S. Balakrishnan

发表

Analytical and clinical validation of a microbial cell-free DNA sequencing test for infectious disease

Martin S. Lindner, Sivan Bercovici, Samuel Yang, 2019, Nature Microbiology.

Epidermal growth factor stimulates translocation of the class II phosphoinositide 3-kinase PI3K-C2 (cid:12) to the nucleus

Simona, Y. Korchev, H. Banfić, 2018 .

Epidermal growth factor stimulates translocation of the class II phosphoinositide 3-kinase PI3K-C2beta to the nucleus.

Y. Korchev, H. Banfić, D. Visnjic, 2009, The Biochemical journal.

PIM kinase inhibition presents a novel targeted therapy against triple-negative breast tumors with elevated MYC expression

Michael T. McManus, Lindsey A. Marsh, C. Yau, 2016 .

Identification of Chemical Inhibitors of β-Catenin-Driven Liver Tumorigenesis in Zebrafish

S. Kakar, D. Stainier, K. Evason, 2015, PLoS genetics.

The oncofetal gene survivin is re-expressed in osteoarthritis and is required for chondrocyte proliferation in vitro

P. Lechler, S. Grässel, R. Straub, 2011, BMC musculoskeletal disorders.

Inhibition of fatty acid oxidation as a therapy for MYC-overexpressing triple-negative breast cancer

Brittany Anderton, Gregor Krings, Sanjeev Balakrishnan, 2016, Nature Medicine.

Expanding the CRISPR imaging toolset with Staphylococcus aureus Cas9 for simultaneous imaging of multiple genomic loci

Wendell A. Lim, Bo Huang, Sanjeev Balakrishnan, 2016, Nucleic acids research.

Single-cell analysis reveals a stem-cell program in human metastatic breast cancer cells

Chih-Yang Wang, Alicia Zhou, Sanjeev Balakrishnan, 2015, Nature.

PIM1 kinase inhibition as a targeted therapy against triple-negative breast tumors with elevated MYC expression

Michael T. McManus, Lindsey A. Marsh, C. Yau, 2016, Nature Medicine.

MYC Dysregulates Mitosis, Revealing Cancer Vulnerabilities.

Aaron N. Chang, H. Rugo, S. Dumont, 2020, Cell reports.

The Oncofetal Gene Survivin Promotes Cell Proliferation and Survival in Primary Human Osteoblastic Cells

P. Lechler, L. Prantl, J. Grifka, 2011, Calcified Tissue International.

The antiapoptotic gene survivin is highly expressed in human chondrosarcoma and promotes drug resistance in chondrosarcoma cells in vitro

P. Lechler, M. Tingart, T. Renkawitz, 2011, BMC Cancer.

MYC‐driven inhibition of the glutamate‐cysteine ligase promotes glutathione depletion in liver cancer

A. Balakrishnan, Guowang Xu, K. Evason, 2017, EMBO reports.

MYC Dysregulates Mitotic Spindle Function Creating a Dependency on TPX2

Aaron N. Chang, A. Goga, Julia Rohrberg, 2018, bioRxiv.

Analytical and clinical validation of a microbial cell-free DNA sequencing test for infectious disease

T. Blauwkamp, F. Christians, David K. Hong, 2019, Nature Microbiology.

Requirement for Class II Phosphoinositide 3-Kinase C2α in Maintenance of Glomerular Structure and Function

D. Powell, K. Moberg, N. Xu, 2010, Molecular and Cellular Biology.

The C. elegans Rab Family: Identification, Classification and Toolkit Construction

Puja Patel, Nilang Gor, Danielle Jorgens, 2012, PloS one.

Epidermal growth factor stimulates translocation of the class II phosphoinositide 3-kinase PI3K-C2 (cid:12) to the nucleus

Simona, Y. Korchev, H. Banfić, 2020 .

Epidermal growth factor stimulates translocation of the class II phosphoinositide 3-kinase PI3K-C2 (cid:12) to the nucleus

Y. Korchev, H. Banfić, D. Visnjic, 2019 .

Inhibition of fatty acid oxidation as a therapy for MYC-overexpressing triple-negative breast cancer

A. Tward, S. Kajimura, D. Nomura, 2016, Nature Medicine.

PIM1 kinase inhibition as a targeted therapy against triple-negative breast tumors with elevated MYC expression

Michael T. McManus, Lindsey A. Marsh, C. Yau, 2016, Nature Medicine.

S100A8/A9 predicts response to PIM kinase and PD-1/PD-L1 inhibition in triple-negative breast cancer mouse models

Roman Camarda, S. Balakrishnan, Dai Horiuchi, 2024, Communications medicine.

S100A8/A9 predicts triple-negative breast cancer response to PIM kinase and PD-1/PD-L1 inhibition

Deepak Kanojia, Roman Camarda, S. Balakrishnan, 2023, bioRxiv.