R. Kitchens

发表

ABCA1 promotes the efflux of bacterial LPS from macrophages and accelerates recovery from LPS-induced tolerance[S]

P. Thompson, K. Gauthier, A. Varley, 2010, Journal of Lipid Research.

Apolipoprotein A-II augments monocyte responses to LPS by suppressing the inhibitory activity of LPS-binding protein

P. Thompson, P. Rensen, J. Berbée, 2008, Innate immunity.

Native High-Density Lipoprotein Augments Monocyte Responses to Lipopolysaccharide (LPS) by Suppressing the Inhibitory Activity of LPS-Binding Protein1

P. Thompson, R. Kitchens, 2006, The Journal of Immunology.

Lipopolysaccharide (LPS)-binding Protein Inhibits Responses to Cell-bound LPS*

P. Tobias, T. Kirkland, P. Thompson, 2003, Journal of Biological Chemistry.

Role of CD14 in cellular recognition of bacterial lipopolysaccharides.

R. Kitchens, 2000, Chemical immunology.

Plasma Lipoproteins Promote the Release of Bacterial Lipopolysaccharide from the Monocyte Cell Surface*

R. Munford, J. Albers, G. Wolfbauer, 1999, The Journal of Biological Chemistry.

Bacterial lipopolysaccharide can enter monocytes via two CD14-dependent pathways.

R. Munford, R. Kitchens, P. Wang, 1998, Journal of immunology.

Distinct from That Used by Lipopolysaccharide Monocytic Cells via a CD 14-Dependent Pathway Lipoproteins and Synthetic Lipopeptides Activate Borrelia burgdorferi and Treponema pallidum

J. Radolf, R. Ulevitch, J. Pugin, 1998 .

Treponema pallidum and Borrelia burgdorferi lipoproteins and synthetic lipopeptides activate monocytic cells via a CD14-dependent pathway distinct from that used by lipopolysaccharide.

J. Radolf, R. Ulevitch, J. Pugin, 1998, Journal of immunology.

Lipopolysaccharide (LPS) partial structures inhibit responses to LPS in a human macrophage cell line without inhibiting LPS uptake by a CD14- mediated pathway

R. Ulevitch, R. Munford, R. Kitchens, 1992, The Journal of experimental medicine.

Stimulus-dependent Deacylation of Bacterial Lipopolysaccharide by Dendritic Cells

S. Fosmire, R. Munford, A. Takashima, 2003, The Journal of experimental medicine.

Phosphatidylinositides Bind to Plasma Membrane CD14 and Can Prevent Monocyte Activation by Bacterial Lipopolysaccharide*

Ping-yuan Wang, R. Munford, R. Kitchens, 1998, The Journal of Biological Chemistry.

Prolonged hepatomegaly in mice that cannot inactivate bacterial endotoxin

D. Rockey, R. Munford, T. Rogers, 2011, Hepatology.

Modulatory effects of sCD14 and LBP on LPS-host cell interactions

P. Thompson, R. Kitchens, 2005, Journal of endotoxin research.

Plasma constituents regulate LPS binding to, and release from, the monocyte cell surface

R. Munford, P. Thompson, R. Kitchens, 2000, Journal of endotoxin research.

Rearrangements of the tal-1 locus as clonal markers for T cell acute lymphoblastic leukemia.

Ó. Jónsson, R. Baer, G. Buchanan, 1991, The Journal of clinical investigation.

Bacterial lipopolysaccharide binds to CD14 in low-density domains of the monocyte-macrophage plasma membrane.

R. Munford, R. Kitchens, P. Wang, 1995, Journal of inflammation.

Enzymatically Deacylated Lipopolysaccharide (LPS) Can Antagonize LPS at Multiple Sites in the LPS Recognition Pathway (*)

R. Munford, R. Kitchens, 1995, The Journal of Biological Chemistry.

Purification and properties of acetyl coenzyme A synthetase from bakers' yeast.

E. Frenkel, R. Kitchens, 1977, The Journal of biological chemistry.

Modulatory effects of sCD14 and LBP on LPS-host cell interactions:

P. Thompson, R. Kitchens, 2005 .

Impact of sepsis-induced changes in plasma on LPS interactions with monocytes and plasma lipoproteins: roles of soluble CD14, LBP, and acute phase lipoproteins

P. Thompson, R. Kitchens, 2003, Journal of endotoxin research.

Intracellular Localization of Hepatic Propionyl‐CoA Carboxylase and Methylmalonyl‐CoA Mutase in Humans and Normal and Vitamin B12 Deficient Rats

E. Frenkel, R. Kitchens, 1975, British journal of haematology.

In tracellular Localization of Hepatic Propionyl-CoA Carboxylase and Me thylmalonyl-CoA Mu tase in Humans and Normal and Vitamin BIZ Deficient Rats

Eugene, R. Kitchens, P. Frenkel, 2022 .

Detection of minimal residual disease in acute lymphoblastic leukemia using immunoglobulin hypervariable region specific oligonucleotide probes.

Ó. Jónsson, R. Kitchens, F. Scott, 1990, Blood.

Detection of minimal residual disease in acute lymphoblastic leukemia using immunoglobulin hypervariable region specific oligonucleotide probes.

Ó. Jónsson, R. Kitchens, R. G. Smith, 1990, Blood.

Phenotypic heterogeneity of TDT+ cells in the blood and bone marrow: implications for surveillance of residual leukemia.

R. Kitchens, R. G. Smith, 1989, Blood.

Plasma CD14 decreases monocyte responses to LPS by transferring cell-bound LPS to plasma lipoproteins.

R. Munford, P. Thompson, R. Kitchens, 2001, The Journal of clinical investigation.

High-performance liquid chromatographic separation of cobalamins.

E. Frenkel, R. Kitchens, R. Prough, 1979, Journal of chromatography.

Measurement of tissue vitamin B12 by radioisotopic competitive inhibition assay and quantitation of tissue cobalamin fractions.

E. Frenkel, R. Kitchens, R. Prough, 1980, Methods in enzymology.

A simplified and rapid quantitative assay for propionic and methylmalonic acids in urine.

E. Frenkel, R. Kitchens, 1975, The Journal of laboratory and clinical medicine.

CD14-dependent internalization of bacterial lipopolysaccharide (LPS) is strongly influenced by LPS aggregation but not by cellular responses to LPS.

R. Munford, R. Kitchens, 1998, Journal of immunology.

Applicability of an enzymatic quantitation of methylmalonic, propionic, and acetic acids in normal and megaloblastic states.

E. Frenkel, R. Kitchens, 1977, Blood.

Effect of vitamin B12 deprivation on the in vivo levels of coenzyme A intermediates associated with propionate metabolism.

L. Hersh, E. Frenkel, R. Frenkel, 1974, The Journal of biological chemistry.

The effect of vitamin B12 deprivation on the enzymes of fatty acid synthesis.

E. Frenkel, R. Kitchens, J. Johnston, 1973, The Journal of biological chemistry.

Lack of effect of riboflavin deficiency on vitamin B12-related metabolic pathways and fatty acid synthesis.

E. Frenkel, H. Savage, R. Kitchens, 1979, The American journal of clinical nutrition.

Lipopolysaccharide-Binding Protein and Phospholipid Transfer Protein Release Lipopolysaccharides from Gram-Negative Bacterial Membranes

R. Munford, J. Albers, G. Wolfbauer, 2000, Infection and Immunity.

Hepatic uptake and deacylation of the LPS in bloodborne LPS-lipoprotein complexes

R. Munford, A. Varley, R. Kitchens, 2012, Innate immunity.

High-Density Lipoprotein Promotes Endothelial Cell Migration and Reendothelialization via Scavenger Receptor-B Type I

D. Rader, Y. Marcel, C. Mineo, 2005, Circulation research.

Apolipoprotein CI stimulates the response to lipopolysaccharide and reduces mortality in Gram‐negative sepsis

R. Kleemann, I. Bakker-Woudenberg, P. Rensen, 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Acute inflammation and infection maintain circulating phospholipid levels and enhance lipopolysaccharide binding to plasma lipoproteins Published, JLR Papers in Press, August 16, 2003. DOI 10.1194/jlr.M300228-JLR200

R. Munford, P. Thompson, R. Kitchens, 2003, Journal of Lipid Research.

The CD14 receptor does not mediate entry of Mycobacterium tuberculosis into human mononuclear phagocytes.

R. Valdivia, B. Samten, P. Barnes, 2003, FEMS immunology and medical microbiology.

[38] Acetyl-CoA synthetase from bakers' yeast (Saccharomyces cerevisiae): EC 6.2.1.1 acetate:CoA ligase (AMP-forming)

E. Frenkel, R. Kitchens, 1981 .

MD-2 binds to bacterial lipopolysaccharide.

P. Tobias, T. Kirkland, K. Soldau, 2001, Journal of endotoxin research.

Extraction and quantification of daunomycin and doxorubicin in tissues.

E. Frenkel, J. Strauss, R. Kitchens, 1980, Journal of chromatography.

Effect of vitamin B-12 deprivation on the rates of synthesis and degradation of rat liver fatty acid synthetase.

E. Frenkel, R. Frenkel, R. Kitchens, 1974, Archives of biochemistry and biophysics.