Raymond W. Zhou

发表

Pathogenesis of multiple sclerosis via environmental and genetic dysregulation of N-glycosylation

Michael Demetriou, C. Li, B. Newton, 2012, Seminars in Immunopathology.

Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants

Zhaoxia Yu, C. Li, B. Newton, 2013, Journal of Neuroimmunology.

Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis

A. Dell, S. Haslam, Gang Wu, 2016, eLife.

Age-associated impairment of T cell immunity is linked to sex-dimorphic elevation of N-glycan branching

P. L. Lee, J. Dennis, J. Pawling, 2022, Nature Aging.

N-glycosylation bidirectionally extends the boundaries of thymocyte positive selection by decoupling Lck from Ca2+ signaling

M. Demetriou, Raymond W. Zhou, A. Grigorian, 2014, Nature Immunology.

Genetics and the environment converge to dysregulate N-glycosylation in multiple sclerosis

J. Dennis, K. Siminovitch, K. Chandy, 2011, Nature communications.

Family studies of Type 1 diabetes reveal additive and epistatic effects between MGAT1 and three other polymorphisms

Zhaoxia Yu, C. Li, B. Newton, 2014, Genes and Immunity.

Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants

Zhaoxia Yu, C. Li, B. Newton, 2013, Journal of Neuroimmunology.