Jenny H. Hong

发表

A Human Ubiquitin Conjugating Enzyme (E2)-HECT E3 Ligase Structure-function Screen*

Tharan Srikumar, Brian Raught, Ryan Doherty, 2012, Molecular & Cellular Proteomics.

Increased BRAF Heterodimerization Is the Common Pathogenic Mechanism for Noonan Syndrome-Associated RAF1 Mutants

Kyoung-Han Kim, B. Neel, Shengqing Gu, 2012, Molecular and Cellular Biology.

The use of ubiquitin lysine mutants to characterize E2–E3 linkage specificity: Mass spectrometry offers a cautionary “tail”

B. Raught, T. Srikumar, Jenny H. Hong, 2015, Proteomics.

KCMF1 (potassium channel modulatory factor 1) Links RAD6 to UBR4 (ubiquitin N-recognin domain-containing E3 ligase 4) and Lysosome-Mediated Degradation*

B. Raught, C. Arrowsmith, T. Srikumar, 2015, Molecular & Cellular Proteomics.

KCMF 1 links RAD 6 to UBR 4 and lysosome-mediated degradation

B. Raught, C. Arrowsmith, T. Srikumar, 2015 .

MEK-ERK pathway modulation ameliorates disease phenotypes in a mouse model of Noonan syndrome associated with the Raf1(L613V) mutation.

B. Neel, Kyoung-Han Kim, T. Araki, 2011, The Journal of clinical investigation.

Recent acquisition of imprinting at the rodent Sfmbt2 locus correlates with insertion of a large block of miRNAs

G. Mastromonaco, I. Caniggia, P. Vrana, 2011, BMC Genomics.

The LXR ligand T0901317 induces severe lipogenesis in the db/db diabetic mouse Published, JLR Papers in Press, August 16, 2003. DOI 10.1194/jlr.M300135-JLR200

R. Lawn, J. W. Chisholm, Jeffrey W. Chisholm, 2003, Journal of Lipid Research.

MEK-ERK pathway modulation ameliorates disease phenotypes in a mouse model of Noonan syndrome associated with the Raf1(L613V) mutation.

B. Neel, Kyoung-Han Kim, T. Araki, 2011, The Journal of clinical investigation.