Yingxin Li

发表

Cardiotoxic and Cardioprotective Features of Chronic &bgr;-Adrenergic Signaling

F. Wang, Hui Gao, E. Gao, 2013, Circulation research.

T-type Ca2+ channels regulate the exit of cardiac myocytes from the cell cycle after birth

S. Houser, Hui Gao, Catherine A Makarewich, 2013, Journal of molecular and cellular cardiology.

Cardiovascular Pharmacology: Open Access β-adrenergic System and Cardiac Physiology and Pathophysiology

Xiongwen Chen, Xiaoying Zhang, Yingxin Li, 2013 .

ÃÂ²-adrenergic System and Cardiac Physiology and Pathophysiology

Xiongwen Chen, Xiaoying Zhang, Yingxin Li, 2012 .

Insulin Inhibits Cardiac Contractility by Inducing a Gi-Biased β2-Adrenergic Signaling in Hearts

D. Parikh, E. Abel, Y. Zhang, 2014, Diabetes.

Hyperphosphorylation of the Cardiac Ryanodine Receptor at Serine 2808 Is Not Involved in Cardiac Dysfunction After Myocardial Infarction

S. Houser, W. Koch, E. Gao, 2012, Circulation research.

Cardiac GRK2 Ablation Induces a Novel Ca Handling Phenotype Resistant to Adverse Alterations and Remodeling After Myocardial Infarction

S. Houser, G. Dorn, W. Koch, 2012 .

Heart Failure Cardiac G-Protein–Coupled Receptor Kinase 2 Ablation Induces a Novel Ca Handling Phenotype Resistant to Adverse Alterations and Remodeling After Myocardial Infarction

S. Houser, G. Dorn, W. Koch, 2012 .

Increasing Cardiac Contractility After Myocardial Infarction Exacerbates Cardiac Injury and Pump Dysfunction

David M. Harris, S. Houser, W. Koch, 2009, Circulation research.

Increasing T‐type calcium channel activity by β‐adrenergic stimulation contributes to β‐adrenergic regulation of heart rates

S. Houser, J. Molkentin, M. Xie, 2017, The Journal of physiology.

Cardiomyocyte PKA Ablation Enhances Basal Contractility While Eliminates Cardiac β-Adrenergic Response Without Adverse Effects on the Heart.

S. Houser, Daohai Yu, Ying Tian, 2019, Circulation research.

Enhanced basal contractility but reduced excitation-contraction coupling efficiency and beta-adrenergic reserve of hearts with increased Cav1.2 activity.

S. Houser, H. Nakayama, J. Molkentin, 2010, American journal of physiology. Heart and circulatory physiology.

PKI-GFP Prevents Cardiac Dysfunction and Myocyte Death Induced by ISO

Naresh, F. Wang, Hui Gao, 2013 .