R. Stroud

发表

Targeted myocardial microinjections of a biocomposite material reduces infarct expansion in pigs.

Christine N. Koval, F. Spinale, Alexander J. Sheehy, 2008, The Annals of thoracic surgery.

Cardiac-restricted overexpression or deletion of tissue inhibitor of matrix metalloproteinase-4: differential effects on left ventricular structure and function following pressure overload-induced hypertrophy.

M. Zile, C. Baicu, Kia N. Zellars, 2014, American journal of physiology. Heart and circulatory physiology.

Long-Term Localized High-Frequency Electric Stimulation Within the Myocardial Infarct: Effects on Matrix Metalloproteinases and Regional Remodeling

Christine N. Koval, F. Spinale, C. B. Kern, 2010, Circulation.

Matrix metalloproteinase abundance in human myocardial fibroblasts: effects of sustained pharmacologic matrix metalloproteinase inhibition.

J. Ikonomidis, F. Spinale, A. Deschamps, 2003, Journal of molecular and cellular cardiology.

Selective spatiotemporal induction of matrix metalloproteinase-2 and matrix metalloproteinase-9 transcription after myocardial infarction.

M. Fini, D. Lovett, J. Bruce, 2006, American journal of physiology. Heart and circulatory physiology.

Differential effect of wall tension on matrix metalloproteinase promoter activation in the thoracic aorta.

Jeffrey A. Jones, J. Ikonomidis, F. Spinale, 2010, The Journal of surgical research.

Selective induction of matrix metalloproteinases and tissue inhibitor of metalloproteinases in atrial and ventricular myocardium in patients with atrial fibrillation.

M. Gold, J. Ikonomidis, J. Wharton, 2006, The American journal of cardiology.

Temporally and Regionally Disparate Differences in Plasmin Activity by Tranexamic Acid

S. Reeves, Christine N. Koval, Daryl L Reust, 2010, Anesthesia and analgesia.

Aortic dilatation with bicuspid aortic valves: cusp fusion correlates to matrix metalloproteinases and inhibitors.

Jeffrey A. Jones, J. Bavaria, J. Gorman, 2012, The Annals of thoracic surgery.

Relationships between plasma levels of matrix metalloproteinases and neurohormonal profile in patients with heart failure

R. McKelvie, Rizwan Afzal, A. Yan, 2008, European journal of heart failure.

Cardiac Restricted Overexpression of Membrane Type-1 Matrix Metalloproteinase Causes Adverse Myocardial Remodeling following Myocardial Infarction*

Christine N. Koval, A. Sinusas, L. Dobrucki, 2010, The Journal of Biological Chemistry.

Dynamic Changes in Matrix Metalloprotienase Activity Within the Human Myocardial Interstitium During Myocardial Arrest and Reperfusion

Christine N. Koval, J. Ikonomidis, F. Spinale, 2008, Circulation.

Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging mice.

Christine N. Koval, F. Spinale, R. Mukherjee, 2008, American journal of physiology. Heart and circulatory physiology.

Effects of age on plasma matrix metalloproteinases (MMPs) and tissue inhibitor of metalloproteinases (TIMPs).

M. Zile, F. Spinale, Catherine D. McClure, 2007, Journal of cardiac failure.

Aprotinin exerts differential and dose-dependent effects on myocardial contractility, oxidative stress, and cytokine release after ischemia-reperfusion.

S. Reeves, M. McEvoy, F. Spinale, 2008, The Annals of thoracic surgery.

Correcting versus resolving respiratory motion in free-breathing whole-heart MRA: a comparison in patients with thoracic aortic disease

U. Schoepf, D. Piccini, A. Varga-Szemes, 2019, European Radiology Experimental.

Plasma biomarkers for distinguishing etiologic subtypes of thoracic aortic aneurysm disease.

Jeffrey A. Jones, J. Ikonomidis, G. Ferrari, 2013, The Journal of thoracic and cardiovascular surgery.

ascending aortic aneurysms of patients with bicuspid or tricuspid aortic valves Expression of matrix metalloproteinases and endogenous inhibitors within

Jeffery A. Jones, J. Barbour, J. Bavaria, 2007 .

Differential Protein Kinase C Isoform Abundance in Ascending Aortic Aneurysms From Patients With Bicuspid Versus Tricuspid Aortic Valves

Jeffrey A. Jones, J. Bavaria, J. Gorman, 2007, Circulation.

Regulation of membrane type‐1 matrix metalloproteinase activity and intracellular localization in clinical thoracic aortic aneurysms

Elizabeth K. Nadeau, Jeffrey A. Jones, J. Ikonomidis, 2017, The Journal of thoracic and cardiovascular surgery.

Alterations in membrane type-1 matrix metalloproteinase abundance after the induction of thoracic aortic aneurysm in a murine model.

Jeffrey A. Jones, J. Ikonomidis, F. Spinale, 2010, American journal of physiology. Heart and circulatory physiology.

Temporal disparity in the induction of matrix metalloproteinases and tissue inhibitors of metalloproteinases after thoracic aortic aneurysm formation.

Jeffery A. Jones, J. Barbour, J. Ikonomidis, 2006, The Journal of thoracic and cardiovascular surgery.

Effects of Deletion of the Matrix Metalloproteinase 9 Gene on Development of Murine Thoracic Aortic Aneurysms

J. Barbour, M. Lindsey, J. Ikonomidis, 2005, Circulation.

Differential Effects of Epsilon-aminocaproic Acid and Aprotinin on Matrix Metalloproteinase Release in Patients Following Cardiopulmonary Bypass

J. Ikonomidis, F. Spinale, D. Botta, 2008, Journal of cardiovascular pharmacology.

xpression of matrix metalloproteinases and endogenous nhibitors within ascending aortic aneurysms of patients ith bicuspid or tricuspid aortic valves

Jeffery A. Jones, J. Barbour, J. Bavaria, 2007 .

Continuous Localized Monitoring of Plasmin Activity Identifies Differential and Regional Effects of the Serine Protease Inhibitor Aprotinin: Relevance to Antifibrinolytic Therapy

S. Reeves, Daryl L Reust, J. Abernathy, 2010, Journal of cardiovascular pharmacology.

Interstitial plasmin activity with epsilon aminocaproic acid: temporal and regional heterogeneity.

S. Reeves, Christine N. Koval, Daryl L Reust, 2010, The Annals of thoracic surgery.

Changes in plasma profiles of matrix metalloproteinases (MMPs) and tissue inhibitors of MMPs in stress-induced cardiomyopathy.

M. Zile, C. Leier, F. Spinale, 2012, Journal of cardiac failure.

Pressure overload-dependent membrane type 1-matrix metalloproteinase induction: relationship to LV remodeling and fibrosis.

Jeffrey A. Jones, M. Zile, C. Baicu, 2012, American journal of physiology. Heart and circulatory physiology.

Selective Targeting of Matrix Metalloproteinase Inhibition in Post-Infarction Myocardial Remodeling

F. Spinale, A. Deschamps, R. Mukherjee, 2006, Journal of cardiovascular pharmacology.

Caspase Inhibition Modulates Left Ventricular Remodeling Following Myocardial Infarction Through Cellular and Extracellular Mechanisms

F. Spinale, G. P. Escobar, R. Mukherjee, 2010, Journal of cardiovascular pharmacology.

Mesenchymal Cell Transplantation and Myocardial Remodeling After Myocardial Infarction

J. Gorman, R. Gorman, S. Itescu, 2009, Circulation.

Myocardial Interstitial Matrix Metalloproteinase Activity Is Altered by Mechanical Changes in LV Load: Interaction With the Angiotensin Type 1 Receptor

F. Spinale, A. Deschamps, Amy H. Leonardi, 2005, Circulation research.

Accelerated LV remodeling after myocardial infarction in TIMP-1-deficient mice: effects of exogenous MMP inhibition.

M. Zile, J. Ikonomidis, F. Spinale, 2005, American journal of physiology. Heart and circulatory physiology.

Circulating matrix metalloproteinase levels after ventricular septal defect repair in infants.

S. Reeves, F. Crawford, S. DeSantis, 2010, The Journal of thoracic and cardiovascular surgery.

Increased macrophage-derived SPARC precedes collagen deposition in myocardial fibrosis.

Jeffrey A. Jones, M. Zile, C. Baicu, 2018, American journal of physiology. Heart and circulatory physiology.

Interruption of endothelin signaling modifies membrane type 1 matrix metalloproteinase activity during ischemia and reperfusion.

Christine N. Koval, F. Spinale, A. Deschamps, 2008, American journal of physiology. Heart and circulatory physiology.

Aprotinin Exacerbates Left Ventricular Dysfunction After Ischemia/Reperfusion in Mice Lacking Tumor Necrosis Factor Receptor I

S. Reeves, Christine N. Koval, M. McEvoy, 2008, Journal of cardiovascular pharmacology.

Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.

M. Zile, J. Ikonomidis, F. Spinale, 2012, The Journal of thoracic and cardiovascular surgery.

Direct inhibition of the sodium/hydrogen exchanger after prolonged regional ischemia improves contractility on reperfusion independent of myocardial viability.

F. Spinale, G. P. Escobar, R. Mukherjee, 2003, The Journal of thoracic and cardiovascular surgery.

Circulating Matrix Metalloproteinase Levels Following Ventricular Septal Defect Repair in Infants

S. Reeves, A. Atz, Eric M. Graham, 2010 .

Myocyte contractility with caspase inhibition and simulated hyperkalemic cardioplegic arrest.

F. Spinale, R. Mukherjee, Robert E. Stroud, 2004, The Annals of thoracic surgery.

Targeted Overexpression of Tissue Inhibitor of Matrix Metalloproteinase-4 Modifies Post–Myocardial Infarction Remodeling in Mice

F. Spinale, R. Mukherjee, Robert E. Stroud, 2014, Circulation research.

Determinants of extracellular matrix remodelling are differentially expressed in paediatric and adult dilated cardiomyopathy

W. Chung, T. Hsia, Wendy K. Chung, 2011, European journal of heart failure.

Cardiac-Restricted Overexpression of Membrane Type-1 Matrix Metalloproteinase in Mice: Effects on Myocardial Remodeling With Aging

F. Spinale, G. P. Escobar, R. Mukherjee, 2009, Circulation. Heart failure.

Selective endothelin-A receptor inhibition after cardiac surgery: a safety and feasibility study.

S. Reeves, J. Ikonomidis, F. Spinale, 2007, The Annals of thoracic surgery.

Endothelin-A Receptor Inhibition Following Cardiopulmonary Bypass: Cytokines and Receptor Activation

S. Reeves, J. Ikonomidis, F. Spinale, 2009 .

Endothelin-A receptor inhibition after cardiopulmonary bypass: cytokines and receptor activation.

S. Reeves, J. Ikonomidis, F. Spinale, 2008, The Annals of thoracic surgery.

Aprotinin Modifies Left Ventricular Contractility and Cytokine Release After Ischemia-Reperfusion in a Dose-Dependent Manner in a Murine Model

S. Reeves, Christine N. Koval, M. McEvoy, 2009, Anesthesia and analgesia.

Protein Kinase C Isoform Activation and Endothelin-1 Mediated Defects in Myocyte Contractility After Cardioplegic Arrest and Reperfusion

F. Spinale, R. Mukherjee, Robert E. Stroud, 2006, Circulation.

The effects of leukocyte reduction on matrix metalloproteinase release in cardiopulmonary bypass.

J. Ikonomidis, F. Spinale, Robert E. Stroud, 2004, The journal of extra-corporeal technology.

Effects of Aprotinin or Tranexamic Acid on Proteolytic / Cytokine Profiles in Infants Following Cardiac Surgery

S. Reeves, T. Hsia, F. Spinale, 2011 .

Effects of aprotinin or tranexamic acid on proteolytic/cytokine profiles in infants after cardiac surgery.

S. Reeves, T. Hsia, F. Crawford, 2010, The Annals of thoracic surgery.

Differential Effects of Mechanical and Biological Stimuli on Matrix Metalloproteinase Promoter Activation in the Thoracic Aorta

Jeffrey A. Jones, J. Ikonomidis, F. Spinale, 2009, Circulation.

Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling.

F. Spinale, R. Mukherjee, Robert E. Stroud, 2011, American journal of physiology. Heart and circulatory physiology.

Differential effects of protein kinase C isoform activation in endothelin-mediated myocyte contractile dysfunction with cardioplegic arrest and reperfusion.

F. Spinale, A. Deschamps, Amy H. Leonardi, 2006, The Annals of thoracic surgery.

Caspase Inhibition Attenuates Contractile Dysfunction Following Cardioplegic Arrest and Rewarming in the Setting of Left Ventricular Failure

F. Spinale, R. Mukherjee, Robert E. Stroud, 2004, Journal of cardiovascular pharmacology.