Lea Sarov-Blat

发表

Urotensin-II-mediated cardiomyocyte hypertrophy: effect of receptor antagonism and role of inflammatory mediators

N. Aiyar, Z. Ao, D. Johns, 2004, Naunyn-Schmiedeberg's Archives of Pharmacology.

Inhibition of p38 Mitogen-Activated Protein Kinase Improves Nitric Oxide–Mediated Vasodilatation and Reduces Inflammation in Hypercholesterolemia

R. Willette, J. Cockcroft, I. Wilkinson, 2011, Circulation.

Clinical and Population Studies Inhibition of p 38 Mitogen-Activated Protein Kinase Reduces Inflammation After Coronary Vascular Injury in Humans

R. Willette, M. Hurle, S. Jensen, 2010 .

Site-Specific Atherogenic Gene Expression Correlates With Subsequent Variable Lesion Development in Coronary and Peripheral Vasculature

R. Wilensky, M. Lu, E. Mohler, 2008, Arteriosclerosis, thrombosis, and vascular biology.

Effect of treatment for 12 weeks with rilapladib, a lipoprotein-associated phospholipase A2 inhibitor, on arterial inflammation as assessed with 18F-fluorodeoxyglucose-positron emission tomography imaging.

Z. Fayad, M. Farkouh, Esad Vucic, 2014, Journal of the American College of Cardiology.

Evaluation of Dynamic Contrast‐Enhanced MRI Measures of Lung Congestion and Endothelial Permeability in Heart Failure: A Prospective Method Validation Study

M. Graves, C. McEniery, C. Roberts, 2022, Journal of magnetic resonance imaging : JMRI.

In Vivo Activation of Peroxisome Proliferator-Activated Receptor-δ Protects the Heart from Ischemia/Reperfusion Injury in Zucker Fatty Rats

W. Bao, S. Nerurkar, R. Willette, 2008, Journal of Pharmacology and Experimental Therapeutics.

Losmapimod, a novel p38 mitogen-activated protein kinase inhibitor, in non-ST-segment elevation myocardial infarction: a randomised phase 2 trial

Manesh R. Patel, L. Newby, C. Granger, 2014, The Lancet.

Spatially resolved human kidney multi-omics single cell atlas highlights the key role of the fibrotic microenvironment in kidney disease progression

Mingyao Li, P. Grandi, C. Hunter, 2022, bioRxiv.

Human Epicardial Adipose Tissue Is a Source of Inflammatory Mediators

B. Goldstein, Anthony G. Johnson, A. Zalewski, 2003, Circulation.

PPARdelta activation normalizes cardiac substrate metabolism and reduces right ventricular hypertrophy in congestive heart failure.

C. Doe, R. Willette, S. Lenhard, 2007, Journal of cardiovascular pharmacology.

Effects of p38 mitogen-activated protein kinase inhibition on vascular and systemic inflammation in patients with atherosclerosis.

J. H. Rudd, Z. Fayad, F. Gleeson, 2012, JACC. Cardiovascular imaging.

The Drosophila takeout Gene Is a Novel Molecular Link between Circadian Rhythms and Feeding Behavior

M. Rosbash, W. So, Lea Sarov-Blat, 2000, Cell.

Quantitative analysis of orphan G protein-coupled receptor mRNAs by TaqMan Real-Time PCR: G2A and GPR4 lysophospholipid receptor expression in leukocytes and in a rat myocardial infarction-heart failure model.

P. Murdock, Z. Ao, D. Johns, 2005, Methods in molecular biology.

A Low‐Frequency Variant in MAPK14 Provides Mechanistic Evidence of a Link With Myeloperoxidase: A Prognostic Cardiovascular Risk Marker

A. Reiner, M. Ehm, B. Psaty, 2014, Journal of the American Heart Association.

takeout, a Novel DrosophilaGene under Circadian Clock Transcriptional Regulation

R. Allada, Michael J. McDonald, M. Rosbash, 2000, Molecular and Cellular Biology.

The study of LoSmapimod treatment on inflammation and InfarCtSizE (SOLSTICE): design and rationale.

V. Hasselblad, L. Newby, C. Granger, 2012, American heart journal.

Inhibition of p38 Mitogen-Activated Protein Kinase Reduces Inflammation After Coronary Vascular Injury in Humans

R. Willette, M. Hurle, S. Jensen, 2010, Arteriosclerosis, thrombosis, and vascular biology.

PPARδ Activation Normalizes Cardiac Substrate Metabolism and Reduces Right Ventricular Hypertrophy in Congestive Heart Failure

C. Doe, R. Willette, S. Lenhard, 2007 .

Predominance of a Proinflammatory Phenotype in Monocyte-Derived Macrophages From Subjects With Low Plasma HDL-Cholesterol

M. Jaye, R. S. Kiss, Y. Marcel, 2007, Arteriosclerosis, thrombosis, and vascular biology.

The Mutagenesis Protein MucB Interacts with Single Strand DNA Binding Protein and Induces a Major Conformational Change in Its Complex with Single-stranded DNA*

Z. Livneh, Lea Sarov-Blat, 1998, The Journal of Biological Chemistry.

Cathepsin D, a Lysosomal Protease, Regulates ABCA1-mediated Lipid Efflux*

R. S. Kiss, Y. Marcel, R. McPherson, 2006, Journal of Biological Chemistry.

Rationale and design of the Transformative Research in Diabetic Nephropathy (TRIDENT) Study.

R. Townsend, P. Guarnieri, K. Suszták, 2020, Kidney international.

Discovery of substituted maleimides as liver X receptor agonists and determination of a ligand-bound crystal structure.

Angela Smallwood, Ping Wang, David Haigh, 2005, Journal of medicinal chemistry.

Safety, tolerability, pharmacokinetics and pharmacodynamics of losmapimod following a single intravenous or oral dose in healthy volunteers.

A. McGeoch, D. Sprecher, J. Graggaber, 2013, British journal of clinical pharmacology.

Plasmid-encoded MucB protein is a DNA polymerase (pol RI) specialized for lesion bypass in the presence of MucA', RecA, and SSB.

Z. Livneh, M. Goldsmith, Lea Sarov-Blat, 2000, Proceedings of the National Academy of Sciences of the United States of America.