Armiyaw S. Nasamu

发表

A Genome-wide CRISPR Screen in Toxoplasma Identifies Essential Apicomplexan Genes

Tim Wang, Prathapan Thiru, Sebastian Lourido, 2016, Cell.

4-Aryl Pyrrolidines as a Novel Class of Orally Efficacious Antimalarial Agents. Part 1: Evaluation of 4-Aryl- N-benzylpyrrolidine-3-carboxamides.

J. Xu, D. Goldberg, Xiaofen Li, 2019, Journal of medicinal chemistry.

Complex nutrient channel phenotypes despite Mendelian inheritance in a Plasmodium falciparum genetic cross

Ankit Gupta, Anna K. Crater, J. Niles, 2020, PLoS pathogens.

Fast-Acting Small Molecules Targeting Malarial Aspartyl Proteases, Plasmepsins, Inhibit Malaria Infection at Multiple Life Stages.

Poonam, Vandana, D. Goldberg, 2018, ACS infectious diseases.

GeneTargeter: Automated In Silico Design for Genome Editing in the Malaria Parasite, Plasmodium falciparum.

Gaël Chambonnier, J. Niles, S. Dey, 2021, The CRISPR journal.

An integrated platform for genome engineering and gene expression perturbation in Plasmodium falciparum

J. Wagner, J. Niles, Stephen J. Goldfless, 2021, Scientific Reports.

An integrated platform for genome engineering and gene expression perturbation in Plasmodium falciparum

J. Wagner, J. Niles, Stephen J. Goldfless, 2019, bioRxiv.

Synthetic RNA–protein modules integrated with native translation mechanisms to control gene expression in malaria parasites

J. Niles, Stephen J. Goldfless, Suresh M. Ganesan, 2016, Nature Communications.

Potent acyl-CoA synthetase 10 inhibitors kill Plasmodium falciparum by disrupting triglyceride formation

Victoria C. Corey, E. Winzeler, D. Wirth, 2023, Nature Communications.

Muscle-Specific SIRT1 Gain-of-Function Increases Slow-Twitch Fibers and Ameliorates Pathophysiology in a Mouse Model of Duchenne Muscular Dystrophy

L. Guarente, A. Chalkiadaki, Armiyaw S. Nasamu, 2014, PLoS genetics.

Muscle-Specific SIRT 1 Gain-of-Function Increases Slow-Twitch Fibers and Ameliorates Pathophysiology in a Mouse Model of Duchenne Muscular Dystrophy

L. Guarente, A. Chalkiadaki, Armiyaw S. Nasamu, 2014 .

Malaria parasite plasmepsins: More than just plain old degradative pepsins

E. Istvan, D. Goldberg, Armiyaw S. Nasamu, 2020, The Journal of Biological Chemistry.

Assessment of Biological Role and Insight into Druggability of the Plasmodium falciparum Protease Plasmepsin V

D. Goldberg, J. Niles, Armiyaw S. Nasamu, 2018, bioRxiv.

EXP 2 is a nutrient-permeable channel in the vacuolar membrane of Plasmodium and is essential for protein export via

D. Goldberg, J. Zimmerberg, J. Niles, 2018 .

Plasmepsins IX and X are essential and druggable mediators of malaria parasite egress and invasion

N. Tolia, D. Fremont, D. Goldberg, 2017, Science.

Activation of the Plasmodium Egress Effector Subtilisin-Like Protease 1 Is Mediated by Plasmepsin X Destruction of the Prodomain

D. Goldberg, Armiyaw S. Nasamu, Kelly Rubiano, 2023, mBio.

Activation of the Plasmodium egress effector subtilisin-like protease 1 is achieved by plasmepsin X destruction of the propiece

D. Goldberg, Armiyaw S. Nasamu, Kelly Rubiano, 2023, bioRxiv.

Rounding precedes rupture and breakdown of vacuolar membranes minutes before malaria parasite egress from erythrocytes

D. Goldberg, J. Zimmerberg, J. Heuser, 2018, Cellular microbiology.

EXP2 is a nutrient-permeable channel in the vacuolar membrane of Plasmodium and is essential for protein export via PTEX

D. Goldberg, J. Zimmerberg, J. Niles, 2018, Nature Microbiology.