Carin I M Dahlberg

发表

B cell-intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice.

J. Hartwig, L. Notarangelo, J. Walter, 2012, Blood.

IL-2 in the tumor microenvironment is necessary for Wiskott-Aldrich syndrome protein deficient NK cells to respond to tumors in vivo

J. Orange, S. Kaech, Marisa A. P. Baptista, 2016, Scientific Reports.

Activation and Function of Mature B Cells The Rho GTPase Cdc42 Is Essential for the

R. Geha, M. Massaad, Marisa A. P. Baptista, 2015 .

N-WASP Is Essential for the Negative Regulation of B Cell Receptor Signaling

A. Upadhyaya, S. Snapper, Chaohong Liu, 2013, PLoS biology.

Functional Assessment for Clinical Use of Serum-Free Adapted NK-92 Cells

H. Nahi, E. Alici, V. Beljanski, 2019, Cancers.

Natural Killer Cell-Based Therapies Targeting Cancer: Possible Strategies to Gain and Sustain Anti-Tumor Activity

E. Alici, Carin I M Dahlberg, M. Chrobok, 2015, Front. Immunol..

FOXO Dictates Initiation of B Cell Development and Myeloid Restriction in Common Lymphoid Progenitors

P. Woll, H. Qian, Minghui He, 2022, Frontiers in Immunology.

ADP ribose is an endogenous ligand for the purinergic P2Y1 receptor

M. Islam, O. Chevallier, M. Migaud, 2011, Molecular and Cellular Endocrinology.

Foxp3 lacking exons 2 and 7 is unable to confer suppressive ability to regulatory T cells in vivo.

L. Westerberg, Anne-Laure Joly, Carin I M Dahlberg, 2015, Journal of autoimmunity.

Assessment of T Cell Receptor Complex Expression Kinetics in Natural Killer Cells

E. Alici, A. Hussain, Carin I M Dahlberg, 2022, Current issues in molecular biology.

HypermutationClass Switching and Reduced Somatic Mutation Leading to Complete Loss of Ig Activation-Induced Cytidine Deaminase Syndrome: A Spontaneous A Novel Mouse Model for the Hyper-IgM

R. Verdun, Minghui He, T. Visnes, 2014 .

compartment in mice (WASp) causes severe abnormalities of the peripheral B-cell intrinsic deficiency of the Wiskott-Aldrich syndrome protein - B cell

J. Hartwig, F. Frugoni, M. Massaad, 2012 .

Wiskott-Aldrich syndrome protein (WASP) and N-WASP are critical for peripheral B-cell development and function.

F. Alt, L. Notarangelo, S. Snapper, 2012, Blood.

Deletion of WASp and N-WASp in B cells cripples the germinal center response and results in production of IgM autoantibodies.

L. Notarangelo, J. Walter, S. Snapper, 2015, Journal of autoimmunity.

Constitutive activation of WASp in X-linked neutropenia renders neutrophils hyperactive

Minghui He, S. Snapper, D. Lane, 2018, The Journal of clinical investigation.

A Novel Mouse Model for the Hyper-IgM Syndrome: A Spontaneous Activation-Induced Cytidine Deaminase Mutation Leading to Complete Loss of Ig Class Switching and Reduced Somatic Hypermutation

R. Verdun, Minghui He, T. Visnes, 2014, The Journal of Immunology.

FOXO Dictates Initiation of B Cell Development and Myeloid Restriction in Common Lymphoid Progenitors

P. Woll, H. Qian, Minghui He, 2022, bioRxiv.

Deletion of Wiskott–Aldrich syndrome protein triggers Rac2 activity and increased cross-presentation by dendritic cells

S. Snapper, Marisa A. P. Baptista, A. Worth, 2016, Nature Communications.

N-WASP is required for B-cell-mediated autoimmunity in Wiskott-Aldrich syndrome.

L. Notarangelo, S. Snapper, E. Csizmadia, 2016, Blood.