L. Bresciani

NF‐κB pathway: a target for preventing β‐amyloid (Aβ)‐induced neuronal damage and Aβ42 production

G. Borsani, P. Spano, M. Pizzi, 2006, The European journal of neuroscience.

Alterations in presenilin 1 processing by amyloid-β peptide in the rat retina

L. Jen, S. Gentleman, V. Vince, 2007, Experimental Brain Research.

Amyloid-Beta Peptide Is Toxic to Neurons In Vivo via Indirect Mechanisms

L. Jen, S. Gentleman, P. Leclercq, 2002, Neurobiology of Disease.

Metabotropic glutamate receptors negatively coupled to adenylate cyclase inhibit N-methyl-D-aspartate receptor activity and prevent neurotoxicity in mesencephalic neurons in vitro.

S. Fracchia, G. Racagni, A. Ambrosini, 1995, Molecular pharmacology.

Gene expression profile activated by the chemokine CCL5/RANTES in human neuronal cells

A. Mantovani, P. Spano, M. Locati, 2004, Journal of neuroscience research.

Developmental Regulation and Possible Alternative Cleavage of Presenilin 1 in the Rat Retina

L. Jen, S. Gentleman, L. Bresciani, 2002, Molecular and Cellular Neuroscience.

Presenilin-1 is processed into two major cleavage products in neuronal cell lines.

J. Hardy, M. Hutton, M. Caivano, 1996, Neurodegeneration : a journal for neurodegenerative disorders, neuroprotection, and neuroregeneration.

Amyloid beta peptide causes chronic glial cell activation and neuro‐degeneration after intravitreal injection

L. Jen, S. Gentleman, L. Bresciani, 2005, Neuropathology and applied neurobiology.

Biological activity associated with a high molecular weight form of β-amyloid.

D. Allsop, E. Karran, K. H. Jennings, 1999 .