Wilson W. Cui

发表

Non-sense mutations in the dihydropyridine receptor beta1 gene, CACNB1, paralyze zebrafish relaxed mutants.

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2006, Cell calcium.

Summary accordion , a zebrafish behavioral mutant , has a muscle relaxation defect due to a mutation in the ATPase Ca 2 + pump SERCA 1

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2004 .

Zebrafish relatively relaxed mutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2007, Development.

The Zebrafish shocked Gene Encodes a Glycine Transporter and Is Essential for the Function of Early Neural Circuits in the CNS

Hiromi Hirata, R. Geisler, J. Y. Kuwada, 2005, The Journal of Neuroscience.

Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy

Jeremy W. Linsley, Hiromi Hirata, M. Hauser, 2013, Nature Communications.

Zebrafish bandoneon mutants display behavioral defects due to a mutation in the glycine receptor beta-subunit.

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Connexin 39.9 Protein Is Necessary for Coordinated Activation of Slow-twitch Muscle and Normal Behavior in Zebrafish*

K. Kawakami, Hiromi Hirata, J. Y. Kuwada, 2011, The Journal of Biological Chemistry.

accordion, a zebrafish behavioral mutant, has a muscle relaxation defect due to a mutation in the ATPase Ca2+ pump SERCA1

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2004, Development.

shocked Gene is required for the function of a premotor network in the zebrafish CNS.

J. Y. Kuwada, Wilson W. Cui, L. Saint‐Amant, 2004, Journal of neurophysiology.

The zebrafish ennui behavioral mutation disrupts acetylcholine receptor localization and motor axon stability

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2008, Developmental neurobiology.

TRPM7 Is Required within Zebrafish Sensory Neurons for the Activation of Touch-Evoked Escape Behaviors

Jeremy W. Linsley, Hiromi Hirata, J. Y. Kuwada, 2011, The Journal of Neuroscience.

NaV1.6a is required for normal activation of motor circuits normally excited by tactile stimulation

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2010, Developmental neurobiology.

touché Is Required for Touch-Evoked Generator Potentials within Vertebrate Sensory Neurons

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2010, The Journal of Neuroscience.

Early Development in Zebrafish Maturation of Neuromuscular Transmission During

K. Kawakami, J. Y. Kuwada, P. Brehm, 2015 .

Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy

Jeremy W. Linsley, J. Y. Kuwada, Wilson W. Cui, 2013, Nature Communications.