Z. Ling

发表

Hyperglycemia contributes to impaired insulin response in GK rat islets.

C. Östenson, S. Efendić, A. Khan, 2001, Diabetes.

Lipid phosphate phosphatases regulate signal transduction through glycerolipids and sphingolipids.

D. Brindley, Z. Ling, D. English, 2002, Biochimica et biophysica acta.

Quantifying the Carboxylation of Pyruvate in Pancreatic Islets (*)

B. Landau, A. Khan, Akhtar Khan, 1996, The Journal of Biological Chemistry.

Lipid phosphate phosphatase-1 dephosphorylates exogenous lysophosphatidate and thereby attenuates its effects on cell signalling.

D. Brindley, Z. Ling, C. Pilquil, 2001, Prostaglandins & other lipid mediators.

Deficiency of pyruvate dehydrogenase activity in pancreatic islets of diabetic GK rats.

V. Grill, Z. Ling, C. Ostenson, 1995, Endocrinology.

Glucose metabolism in Goto-Kakizaki rat islets.

B. Landau, S. Efendić, S. Abdel-Halim, 1998, Endocrinology.

Inhibitory effects of fatty acids on glucose-regulated B-cell function: association with increased islet triglyceride stores and altered effect of fatty acid oxidation on glucose metabolism.

Yun-ping Zhou, V. Grill, Z. Ling, 1996, Metabolism: clinical and experimental.

Effects of 3-mercaptopicolinic acid and a derivative of chlorogenic acid (S-3483) on hepatic and islet glucose-6-phosphatase activity.

B. Landau, A. Herling, S. Efendić, 1998, European journal of pharmacology.

Increased glucocorticoid sensitivity in islet beta-cells: effects on glucose 6-phosphatase, glucose cycling and insulin release

C. Östenson, B. Landau, J. Gustafsson, 1998, Diabetologia.

Pancreatic beta cells are important targets for the diabetogenic effects of glucocorticoids.

C. Östenson, J. Gustafsson, S. Efendić, 1997, The Journal of clinical investigation.

Co-ordinate regulation of growth factor receptors and lipid phosphate phosphatase-1 controls cell activation by exogenous lysophosphatidate.

D. Brindley, Z. Ling, C. Pilquil, 2001, Biochemical Society transactions.