Ji Zhou

发表

Hyperhomocysteinemia induced by methionine supplementation does not independently cause atherosclerosis in C57BL/6J mice

Yuan-Yuan Shi, B. Trigatti, V. Tedesco, 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Polyphosphate-induced Thrombosis in Mice is Factor XII-Dependent and is Attenuated by Histidine-rich Glycoprotein.

J. Weitz, J. Crosby, A. Revenko, 2021, Blood advances.

Arterial thrombosis is accelerated in mice deficient in histidine-rich glycoprotein.

B. Monia, W. Jahnen-Dechent, J. Weitz, 2015, Blood.

Inferior Vena Cava Ligation Rapidly Induces Tissue Factor Expression and Venous Thrombosis in Rats

J. Weitz, P. Gross, Ji Zhou, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Activation of the Unfolded Protein Response Occurs at All Stages of Atherosclerotic Lesion Development in Apolipoprotein E–Deficient Mice

R. Austin, Ji Zhou, S. Lhoták, 2005, Circulation.

Immunohistochemical detection of the unfolded protein response in atherosclerotic plaques.

R. Austin, Ji Zhou, S. Lhoták, 2011, Methods in enzymology.

Dietary Supplementation With Methionine and Homocysteine Promotes Early Atherosclerosis but Not Plaque Rupture in ApoE-Deficient Mice

E. Falk, R. Austin, C. Danielsen, 2001, Arteriosclerosis, thrombosis, and vascular biology.

Association of Multiple Cellular Stress Pathways With Accelerated Atherosclerosis in Hyperhomocysteinemic Apolipoprotein E-Deficient Mice

E. Falk, G. Werstuck, M. Ritskes-Hoitinga, 2004, Circulation.

Macrophages are associated with lipid-rich carotid artery plaques, echolucency on B-mode imaging, and elevated plasma lipid levels.

Børge G Nordestgaard, Erling Falk, Henrik Sillesen, 2002, Journal of vascular surgery.

THROMBOSIS AND HEMOSTASIS Arterial thrombosis is accelerated in mice de ﬁ cient in histidine-rich glycoprotein

B. Monia, W. Jahnen-Dechent, J. Weitz, 2015 .

Arterial thrombosis is accelerated in histidine-rich glycoprotein deficient mice

B. Monia, W. Jahnen-Dechent, J. Weitz, 2015 .

Incidence and Predictors of Atrial Fibrillation After Aortic Repairs

S. Sengupta, Yuan-Yuan Shi, N. Maeda, 2004 .

Effects of vitamin supplementation and hyperhomocysteinemia on atherosclerosis in apoE-deficient mice.

E. Falk, M. L. Larsen, M. Ritskes-Hoitinga, 2003, Atherosclerosis.

Delayed but not Early Treatment with DNase Reduces Organ Damage and Improves Outcome in a Murine Model of Sepsis

D. Dwivedi, T. Gould, A. Fox-Robichaud, 2015, Shock.

Hyperhomocysteinemia and its role in the development of atherosclerosis.

G. Werstuck, R. Austin, Ji Zhou, 2003, Clinical biochemistry.

TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia*

E. Falk, R. Kaufman, D. Tang, 2003, Journal of Biological Chemistry.

Induction of the unfolded protein response after monocyte to macrophage differentiation augments cell survival in early atherosclerotic lesions

A. Ingram, Sana Basseri, R. Austin, 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Differential Expression of PCSK9 Modulates Infection, Inflammation, and Coagulation in a Murine Model of Sepsis

A. Prat, N. Seidah, D. Dwivedi, 2016, Shock.

Homocysteine and atherothrombosis

E. Falk, Ji Zhou, J. Møller, 2001, Lipids.

Contributions of hyperhomocysteinemia to atherosclerosis: Causal relationship and potential mechanisms

R. Austin, Ji Zhou, 2009, BioFactors.

Deficiency of TDAG51 Protects Against Atherosclerosis by Modulating Apoptosis, Cholesterol Efflux, and Peroxiredoxin‐1 Expression

J. Capone, M. Malloy, C. Pullinger, 2013, Journal of the American Heart Association.

Peroxynitrite Causes Endoplasmic Reticulum Stress and Apoptosis in Human Vascular Endothelium: Implications in Atherogenesis

R. Austin, J. Dickhout, G. S. Hossain, 2005, Arteriosclerosis, thrombosis, and vascular biology.

Thalidomide inhibits early atherogenesis in apoE-deficient mice.

E. Falk, A. Daugherty, T. Eriksson, 2003, APMIS. Supplementum.

Histidine-rich glycoprotein binds DNA and RNA and attenuates their capacity to activate the intrinsic coagulation pathway

J. Weitz, Trang T. Vu, B. Leslie, 2015, Thrombosis and Haemostasis.

Homocysteine-induced endoplasmic reticulum stress causes dysregulation of the cholesterol and triglyceride biosynthetic pathways.

Yuan-Yuan Shi, N. Maeda, G. Werstuck, 2001, The Journal of clinical investigation.

Effect of Different Doses of Acetylsalicylic Acid on the Antithrombotic Activity of Clopidogrel in a Mouse Arterial Thrombosis Model

J. Weitz, M. Cattaneo, P. Gross, 2018, Arteriosclerosis, thrombosis, and vascular biology.

Induction of the unfolded protein response after monocyte to macrophage differentiation augments cell survival in early atherosclerotic lesions

A. Ingram, Sana Basseri, R. Austin, 2011 .

Remodelling of the zero-stress state and residual strains in apoE-deficient mouse aorta.

E. Falk, H. Gregersen, Jingbo Zhao, 2007, Biorheology.

Induction of the unfolded protein response after monocyte to macrophage differentiation augments cell survival in early atherosclerotic lesions

A. Ingram, Sana Basseri, R. Austin, 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Homocysteine-induced endoplasmic reticulum stress causes dysregulation of the cholesterol and triglyceride biosynthetic pathways.

Yuan-Yuan Shi, N. Maeda, G. Werstuck, 2001, The Journal of clinical investigation.

Histidine-rich glycoprotein attenuates catheter thrombosis

J. Weitz, A. Revenko, Ji Zhou, 2023, Blood advances.