William T. Rivers

发表

Targeted myocardial microinjections of a biocomposite material reduces infarct expansion in pigs.

Christine N. Koval, F. Spinale, Alexander J. Sheehy, 2008, The Annals of thoracic surgery.

Cardiac-restricted overexpression or deletion of tissue inhibitor of matrix metalloproteinase-4: differential effects on left ventricular structure and function following pressure overload-induced hypertrophy.

M. Zile, C. Baicu, Kia N. Zellars, 2014, American journal of physiology. Heart and circulatory physiology.

Long-Term Localized High-Frequency Electric Stimulation Within the Myocardial Infarct: Effects on Matrix Metalloproteinases and Regional Remodeling

Christine N. Koval, F. Spinale, C. B. Kern, 2010, Circulation.

Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging mice.

Christine N. Koval, F. Spinale, R. Mukherjee, 2008, American journal of physiology. Heart and circulatory physiology.

Continuous Localized Monitoring of Plasmin Activity Identifies Differential and Regional Effects of the Serine Protease Inhibitor Aprotinin: Relevance to Antifibrinolytic Therapy

S. Reeves, Daryl L Reust, J. Abernathy, 2010, Journal of cardiovascular pharmacology.

Calpain inhibition preserves myocardial structure and function following myocardial infarction.

M. Zile, F. Spinale, S. Mani, 2009, American journal of physiology. Heart and circulatory physiology.

Aprotinin Exacerbates Left Ventricular Dysfunction After Ischemia/Reperfusion in Mice Lacking Tumor Necrosis Factor Receptor I

S. Reeves, Christine N. Koval, M. McEvoy, 2008, Journal of cardiovascular pharmacology.

Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.

M. Zile, J. Ikonomidis, F. Spinale, 2012, The Journal of thoracic and cardiovascular surgery.

Inhibition of class I histone deacetylase activity represses matrix metalloproteinase-2 and -9 expression and preserves LV function postmyocardial infarction.

D. Menick, F. Spinale, D. Kimbrough, 2015, American journal of physiology. Heart and circulatory physiology.

Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling.

F. Spinale, R. Mukherjee, Robert E. Stroud, 2011, American journal of physiology. Heart and circulatory physiology.

Hemodynamics and myocardial blood flow patterns after placement of a cardiac passive restraint device in a model of dilated cardiomyopathy.

N. Baker, J. Ikonomidis, F. Spinale, 2011, The Journal of thoracic and cardiovascular surgery.

Cardiopulmonary Resuscitation Quality Issues.

L. White, B. Clemency, William T. Rivers, 2018, Cardiology clinics.

Short-term disruption in regional left ventricular electrical conduction patterns increases interstitial matrix metalloproteinase activity.

Christine N. Koval, F. Spinale, R. Mukherjee, 2010, American journal of physiology. Heart and circulatory physiology.

Heterogeneity in MT1-MMP activity with ischemia-reperfusion and previous myocardial infarction: relation to regional myocardial function.

Christine N. Koval, F. Spinale, J. Dixon, 2010, American journal of physiology. Heart and circulatory physiology.

Cardiac-restricted overexpression or deletion of tissue inhibitor of matrix metalloproteinase-4: differential effects on left ventricular structure and function following pressure overload-induced hypertrophy.

M. Zile, C. Baicu, Kia N. Zellars, 2014, American journal of physiology. Heart and circulatory physiology.

Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling.

F. Spinale, R. Mukherjee, Robert E. Stroud, 2011, American journal of physiology. Heart and circulatory physiology.