But Not for Long-Term Axon Maintenance

S. McMahon, C. Birchmeier, D. Bennett, 2009, The Journal of Neuroscience.

Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons

A. Dickenson, Fan Wang, J. Wood, 2012, Nature Communications.

Deletion of Annexin 2 Light Chain p11 in Nociceptors Causes Deficits in Somatosensory Coding and Pain Behavior

A. Dickenson, V. Gerke, J. Wood, 2006, The Journal of Neuroscience.

Nociceptor-expressed ephrin-B2 regulates inflammatory and neuropathic pain

K. Kullander, J. Wood, M. Lagerström, 2010, Molecular pain.

Nerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice

S. McMahon, G. Mallucci, J. Wood, 2006, Molecular pain.

Neuropathic pain develops normally in mice lacking both Nav1.7 and Nav1.8

J. Wood, M. Nassar, A. Levato, 2005, Molecular pain.

Absence of Whisker-Related Pattern Formation in Mice with NMDA Receptors Lacking Coincidence Detection Properties and Calcium Signaling

S. Hunt, I. Dean, A. Gibb, 2003, The Journal of Neuroscience.

Single-channel currents from recombinant NM DANRla /NR2D receptors expressed in Xenopus oocytes

D. Wyllie, R. Schoepfer, M. Nassar, 1996, Proceedings of the Royal Society of London. Series B: Biological Sciences.

Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain

Paul F. Alewood, Irina Vetter, Katharina Zimmermann, 2016, Toxins.

A P2X purinoceptor cDNA conferring a novel pharmacological profile

Geoffrey Burnstock, Yi Zhang, G. Burnstock, 1995, FEBS letters.

Painful and painless mutations of SCN9A and SCN11A voltage-gated sodium channels

M. Nassar, M. Baker, 2020, Pflügers Archiv - European Journal of Physiology.

Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain.

G. Forlani, A. Dickenson, J. Wood, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Genetic ablation of delta opioid receptors in nociceptive sensory neurons increases chronic pain and abolishes opioid analgesia

D. Filliol, C. Gaveriaux-Ruff, R. Maldonado, 2011, PAIN.

Mouse DRG Cell Line with Properties of Nociceptors

D. Grundy, M. Holley, M. Nassar, 2015, PloS one.

Nociceptor-derived brain-derived neurotrophic factor regulates acute and inflammatory but not neuropathic pain

S. McMahon, S. Pezet, M. Fitzgerald, 2006, Molecular and Cellular Neuroscience.

Role of Na(v)1.9 in activity-dependent axon growth in motoneurons.

Preeti Yadav, Benjamin Dombert, Michael Sendtner, 2012, Human molecular genetics.

Veratridine produces distinct calcium response profiles in mouse Dorsal Root Ganglia neurons

D. Grundy, M. Nassar, Ciara G. Doran, 2017, Scientific Reports.

System: Mechanisms and Functions Neurotrophin-Mediated Rapid Signaling in the Central Nervous

A. Konnerth, H. Yeh, P. Castillo, 2012 .

Small RNAs Control Sodium Channel Expression, Nociceptor Excitability, and Pain Thresholds

Matthias Merkenschlager, J. Perkins, C. Asante, 2010, The Journal of Neuroscience.

Comparative studies between growth regulators and nanoparticles on growth and mitotic index of pea plants under salinity

N. Elsheery, M. Nassar, S. El-Hefnawy, 2020 .

NaV1.7 and pain: contribution of peripheral nerves

R. Carr, C. Weidner, P. Reeh, 2017, Pain.

Na+ current properties in islet α- and β-cells reflect cell-specific Scn3a and Scn9a expression

F. Ashcroft, P. Rorsman, F. Hofmann, 2014, The Journal of physiology.

Behavioral deficits and subregion-specific suppression of LTP in mice expressing a population of mutant NMDA receptors throughout the hippocampus.

T. Bliss, R. Morris, C. Specht, 2009, Learning & memory.

The antimicrobial potential of Lactobacillus acidophilus on pathogenic bacteria causing diarrhea

Z. Ibrahim, M. Nassar, R. Bassyouni, 2015 .

Determination of NMDA NR1 subunit copy number in recombinant NMDA receptors

P. Stern, D. Colquhoun, D. Wyllie, 1995, Proceedings of the Royal Society of London. Series B: Biological Sciences.

Recombinant nicotinic receptors, expressed in Xenopus oocytes, do not resemble native rat sympathetic ganglion receptors in single‐channel behaviour.

D. Colquhoun, R. Schoepfer, M. Nassar, 1997, The Journal of physiology.

Multiple roles for NaV1.9 in the activation of visceral afferents by noxious inflammatory, mechanical, and human disease–derived stimuli

Qasim Aziz, Vincent Cibert-Goton, John N. Wood, 2014, PAIN®.

The Cell and Molecular Basis of Mechanical, Cold, and Inflammatory Pain

C. Asante, A. Dickenson, Steve Marsh, 2008, Science.

RET Signaling Is Required for Survival and Normal Function of Nonpeptidergic Nociceptors

Robert W Gereau, Hideki Enomoto, Masato Hoshi, 2010, The Journal of Neuroscience.

Nociceptor-specific gene deletion using heterozygous NaV1.8-Cre recombinase mice

G. Forlani, A. Dickenson, J. Wood, 2005, Pain.

Parallel “Pain” Pathways Arise from Subpopulations of Primary Afferent Nociceptor

John N. Wood, Allan I. Basbaum, A. Basbaum, 2005, Neuron.

GRK2: A Novel Cell-Specific Regulator of Severity and Duration of Inflammatory Pain

G. Dorn, A. Kavelaars, Cindy T. J. van Velthoven, 2010, The Journal of Neuroscience.

Pain without Nociceptors? Nav1.7-Independent Pain Mechanisms

J. Wood, S. Santana-Varela, S. Falk, 2014, Cell reports.

Mice lacking Kcns1 in peripheral neurons show increased basal and neuropathic pain sensitivity

S. McMahon, K. Futai, F. Denk, 2018, Pain.

Tamoxifen induces cellular stress in the nervous system by inhibiting cholesterol synthesis

S. McMahon, L. Ramer, M. Ramer, 2015, Acta Neuropathologica Communications.

Reduced excitability and impaired nociception in peripheral unmyelinated fibers from Nav1.9-null mice

R. Carr, C. Weidner, P. Reeh, 2017, Pain.

Tamoxifen induces cellular stress in the nervous system by inhibiting cholesterol synthesis

S. McMahon, L. Ramer, M. Ramer, 2015, Acta Neuropathologica Communications.

GTP up‐regulated persistent Na+ current and enhanced nociceptor excitability require NaV1.9

J. Wood, M. Nassar, Johan A. R. Östman, 2008, The Journal of physiology.

Use dependence of peripheral nociceptive conduction in the absence of tetrodotoxin‐resistant sodium channel subtypes

C. Weidner, P. Reeh, M. Nassar, 2016, The Journal of physiology.

Sodium channels in primary sensory neurons: relationship to pain states.

A. Akopian, J. Wood, Yanning Ding, 2002, Novartis Foundation symposium.