A. Chee

F. Metzger, A. Hoeflich, G. Lynch, 2016, Growth hormone & IGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society.

Tranilast administration reduces fibrosis and improves fatigue resistance in muscles of mdx dystrophic mice

D. Stapleton, G. Lynch, R. Koopman, 2014, Fibrogenesis & tissue repair.

Glycine administration attenuates progression of dystrophic pathology in prednisolone-treated dystrophin/utrophin null mice

G. Lynch, R. Koopman, D. J. Ham, 2019, Scientific Reports.

Functional β-Adrenoceptors Are Important for Early Muscle Regeneration in Mice through Effects on Myoblast Proliferation and Differentiation

G. Lynch, P. Gregorevic, A. Chee, 2014, PloS one.

Functional b -Adrenoceptors Are Important for Early Muscle Regeneration in Mice through Effects on Myoblast Proliferation and Differentiation

G. Lynch, P. Gregorevic, A. Chee, 2014 .

L-Citrulline Protects Skeletal Muscle Cells from Cachectic Stimuli through an iNOS-Dependent Mechanism

G. Lynch, R. Koopman, D. J. Ham, 2015, PloS one.

Muscle-specific deletion of SOCS3 increases the early inflammatory response but does not affect regeneration after myotoxic injury

D. Stapleton, G. Lynch, R. Koopman, 2016, Skeletal Muscle.

Mas Receptor Activation Slows Tumor Growth and Attenuates Muscle Wasting in Cancer.

A. Judge, D. Stapleton, J. Trevino, 2018, Cancer research.

Control of Glucocorticoid Receptor Levels by PTEN Establishes a Failsafe Mechanism for Tumor Suppression.

R. Bronson, R. Hobbs, R. Daly, 2020, Molecular cell.

Alterations in Notch signalling in skeletal muscles from mdx and dko dystrophic mice and patients with Duchenne muscular dystrophy

A. Russell, C. Angelini, S. Lamon, 2014, Experimental physiology.

Phosphorylation within the cysteine-rich region of dystrophin enhances its association with β-dystroglycan and identifies a potential novel therapeutic target for skeletal muscle wasting.

D. Goodlett, S. Shaffer, J. Chamberlain, 2014, Human molecular genetics.

Early functional muscle regeneration after myotoxic injury in mice is unaffected by nNOS absence.

G. McConell, G. Lynch, A. Chee, 2011, American Journal of Physiology. Regulatory Integrative and Comparative Physiology.

cancerof exertional dyspnea in patients with

Mellar P. Davis, I. Mcbride, L. Ferreira, 2015 .

Inhibition of the renin–angiotensin system improves physiological outcomes in mice with mild or severe cancer cachexia

G. Lynch, K. Murphy, A. Chee, 2013, International journal of cancer.

Muscle-specific deletion of SOCS3 increases the early inflammatory response but does not affect regeneration after myotoxic injury

D. Stapleton, G. Lynch, R. Koopman, 2016, Skeletal Muscle.

Muscle‐specific deletion of SOCS3 does not reduce the anabolic response to leucine in a mouse model of acute inflammation

D. Stapleton, G. Lynch, R. Koopman, 2017, Cytokine.

Phosphorylation within the cysteine-rich region of dystrophin enhances its association with β-dystroglycan and identifies a potential novel therapeutic target for skeletal muscle wasting.

D. Goodlett, S. Shaffer, J. Chamberlain, 2014, Human molecular genetics.

Deletion of suppressor of cytokine signaling 3 (SOCS3) in muscle stem cells does not alter muscle regeneration in mice after injury

G. Lynch, R. Koopman, M. Caldow, 2019, PLoS ONE.

Early functional muscle regeneration after myotoxic injury in mice is unaffected by nNOS absence.

G. McConell, G. Lynch, A. Chee, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

Alterations in Notch signalling in skeletal muscles from mdx and dko dystrophic mice and patients with Duchenne muscular dystrophy

A. Russell, C. Angelini, S. Lamon, 2014, Experimental physiology.

Glycine supplementation during calorie restriction accelerates fat loss and protects against further muscle loss in obese mice.

G. Lynch, R. Koopman, D. J. Ham, 2016, Clinical nutrition.