J. D. Etlinger

发表

Removal of Z-lines and alpha-actinin from isolated myofibrils by a calcium-activated neutral protease.

M. Rabinowitz, D. Fischman, R. Zak, 1975, The Journal of biological chemistry.

Mechanism of work-induced hypertrophy of skeletal muscle.

A. Goldberg, D. Goldspink, J. D. Etlinger, 1975, Medicine and science in sports.

Regulation of protein degradation in muscle by calcium. Evidence for enhanced nonlysosomal proteolysis associated with elevated cytosolic calcium.

K. Matsumoto, P. Bernstein, J. D. Etlinger, 1985, The Journal of biological chemistry.

STEREOTACTIC RADIOSURGERY IMPROVES LOCOMOTOR RECOVERY AFTER SPINAL CORD INJURY IN RATS

A. Alfieri, C. Moorthy, N. Ouyang, 2008, Neurosurgery.

Role of the Polyol Pathway in Locomotor Recovery and Wallerian Degeneration after Spinal Cord Contusion Injury

N. Ouyang, J. D. Etlinger, R. Zeman, 2021, Neurotrauma reports.

Regulation of myofibrillar accumulation in chick muscle cultures: evidence for the involvement of calcium and lysosomes in non-uniform turnover of contractile proteins

J. D. Etlinger, G. Silver, 1985, The Journal of cell biology.

Smurf1 ubiquitin ligase causes downregulation of BMP receptors and is induced in monocrotaline and hypoxia models of pulmonary arterial hypertension

R. Mathew, Jing Huang, J. D. Etlinger, 2010, Experimental biology and medicine.

Partial functional recovery after complete spinal cord transection by combined chondroitinase and clenbuterol treatment

J. D. Etlinger, R. Zeman, Fusheng Bai, 2010, Pflügers Archiv - European Journal of Physiology.

Clenbuterol, a beta 2-receptor agonist, reduces net bone loss in denervated hindlimbs.

A. Hirschman, J. D. Etlinger, R. Zeman, 1991, The American journal of physiology.

Association of the chaperone glucose-regulated protein 58 (GRP58/ER-60/ERp57) with Stat3 in cytosol and plasma membrane complexes.

V. Fried, P. Sehgal, M. Shah, 2002, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.

Properties of abnormal proteins degraded rapidly in reticulocytes. Intracellular aggregation of the globin molecules prior to hydrolysis.

A. Goldberg, Y. Klemes, J. D. Etlinger, 1981, The Journal of biological chemistry.

Isolation of newly synthesised myosin filaments from skeletal muscle homogenates and myofibrils

M. Rabinowitz, D. Fischman, R. Zak, 1975, Nature.

SELECTIVE DEGRADATION OF ABNORMAL PROTEINS IN ANIMAL AND BACTERIAL CELLS

A. Goldberg, Y. Klemes, J. D. Etlinger, 1978 .

Phosphorylation of the proteasome activator PA28 is required for proteasome activation.

J. D. Etlinger, N. Li, K. Lerea, 1996, Biochemical and biophysical research communications.

Copurification of casein kinase II with 20 S proteasomes and phosphorylation of a 30-kDa proteasome subunit.

J. D. Etlinger, R. Ludemann, K. Lerea, 1993, The Journal of biological chemistry.

Calcium-dependent regulation of protein synthesis and degradation in muscle

J. D. Etlinger, T. Kameyama, 1979, Nature.

STUDIES OF THE SELECTIVITY AND MECHANISMS OF INTRACELLULAR PROTEIN DEGRADATION

A. Goldberg, J. D. Etlinger, J. D. Kowit, 1976 .

Control of protein degradation in reticulocytes and reticulocyte extracts by hemin.

A. Goldberg, J. D. Etlinger, 1980, The Journal of biological chemistry.

Endogenous inhibitor of nonlysosomal high molecular weight protease and calcium-dependent protease.

J. D. Etlinger, K. Murakami, 1986, Proceedings of the National Academy of Sciences of the United States of America.

Loss of ATP-dependent proteolysis with maturation of reticulocytes and erythrocytes.

J. D. Etlinger, S. Speiser, 1982, The Journal of biological chemistry.

Inhibition of intracellular proteolysis in muscle cultures by multiplication-stimulating activity. Comparison of effects of multiplication-stimulating activity and insulin on proteolysis, protein synthesis, amino acid uptake, and sugar transport.

J. D. Etlinger, R. Janeczko, 1984, The Journal of biological chemistry.

A soluble adenosine triphosphate-dependent proteolytic system in human peripheral red blood cells.

J. D. Etlinger, R. F. Rieder, A. Ibrahim, 1986, Blood.

Degradation of proteins with blocked amino groups by cytoplasmic proteases.

J. D. Etlinger, K. Murakami, 1987, Biochemical and biophysical research communications.

Calcium Regulation of Lysosomal Proteolysis in Skeletal Muscle a

P. Bernstein, J. D. Etlinger, R. Ludemann, 1986, Annals of the New York Academy of Sciences.

Clenbuterol retards loss of motor function in motor neuron degeneration mice

J. D. Etlinger, R. Zeman, Hong Peng, 2004, Experimental Neurology.

ATP-dependent proteolysis in red blood cell precursors.

J. D. Etlinger, R. F. Rieder, A. Ibrahim, 1987, Birth defects original article series.

Beta2-adrenoreceptor agonist-enhanced recovery of locomotor function after spinal cord injury is glutathione dependent.

J. D. Etlinger, R. Zeman, Hong Peng, 2006, Journal of neurotrauma.

Clenbuterol, a β2-Adrenoceptor Agonist, Improves Locomotor and Histological Outcomes after Spinal Cord Contusion in Rats

J. D. Etlinger, R. Zeman, Hong Peng, 1999, Experimental Neurology.

M and Z Band Components and the Assembly of Myofibrils

D. Fischman, J. D. Etlinger, 1973 .

X-irradiation of the Contusion Site Improves Locomotor and Histological Outcomes in Spinal Cord-Injured Rats

W. Couldwell, C. Moorthy, P. Visintainer, 2001, Experimental Neurology.

Clenbuterol, a beta2-adrenoceptor agonist, reduces scoliosis due to partial transection of rat spinal cord.

J. D. Etlinger, R. Zeman, Y. Zhang, 1997, The American journal of physiology.

Therapeutic target for external beam x-irradiation in experimental spinal cord injury.

C. Moorthy, J. D. Etlinger, R. Zeman, 2020, Journal of neurosurgery. Spine.

Improved functional recovery with oxandrolone after spinal cord injury in rats

W. Bauman, C. Cardozo, N. Ouyang, 2009, Neuroreport.

Tempol, a nitroxide antioxidant, improves locomotor and histological outcomes after spinal cord contusion in rats.

J. D. Etlinger, R. Murali, K. Das, 2004, Journal of neurotrauma.

Inhibition of x-irradiation-enhanced locomotor recovery after spinal cord injury by hyperbaric oxygen or the antioxidant nitroxide tempol.

C. Moorthy, J. D. Etlinger, R. Murali, 2007, Journal of neurosurgery. Spine.

Regulation of proteolysis in erythroid cells.

J. D. Etlinger, E. Wajnberg, S. Speiser, 1983, Biomedica biochimica acta.

Clenbuterol, a beta 2-agonist, retards atrophy in denervated muscles.

J. D. Etlinger, R. Ludemann, R. Zeman, 1987, The American journal of physiology.

Perfusion imaging of spinal cord contusion: injury-induced blockade and partial reversal by β2-agonist treatment in rats.

J. D. Etlinger, R. Zeman, Abraham Brown, 2014, Journal of neurosurgery. Spine.

Slow to fast alterations in skeletal muscle fibers caused by clenbuterol, a beta 2-receptor agonist.

J. D. Etlinger, T. Easton, R. Ludemann, 1988, The American journal of physiology.

Clenbuterol reduces degeneration of exercised or aged dystrophic (mdx) muscle

M. Danon, J. D. Etlinger, R. Zeman, 2000, Muscle & nerve.

Compositional studies of myofibrils from rabbit striated muscle

D. Fischman, R. Zak, J. D. Etlinger, 1976, The Journal of cell biology.

Regulation of IL-6 signaling by p53: STAT3- and STAT5-masking in p53-Val135-containing human hepatoma Hep3B cell lines.

P. Sehgal, J. D. Etlinger, R. Rayanade, 1998, Journal of immunology.

Human Hepatoma Hep 3 B Cell Lines-Containing 135 STAT 5-Masking in p 53-Val Regulation of IL-6 Signaling by p 53 : STAT 3-and

P. Sehgal, J. D. Etlinger, R. Rayanade, 1998 .

A soluble ATP-dependent proteolytic system responsible for the degradation of abnormal proteins in reticulocytes.

A. Goldberg, J. D. Etlinger, 1977, Proceedings of the National Academy of Sciences of the United States of America.

Clenbuterol, a beta 2-agonist, retards wasting and loss of contractility in irradiated dystrophic mdx muscle.

J. D. Etlinger, R. Zeman, Y. Zhang, 1994, The American journal of physiology.

Fructated protein is more resistant to ATP-dependent proteolysis than glucated protein possibly as a result of higher content of Maillard fluorophores.

J. D. Etlinger, G. Suárez, J. Maturana, 1995, Archives of biochemistry and biophysics.

Cellular Physiology of STAT3: Where’s the Cytoplasmic Monomer?*

V. Fried, P. Sehgal, J. D. Etlinger, 1999, The Journal of Biological Chemistry.

Regulation of Ca2+-dependent protein turnover in skeletal muscle by thyroxine.

P. Bernstein, J. D. Etlinger, R. Ludemann, 1986, The Biochemical journal.

hypoxia models of pulmonary arterial hypertension Smurf 1 ubiquitin ligase causes downregulation of BMP receptors and is induced in monocrotaline and

R. Mathew, Jing Huang, J. D. Etlinger, 2010 .

Biomolecular condensates in cell biology and virology: phase-separated membraneless organelles (MLOs).

P. Sehgal, K. Lerea, J. D. Etlinger, 2020, Analytical biochemistry.

Role of SMURF1 ubiquitin ligase in BMP receptor trafficking and signaling.

J. D. Etlinger, Koko Murakami, 2019, Cellular signalling.

A monoclonal antibody that distinguishes latent and active forms of the proteasome (multicatalytic proteinase complex).

J. D. Etlinger, D. Weitman, 1992, The Journal of biological chemistry.

Isolation and characterization of a novel endogenous inhibitor of the proteasome.

J. D. Etlinger, X. C. Li, M. Gu, 1991, Biochemistry.

Proteasome- and p53-dependent Masking of Signal Transducer and Activator of Transcription (STAT) Factors*

S. Ōmura, S. Sharma, P. Sehgal, 1997, The Journal of Biological Chemistry.

Ubiquitinated proteasome inhibitor is a component of the 26 S proteasome complex.

J. D. Etlinger, X. Li, 1992, Biochemistry.

Clenbuterol, a β2-agonist, retards atrophy in denervated muscles

J. D. Etlinger, R. Ludemann, R. Zeman, 1987 .

Endocytosis, proteolysis, and exocytosis of exogenous proteins by cultured myotubes.

J. D. Etlinger, R. Janeczko, R. Carrière, 1985, The Journal of biological chemistry.

A particle-associated ATP-dependent proteolytic activity in erythroleukemia cells.

J. D. Etlinger, R. F. Rieder, A. Ibrahim, 1985, The Journal of biological chemistry.

240-kDa proteasome inhibitor (CF-2) is identical to delta-aminolevulinic acid dehydratase.

J. D. Etlinger, M. Gu, G. Guo, 1994, The Journal of biological chemistry.