David Cantu

发表

Glycolytic inhibitor 2-deoxyglucose prevents cortical hyperexcitability after traumatic brain injury

F. Noubary, M. Whalen, Jenny B. Koenig, 2018, bioRxiv.

Oxidative Inactivation of Mitochondrial Aconitase Results in Iron and H2O2-Mediated Neurotoxicity in Rat Primary Mesencephalic Cultures

J. Schaack, Manisha N. Patel, David Cantu, 2009, PloS one.

Glycolytic inhibitor 2-deoxyglucose prevents cortical hyperexcitability after traumatic brain injury.

F. Noubary, M. Whalen, Jenny B. Koenig, 2019, JCI insight.

Generation of reactive oxygen species in 1-methyl-4-phenylpyridinium (MPP+) treated dopaminergic neurons occurs as an NADPH oxidase-dependent two-wave cascade

M. Das, Susan M. Jones, W. Griffin, 2011, Journal of Neuroinflammation.

Traumatic Brain Injury Increases Cortical Glutamate Network Activity by Compromising GABAergic Control.

A. Taylor-Weiner, C. Dulla, G. Tesco, 2015, Cerebral cortex.

Mitochondrial aconitase knockdown attenuates paraquat‐induced dopaminergic cell death via decreased cellular metabolism and release of iron and H2O2

Manisha N. Patel, R. Fulton, Derek A. Drechsel, 2011, Journal of neurochemistry.

Astrocytic glutamate uptake is slow and does not limit neuronal NMDA receptor activation in the neonatal neocortex

N. Danbolt, C. Dulla, David Cantu, 2015, Glia.

Excitation in Neocortical Layer V Pyramidal Cells Decrease in Synaptic Inhibition and Increase in Acute Injury to Superficial Cortex Leads to a

G. Stanley, M. Ding, A. Taylor-Weiner, 2015 .

In vivo KPT-350 treatment decreases cortical hyperexcitability following traumatic brain injury

S. Tamir, C. Dulla, Daniel E. Croker, 2020, Brain injury.

Glycolytic inhibitor 2-deoxyglucose prevents cortical hyperexcitability after traumatic brain injury

F. Noubary, M. Whalen, Jenny B. Koenig, 2018, bioRxiv.