J. Bai

发表

Adenovirus mediated overexpression of CYP2E1 increases sensitivity of HepG2 cells to acetaminophen induced cytotoxicity

A. Cederbaum, J. Bai, Jingxiang Bai, 2004, Molecular and Cellular Biochemistry.

Cycloheximide Protects HepG 2 Cells from Serum Withdrawal-Induced Apoptosis by Decreasing p 53 and Phosphorylated p 53 Levels

A. Cederbaum, J. Bai, Jingxiang Bai, 2006 .

Cycloheximide Protects HepG2 Cells from Serum Withdrawal-Induced Apoptosis by Decreasing p53 and Phosphorylated p53 Levels

A. Cederbaum, J. Bai, Jingxiang Bai, 2006, Journal of Pharmacology and Experimental Therapeutics.

Catalase and estradiol inhibit mitochondrial protein S-glutathionylation

J. Odin, Bin Hu, J. Bai, 2012, Molecular and Cellular Biochemistry.

Adenovirus-mediated expression of CYP2E1 produces liver toxicity in mice.

A. Cederbaum, J. Bai, Jingxiang Bai, 2006, Toxicological sciences : an official journal of the Society of Toxicology.

Overexpression of CYP2E1 in Mitochondria Sensitizes HepG2 Cells to the Toxicity Caused by Depletion of Glutathione*

A. Cederbaum, J. Bai, Jingxiang Bai, 2006, Journal of Biological Chemistry.

Adenovirus-mediated overexpression of catalase in the cytosolic or mitochondrial compartment protects against toxicity caused by glutathione depletion in HepG2 cells expressing CYP2E1.

A. Cederbaum, M. Marí, J. Bai, 2002, The Journal of pharmacology and experimental therapeutics.

Mitochondrial Catalase and Oxidative Injury

A. Cederbaum, J. Bai, Jingxiang Bai, 2001, Neurosignals.

Polo-like kinase 3 functions as a tumor suppressor and is a negative regulator of hypoxia-inducible factor-1 alpha under hypoxic conditions.

W. Dai, Ying Huang, Rulong Z. Shen, 2008, Cancer research.

Apoptosis and the liver: relation to autoimmunity and related conditions.

J. Odin, J. Bai, 2003, Autoimmunity reviews.

Cytochrome P450 2E1 contributes to ethanol‐induced fatty liver in mice

A. Cederbaum, Xiaodong Wang, J. Zhuge, 2008, Hepatology.

Toxicity by pyruvate in HepG2 cells depleted of glutathione: role of mitochondria.

A. Cederbaum, M. Marí, J. Bai, 2002, Free radical biology & medicine.

A genome-wide screen in Saccharomyces cerevisiae reveals pathways affected by arsenic toxicity.

Wei Dai, John P Rooney, Thomas J. Begley, 2009, Genomics.

CYP2E1-dependent toxicity and oxidative stress in HepG2 cells.

Defeng Wu, A. Cederbaum, M. Marí, 2001, Free radical biology & medicine.

Adenovirus-mediated Overexpression of Catalase in the Cytosolic or Mitochondrial Compartment Protects against Cytochrome P450 2E1-dependent Toxicity in HepG2 Cells*

A. Cederbaum, J. Bai, Jingxiang Bai, 2001, The Journal of Biological Chemistry.

Overexpression of Catalase in the Mitochondrial or Cytosolic Compartment Increases Sensitivity of HepG2 Cells to Tumor Necrosis Factor-α-induced Apoptosis*

A. Cederbaum, J. Bai, Jingxiang Bai, 2000, The Journal of Biological Chemistry.

Overexpression of Catalase in Cytosolic or Mitochondrial Compartment Protects HepG2 Cells against Oxidative Injury*

J. Melendez, A. Cederbaum, Ana M. Rodriguez, 1999, The Journal of Biological Chemistry.

Catalase Protects HepG2 Cells from Apoptosis Induced by DNA-damaging Agents by Accelerating the Degradation of p53*

A. Cederbaum, J. Bai, Jingxiang Bai, 2003, The Journal of Biological Chemistry.

A genome-wide deletion mutant screen identifies pathways affected by nickel sulfate in Saccharomyces cerevisiae

Thomas J. Begley, T. Begley, W. Dai, 2009, BMC Genomics.

Covalent modifications of histones during mitosis and meiosis

W. Dai, Dazhong Xu, Max Costa, 2009, Cell cycle.

Catalase and estradiol inhibit mitochondrial protein S-glutathionylation

J. Odin, Bin Hu, J. Bai, 2012, Molecular and Cellular Biochemistry.