M. Schwarzkopf

Reduced sialylation status in UDP-N-acetylglucosamine-2-epimerase/N-acetylmannosamine kinase (GNE)-deficient mice

R. Horstkorte, Wenke Weidemann, D. Gagiannis, 2007, Glycoconjugate Journal.

Sialylation is essential for early development in mice

K. Knobeloch, W. Reutter, I. Horak, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Quantitation of CXCR4 Expression in Myocardial Infarction Using 99mTc-Labeled SDF-1α

H. Ly, J. Frangioni, R. Hajjar, 2008, Journal of Nuclear Medicine.

β2-Adrenergic Receptor Signaling in the Cardiac Myocyte is Modulated by Interactions With CXCR4

P. Altman, R. Hajjar, G. Haddad, 2010, Journal of cardiovascular pharmacology.

Chronic p 53 activity leads to skeletal muscle atrophy and muscle stem cell perturbation

D. Sassoon, D. Coletti, G. Marazzi, 2008 .

Deletion of CXCR4 in cardiomyocytes exacerbates cardiac dysfunction following isoproterenol administration

J. Chen, M. Schwarzkopf, L. Bénard, 2014, Gene Therapy.

Tissue expression and amino acid sequence of murine UDP-N-acetylglucosamine-2-epimerase/N-acetylmannosamine kinase.

W. Reutter, K. Danker, L. Lucka, 1999, European journal of biochemistry.

Muscle cachexia is regulated by a p53-PW1/Peg3-dependent pathway.

D. Sassoon, D. Coletti, G. Marazzi, 2006, Genes & development.

Molecular cloning and characterization of murine and human N-acetylglucosamine kinase.

W. Reutter, M. Berger, S. Hinderlich, 2000, European journal of biochemistry.