N. Beatini

Human Vascular Endothelial Cells Are a Rich and Regulatable Source of Secretory Sphingomyelinase

K. Williams, I. Tabas, S. Schissel, 1998, The Journal of Biological Chemistry.

Evidence That the Initial Up-regulation of Phosphatidylcholine Biosynthesis in Free Cholesterol-loaded Macrophages Is an Adaptive Response That Prevents Cholesterol-induced Cellular Necrosis

I. Tabas, N. Beatini, G. Keesler, 1996, The Journal of Biological Chemistry.

Effect and cellular site of action of cysteine protease inhibitors on the cholesterol esterification pathway in macrophages and Chinese hamster ovary cells.

F. Maxfield, X. Zha, I. Tabas, 1995, Biochemistry.

Unique Cellular Events Occurring during the Initial Interaction of Macrophages with Matrix-retained or Methylated Aggregated Low Density Lipoprotein (LDL)

F. Maxfield, H. Du, R. Ghosh, 1999, The Journal of Biological Chemistry.

Immunolocalization of Acyl-Coenzyme A:CholesterolO-Acyltransferase in Macrophages*

Robert V Farese, Hong Wang, F. Maxfield, 1998, The Journal of Biological Chemistry.

The reactivity of desmosterol and other shellfish- and xanthomatosis-associated sterols in the macrophage sterol esterification reaction.

I. Tabas, S. Feinmark, N. Beatini, 1989, The Journal of clinical investigation.

Identification and characterization of an acyl-CoA:triterpene acyltransferase activity in rabbit and human tissues.

J. Clader, I. Tabas, L. L. Chen, 1991, Journal of lipid research.

Enrichment of acyl coenzyme A:cholesterol O-acyltransferase near trans-golgi network and endocytic recycling compartment.

F. Maxfield, O. Quehenberger, T. T. Soe, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Acute systemic inflammation up-regulates secretory sphingomyelinase in vivo: a possible link between inflammatory cytokines and atherogenesis.

M. Wong, J. Licinio, K. Williams, 2000, Proceedings of the National Academy of Sciences of the United States of America.

The actin cytoskeleton is important for the stimulation of cholesterol esterification by atherogenic lipoproteins in macrophages.

F. Maxfield, J. Myers, X. Zha, 1994, The Journal of biological chemistry.