Jill A. Rafael-Fortney

D. Guttridge, C. Gomez-Sanchez, E. Gomez-Sanchez, 2015, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Claudin-5 localizes to the lateral membranes of cardiomyocytes and is altered in utrophin/dystrophin-deficient cardiomyopathic mice.

B. Gong, J. Rafael-Fortney, A. Merriam, 2005, Journal of molecular and cellular cardiology.

Improvement of cardiac contractile function by peptide-based inhibition of NF-κB in the utrophin/dystrophin-deficient murine model of muscular dystrophy

D. Guttridge, J. Rafael-Fortney, Dawn A. Delfín, 2011, Journal of Translational Medicine.

Peptide-Based Inhibition of NF-κB Rescues Diaphragm Muscle Contractile Dysfunction in a Murine Model of Duchenne Muscular Dystrophy

Michael A. Freitas, J. Mendell, P. Robbins, 2011, Molecular medicine.

Utrophin deficiency worsens cardiac contractile dysfunction present in dystrophin-deficient mdx mice.

J. Rafael-Fortney, P. Janssen, Jill A. Rafael-Fortney, 2005, American journal of physiology. Heart and circulatory physiology.

Mineralocorticoid receptor antagonism by finerenone is sufficient to improve function in preclinical muscular dystrophy

S. Raman, Jeovanna Lowe, J. Rafael-Fortney, 2020, ESC heart failure.

Metabolic Dysfunction and Altered Mitochondrial Dynamics in the Utrophin-Dystrophin Deficient Mouse Model of Duchenne Muscular Dystrophy

Jeovanna Lowe, M. Periasamy, J. Rafael-Fortney, 2015, PloS one.

CASK and Dlg form a PDZ protein complex at the mammalian neuromuscular junction

J. Rafael-Fortney, Jill A. Rafael-Fortney, Tessily A Mays, 2004, Muscle & nerve.

Eplerenone for early cardiomyopathy in Duchenne muscular dystrophy: a randomised, double-blind, placebo-controlled trial

S. Raman, J. Kissel, L. Cripe, 2015, The Lancet Neurology.

Muscle Twitch Kinetics Are Dependent on Muscle Group, Disease State, and Age in Duchenne Muscular Dystrophy Mouse Models

Jonathan P. Davis, Jeovanna Lowe, J. Rafael-Fortney, 2020, Frontiers in Physiology.

Micro-dystrophin gene therapy demonstrates long-term cardiac efficacy in a severe Duchenne muscular dystrophy model

J. Chamberlain, Jeovanna Lowe, J. Rafael-Fortney, 2023, Molecular therapy. Methods & clinical development.

Mineralocorticoid Receptor Antagonists in Muscular Dystrophy Mice During Aging and Exercise.

C. Gomez-Sanchez, E. Gomez-Sanchez, Jeovanna Lowe, 2018, Journal of neuromuscular diseases.

Renin-angiotensin-aldosterone system inhibitors improve membrane stability and change gene-expression profiles in dystrophic skeletal muscles.

S. Bhattacharya, N. Weisleder, Jeovanna Lowe, 2017, American journal of physiology. Cell physiology.

Duchenne Muscular Dystrophy Mice and Men: Can Understanding a Genetic Cardiomyopathy Inform Treatment of Other Myocardial Diseases?

S. Raman, J. Rafael-Fortney, Jill A. Rafael-Fortney, 2016, Circulation research.

Standard Operating Procedures (SOPs) for Evaluating the Heart in Preclinical Studies of Duchenne Muscular Dystrophy

D. Kass, K. Kinnett, D. Duan, 2016, Journal of Cardiovascular Translational Research.

Prednisolone Attenuates Improvement of Cardiac and Skeletal Contractile Function and Histopathology by Lisinopril and Spironolactone in the mdx Mouse Model of Duchenne Muscular Dystrophy

S. Raman, T. Tran, K. E. Schill, 2014, PloS one.

Myeloid cells are capable of synthesizing aldosterone to exacerbate damage in muscular dystrophy.

C. Gomez-Sanchez, Steven S. Welc, E. Gomez-Sanchez, 2016, Human molecular genetics.

Mineralocorticoid receptor antagonists and glucocorticoids differentially affect skeletal muscle inflammation and pathology in muscular dystrophy

Jeovanna Lowe, J. Rafael-Fortney, S. Bansal, 2022, JCI insight.

Early Inflammation in Muscular Dystrophy Differs Between Limb and Respiratory Muscles and Increases with Dystrophic Severity.

Jeovanna Lowe, J. Rafael-Fortney, S. Bansal, 2021, The American journal of pathology.

Lengthening-contractions in isolated myocardium impact force development and worsen cardiac contractile function in the mdx mouse model of muscular dystrophy.

J. Rafael-Fortney, Dawn A. Delfín, P. Janssen, 2011, Journal of applied physiology.

Claudin-5 levels are reduced in human end-stage cardiomyopathy.

Amit A. Doshi, P. Binkley, K. Margulies, 2008, Journal of molecular and cellular cardiology.

The force-temperature relationship in healthy and dystrophic mouse diaphragm; implications for translational study design

J. Rafael-Fortney, N. Rastogi, P. Janssen, 2012, Front. Physio..

Rationally engineered Troponin C modulates in vivo cardiac function and performance in health and disease

Jonathan P. Davis, Bo Zhang, V. Fedorov, 2016, Nature Communications.

Early treatment with lisinopril and spironolactone preserves cardiac and skeletal muscle in Duchenne muscular dystrophy mice.

S. Raman, Dawn A. Delfín, Benjamin D. Canan, 2011, Circulation.

Peptide-Based Inhibition of NF-κB Rescues Diaphragm Muscle Contractile Dysfunction in a Murine Model of Duchenne Muscular Dystrophy

Michael A. Freitas, J. Mendell, P. Robbins, 2011, Molecular medicine.

Cardiomyopathy in the dystrophin/utrophin-deficient mouse model of severe muscular dystrophy is characterized by dysregulation of matrix metalloproteinases

S. Raman, J. Rafael-Fortney, Dawn A. Delfín, 2012, Neuromuscular Disorders.

Sustaining cardiac claudin-5 levels prevents functional hallmarks of cardiomyopathy in a muscular dystrophy mouse model.

K. E. Schill, J. Rafael-Fortney, Dawn A. Delfín, 2012, Molecular therapy : the journal of the American Society of Gene Therapy.

Early treatment with lisinopril and spironolactone preserves cardiac and skeletal muscle in Duchenne muscular dystrophy mice.

S. Raman, T. Tran, J. Rafael-Fortney, 2011, Circulation.

Renin-angiotensin-aldosterone system inhibitors 3 improve membrane stability and change gene 4 expression profiles in dystrophic skeletal muscles

Jeovanna Lowe, Jill A. Rafael-Fortney, Jessica Chadwick, 2016 .

Claudin-5 levels are reduced from multiple cell types in human failing hearts and are associated with mislocalization of ephrin-B1.

V. Fedorov, P. Mohler, A. Kilic, 2015, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.

Somatic gene therapy to treat heart failure is one step closer to reality.

M. Periasamy, Jill A. Rafael-Fortney, 2009, Cardiovascular research.

Impairment of Diastolic Function by Lack of Frequency-Dependent Myofilament Desensitizationin Rabbit Right Ventricular Hypertrophy

R. Hamlin, Subash C. Gupta, M. Periasamy, 2009, Circulation. Heart failure.

Troponin I Tyrosine Phosphorylation Beneficially Accelerates Diastolic Function

T. McKinsey, N. Parinandi, Jeovanna Lowe, 2023, Circulation research.

The Angiotensin Converting Enzyme Inhibitor Lisinopril Improves Muscle Histopathology but not Contractile Function in a Mouse Model of Duchenne Muscular Dystrophy

S. Raman, T. Tran, Jeovanna Lowe, 2015, Journal of neuromuscular diseases.

Mineralocorticoid Receptor Signaling in the Inflammatory Skeletal Muscle Microenvironments of Muscular Dystrophy and Acute Injury

J. Rafael-Fortney, Z. Howard, Jill A. Rafael-Fortney, 2022, Frontiers in Pharmacology.

Gene expression effects of glucocorticoid and mineralocorticoid receptor agonists and antagonists on normal human skeletal muscle.

C. Gomez-Sanchez, E. Gomez-Sanchez, J. Rafael-Fortney, 2017, Physiological genomics.

Glutamate receptors localize postsynaptically at neuromuscular junctions in mice

A. Chishti, T. Hanada, J. Rafael-Fortney, 2009, Muscle & nerve.

Mineralocorticoid Receptor Signaling Contributes to Normal Muscle Repair After Acute Injury

C. Gomez-Sanchez, E. Gomez-Sanchez, Jeovanna Lowe, 2019, Front. Physiol..

Duchenne Muscular Dystrophy Mice and Men: Can Understanding a Genetic Cardiomyopathy Inform Treatment of Other Myocardial Diseases?

S. Raman, Jill A. Rafael-Fortney, Jessica Chadwick, 2016, Circulation research.