L. Salvadó

发表

TNF-α inhibits PPARβ/δ activity and SIRT1 expression through NF-κB in human adipocytes.

F. Tinahones, M. Wabitsch, J. Vendrell, 2012, Biochimica et biophysica acta.

Enhanced fatty acid oxidation in adipocytes and macrophages reduces lipid-induced triglyceride accumulation and inflammation.

F. Villarroya, N. Casals, D. Serra, 2015, American journal of physiology. Endocrinology and metabolism.

Tau hyperphosphorylation and increased BACE1 and RAGE levels in the cortex of PPARβ/δ-null mice.

W. Wahli, J. del Valle, M. Pallàs, 2013, Biochimica et biophysica acta.

PPARβ/δ ameliorates fructose-induced insulin resistance in adipocytes by preventing Nrf2 activation.

W. Wahli, J. Vendrell, E. Barroso, 2015, Biochimica et biophysica acta.

PPARβ/δamelioratesfructose-inducedinsulinresistanceinadipocytesby preventing Nrf2 activation

W. Wahli, J. Vendrell, E. Barroso, 2015 .

Targeting endoplasmic reticulum stress in insulin resistance

E. Barroso, X. Palomer, M. Vázquez-Carrera, 2015, Trends in Endocrinology & Metabolism.

Targeting PPARβ/δ for the treatment of type 2 diabetes mellitus

E. Barroso, X. Palomer, M. Vázquez-Carrera, 2012, Expert opinion on therapeutic targets.

Targeting PPARb/d for the treatment of type 2 diabetes mellitus

E. Barroso, X. Palomer, M. Vázquez-Carrera, 2012 .

Oleate prevents saturated-fatty-acid-induced ER stress, inflammation and insulin resistance in skeletal muscle cells through an AMPK-dependent mechanism

A. Gómez-Foix, E. Barroso, X. Palomer, 2013, Diabetologia.

An overview of the crosstalk between inflammatory processes and metabolic dysregulation during diabetic cardiomyopathy.

E. Barroso, X. Palomer, M. Vázquez-Carrera, 2013, International journal of cardiology.

PPARβ/δ attenuates palmitate-induced endoplasmic reticulum stress and induces autophagic markers in human cardiac cells.

W. Wahli, L. Michalik, E. Barroso, 2014, International journal of cardiology.

Corrigendum to "The BACE1 product sAPPβ induces ER stress and inflammation and impairs insulin signaling" [Metab Clin Exp 85 (2018) 59-75].

D. Serra, J. Vendrell, J. Pizarro, 2019, Metabolism: Clinical and Experimental.

The BACE1 product sAPPβ induces ER stress and inflammation and impairs insulin signaling.

D. Serra, J. Vendrell, J. Pizarro, 2018, Metabolism: clinical and experimental.

Peroxisome Proliferator-Activated Receptor β/δ (PPAR β/δ) as a Potential Therapeutic Target for Dyslipidemia

E. Barroso, X. Palomer, M. Vázquez-Carrera, 2012 .

PPARβ/δ prevents endoplasmic reticulum stress-associated inflammation and insulin resistance in skeletal muscle cells through an AMPK-dependent mechanism

W. Wahli, L. Michalik, A. Gómez-Foix, 2014, Diabetologia.

The Role of Peroxisome Proliferator-Activated Receptor β/δ on the Inflammatory Basis of Metabolic Disease

E. Barroso, X. Palomer, M. Vázquez-Carrera, 2010, PPAR research.