Bing-Guo Li

Treatment of burned rats with insulin-like growth factor I inhibits the catabolic response in skeletal muscle.

J. Fischer, P. Hasselgren, C. Fang, 1998, American journal of physiology. Regulatory, integrative and comparative physiology.

Effect Of In vivo Infusion of Granulocyte Colony-Stimulating Factor on Immune Function

J. Alexander, C. Ogle, J. Ogle, 2002, Shock.

Protein breakdown in muscle from burned rats is blocked by insulin-like growth factor i and glycogen synthase kinase-3beta inhibitors.

P. Hasselgren, J. James, G. Warden, 2005, Endocrinology.

GSK-3beta activity is increased in skeletal muscle after burn injury in rats.

D. Supp, P. Hasselgren, J. James, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

Insulin-like growth factor-I inhibits dexamethasone-induced proteolysis in cultured L6 myotubes through PI3K/Akt/GSK-3beta and PI3K/Akt/mTOR-dependent mechanisms.

P. Hasselgren, C. Fang, Bing-Guo Li, 2005, The international journal of biochemistry & cell biology.

Thermal Injury Induces the Development of Inflammatory Macrophages from Nonadherent Bone Marrow Cells

J. Alexander, C. Ogle, J. Ogle, 1997, Inflammation.

Insulin-like growth factor-I blocks dexamethasone-induced protein degradation in cultured myotubes by inhibiting multiple proteolytic pathways: 2002 ABA paper.

P. Hasselgren, G. Warden, C. Fang, 2004, The Journal of burn care & rehabilitation.

Insulin-like growth factor-I inhibits lysosomal and proteasome-dependent proteolysis in skeletal muscle after burn injury.

P. Hasselgren, C. Wray, C. Fang, 2002, The Journal of burn care & rehabilitation.