E. Busanello

发表

Disturbance of redox homeostasis by ornithine and homocitrulline in rat cerebellum: a possible mechanism of cerebellar dysfunction in HHH syndrome.

G. Leipnitz, M. Wajner, M. Grings, 2013, Life sciences.

Long-chain 3-hydroxy fatty acids accumulating in LCHAD and MTP deficiencies induce oxidative stress in rat brain

M. Wajner, C. Fernandes, M. Grings, 2010, Neurochemistry International.

Medium-chain fatty acids accumulating in MCAD deficiency elicit lipid and protein oxidative damage and decrease non-enzymatic antioxidant defenses in rat brain

M. Wajner, C. Dutra-filho, P. F. Schuck, 2009, Neurochemistry International.

Mitochondrial Oxidative Stress and Calcium-Dependent Permeability Transition are Key Players in the Mechanisms of Statins-Associated Side Effects

A. Vercesi, H. Oliveira, E. Busanello, 2017, Mitochondrial Diseases.

In vivo experimental evidence that the major metabolites accumulating in 3-hydroxy-3-methylglutaryl-CoA lyase deficiency induce oxidative stress in striatum of developing rats: a potential pathophysiological mechanism of striatal damage in this disorder.

G. Leipnitz, M. Wajner, C. Fernandes, 2013, Molecular genetics and metabolism.

Leucine-rich diet induces a shift in tumour metabolism from glycolytic towards oxidative phosphorylation, reducing glucose consumption and metastasis in Walker-256 tumour-bearing rats

A. Vercesi, C. Ramos, S. Consonni, 2019, Scientific Reports.

Impairment of brain redox homeostasis caused by the major metabolites accumulating in hyperornithinemia–hyperammonemia–homocitrullinuria syndrome in vivo

G. Leipnitz, M. Wajner, C. Fernandes, 2012, Metabolic Brain Disease.

3-Methylcrotonylglycine Disrupts Mitochondrial Energy Homeostasis and Inhibits Synaptic Na+,K+-ATPase Activity in Brain of Young Rats

M. Wajner, C. Ribeiro, Â. Zanatta, 2011, Cellular and Molecular Neurobiology.

Toxicity of octanoate and decanoate in rat peripheral tissues: evidence of bioenergetic dysfunction and oxidative damage induction in liver and skeletal muscle

M. Wajner, G. Scaini, E. Streck, 2012, Molecular and Cellular Biochemistry.

Mitochondrial bioenergetics deregulation caused by long-chain 3-hydroxy fatty acids accumulating in LCHAD and MTP deficiencies in rat brain: a possible role of mPTP opening as a pathomechanism in these disorders?

M. Wajner, D. Gelain, N. Gregersen, 2014, Biochimica et biophysica acta.

Disruption of Mitochondrial Homeostasis by Phytanic Acid in Cerebellum of Young Rats

C. Vargas, M. Wajner, A. U. Amaral, 2012, The Cerebellum.

Disruption of oxidative phosphorylation and synaptic Na(+), K(+)-ATPase activity by pristanic acid in cerebellum of young rats.

M. Wajner, C. Ribeiro, Â. Zanatta, 2014, Life sciences.

Marked inhibition of Na+, K+ - ATPase activity and the respiratory chain by phytanic acid in cerebellum from young rats: possible underlying mechanisms of cerebellar ataxia in Refsum disease

G. Leipnitz, C. Vargas, M. Wajner, 2013, Journal of Bioenergetics and Biomembranes.

Neurochemical Evidence that Pristanic Acid Impairs Energy Production and Inhibits Synaptic Na+, K+-ATPase Activity in Brain of Young Rats

M. Wajner, M. Grings, C. Viegas, 2011, Neurochemical Research.

Facilitation of Ca2+‐induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment

A. Vercesi, T. R. Figueira, H. Oliveira, 2018, Cell biology international.

Reduction of Na+, K+-ATPase activity and expression in cerebral cortex of glutaryl-CoA dehydrogenase deficient mice: a possible mechanism for brain injury in glutaric aciduria type I.

M. Wajner, C. Fernandes, B. Seminotti, 2012, Molecular genetics and metabolism.

Marked reduction of Na(+), K(+)-ATPase and creatine kinase activities induced by acute lysine administration in glutaryl-CoA dehydrogenase deficient mice.

M. Wajner, C. Fernandes, B. Seminotti, 2012, Molecular genetics and metabolism.

Long-chain 3-hydroxy fatty acids accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial trifunctional protein deficiencies uncouple oxidative phosphorylation in heart mitochondria

M. Wajner, A. U. Amaral, M. Grings, 2013, Journal of Bioenergetics and Biomembranes.

Disturbance of mitochondrial energy homeostasis caused by the metabolites accumulating in LCHAD and MTP deficiencies in rat brain.

M. Wajner, A. U. Amaral, M. Grings, 2010, Life sciences.

Disturbance of the glutamatergic system by glutaric acid in striatum and cerebral cortex of glutaryl-CoA dehydrogenase-deficient knockout mice: Possible implications for the neuropathology of glutaric acidemia type I

D. Souza, M. Wajner, C. Fernandes, 2014, Journal of the Neurological Sciences.

Pravastatin Chronic Treatment Sensitizes Hypercholesterolemic Mice Muscle to Mitochondrial Permeability Transition: Protection by Creatine or Coenzyme Q10

A. Vercesi, H. Oliveira, R. Catharino, 2017, Front. Pharmacol..

Evidence that the major metabolites accumulating in medium-chain acyl-CoA dehydrogenase deficiency disturb mitochondrial energy homeostasis in rat brain

F. Klamt, M. Wajner, G. C. Ferreira, 2009, Brain Research.

Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition.

A. Vercesi, A. Kowaltowski, T. R. Figueira, 2018, Free radical biology & medicine.

Disruption of Mitochondrial Homeostasis by Phytanic Acid in Cerebellum of Young Rats

C. Vargas, M. Wajner, A. U. Amaral, 2013, The Cerebellum.

Coenzyme Q10 or Creatine Counteract Pravastatin-Induced Liver Redox Changes in Hypercholesterolemic Mice

A. Vercesi, H. Oliveira, R. Catharino, 2018, Front. Pharmacol..

In vitro evidence that phytanic acid compromises Na+,K+-ATPase activity and the electron flow through the respiratory chain in brain cortex from young rats

C. Vargas, M. Wajner, M. Grings, 2010, Brain Research.

Chronic postnatal ornithine administration to rats provokes learning deficit in the open field task

G. Leipnitz, C. Vargas, M. Wajner, 2012, Metabolic Brain Disease.

3-Methylcrotonylglycine Disrupts Mitochondrial Energy Homeostasis and Inhibits Synaptic Na+,K+-ATPase Activity in Brain of Young Rats

M. Wajner, C. Ribeiro, Â. Zanatta, 2012, Cellular and Molecular Neurobiology.

Neurodevelopmental and cognitive behavior of glutaryl-CoA dehydrogenase deficient knockout mice.

M. Wajner, D. Koeller, S. Goodman, 2013, Life sciences.

Impairment of brain redox homeostasis caused by the major metabolites accumulating in hyperornithinemia–hyperammonemia–homocitrullinuria syndrome in vivo

G. Leipnitz, M. Wajner, C. Fernandes, 2012, Metabolic Brain Disease.

Dual mechanism of brain damage induced in vivo by the major metabolites accumulating in hyperornithinemia–hyperammonemia–homocitrullinuria syndrome

A. Sitta, C. Vargas, M. Wajner, 2011, Brain Research.

Toxicity of octanoate and decanoate in rat peripheral tissues: evidence of bioenergetic dysfunction and oxidative damage induction in liver and skeletal muscle

M. Wajner, G. Scaini, E. Streck, 2012, Molecular and Cellular Biochemistry.

Neurotoxic Effects of trans-Glutaconic Acid in Rats

P. F. Schuck, G. C. Ferreira, A. Tonin, 2013, Oxidative medicine and cellular longevity.

In vitro evidence that d-serine disturbs the citric acid cycle through inhibition of citrate synthase activity in rat cerebral cortex

M. Wajner, Patrícia Fernanda Schuck, Moacir Wajner, 2009, Brain Research.

Pristanic Acid Provokes Lipid, Protein, and DNA Oxidative Damage and Reduces the Antioxidant Defenses in Cerebellum of Young Rats

D. Souza, C. Vargas, M. Wajner, 2014, The Cerebellum.

Experimental evidence that pristanic acid disrupts mitochondrial homeostasis in brain of young rats

C. Vargas, M. Wajner, A. U. Amaral, 2012, Journal of neuroscience research.

Chronic postnatal ornithine administration to rats provokes learning deficit in the open field task

G. Leipnitz, C. Vargas, M. Wajner, 2012, Metabolic Brain Disease.