J. Maddox

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Metabonomic evaluation of idiosyncrasy-like liver injury in rats cotreated with ranitidine and lipopolysaccharide.

Royston Goodacre, George G Harrigan, R. Goodacre, 2006, Toxicology and applied pharmacology.

Modeling inflammation-drug interactions in vitro: a rat Kupffer cell-hepatocyte coculture system.

P. Ganey, R. Roth, D. Amacher, 2006, Toxicology in vitro : an international journal published in association with BIBRA.

Coagulation-Mediated Hypoxia and Neutrophil-Dependent Hepatic Injury in Rats Given Lipopolysaccharide and Ranitidine

J. Luyendyk, P. Ganey, R. Roth, 2005, Journal of Pharmacology and Experimental Therapeutics.

Role of hepatic fibrin in idiosyncrasy‐like liver injury from lipopolysaccharide‐ranitidine coexposure in rats

J. Luyendyk, P. Ganey, R. Roth, 2004, Hepatology.

Modes of cell death in rat liver after monocrotaline exposure.

C. Rondelli, B. Copple, P. Ganey, 2003, Toxicological sciences : an official journal of the Society of Toxicology.

Role of the coagulation system in acetaminophen‐induced hepatotoxicity in mice

J. Luyendyk, N. Mackman, P. Ganey, 2007, Hepatology.

Aspirin-triggered 15-Epi-Lipoxin A4 (LXA4) and LXA4 Stable Analogues Are Potent Inhibitors of Acute Inflammation: Evidence for Anti-inflammatory Receptors

T. Takano, C. Serhan, N. Petasis, 1997, The Journal of experimental medicine.

Lipoxin A4 and B4 are potent stimuli for human monocyte migration and adhesion: selective inactivation by dehydrogenation and reduction

C. Serhan, J. Maddox, Jane E Maddox, 1996, The Journal of experimental medicine.

Lipoxin A4 Stable Analogs Are Potent Mimetics That Stimulate Human Monocytes and THP-1 Cells via a G-protein-linked Lipoxin A4 Receptor*

T. Takano, C. Serhan, N. Petasis, 1997, The Journal of Biological Chemistry.

Design of lipoxin A4 stable analogs that block transmigration and adhesion of human neutrophils.

I. Akritopoulou‐Zanze, C. Serhan, N. Petasis, 1995, Biochemistry.

Activation of human monocytes and the acute monocytic leukemia cell line (THP-1) by lipoxins involves unique signaling pathways for lipoxin A4 versus lipoxin B4: evidence for differential Ca2+ mobilization.

C. Serhan, J. Maddox, M. Romano, 1996, Journal of immunology.

Identification of a human cDNA encoding a functional high affinity lipoxin A4 receptor

C. Serhan, H. Perez, J. Maddox, 1994, The Journal of experimental medicine.

Lipoxin B4 regulates human monocyte/neutrophil adherence and motility: design of stable lipoxin B4 analogs with increased biologic activity

C. Clish, C. Serhan, N. Petasis, 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Bacterial- and Viral-Induced Inflammation Increases Sensitivity to Acetaminophen Hepatotoxicity

C. Cuff, P. Ganey, J. Pestka, 2009, Journal of toxicology and environmental health. Part A.

The Role of Hypoxia-Inducible Factor-1 (cid:1) in Acetaminophen Hepatotoxicity

B. Copple, J. Luyendyk, P. Ganey, 2011 .

Lipoxin a 4 Stable Analogs Are Potent Mimetics That Stimulate Human Monocytes and THP-1 Cells via a G-protein-linked Lipoxin a 4 Receptor

T. Takano, C. Serhan, N. Petasis, 1997 .

Unique gene expression and hepatocellular injury in the lipopolysaccharide-ranitidine drug idiosyncrasy rat model: comparison with famotidine.

J. Luyendyk, P. Ganey, B. Car, 2006, Toxicological sciences : an official journal of the Society of Toxicology.

Dietary selenium effects on milk eicosanoid concentration in dairy cows during coliform mastitis.

C. C. Reddy, R. W. Scholz, J. Maddox, 1991, Prostaglandins.

Effects of selenium status on bovine mononuclear cell function.

L. Sordillo, J. Maddox, C. R. Hicks, 1993, Zentralblatt fur Veterinarmedizin. Reihe A.

15-deoxy prostaglandin J2 enhances allyl alcohol-induced toxicity in rat hepatocytes.

P. Ganey, R. Roth, Alison Domzalski, 2004, Toxicological sciences : an official journal of the Society of Toxicology.

Ranitidine Treatment during a Modest Inflammatory Response Precipitates Idiosyncrasy-Like Liver Injury in Rats

J. Luyendyk, P. Ganey, R. Roth, 2003, Journal of Pharmacology and Experimental Therapeutics.

Increased neutrophil adherence and adhesion molecule mRNA expression in endothelial cells during selenium deficiency

L. Sordillo, C. C. Reddy, J. Maddox, 1999, Journal of leukocyte biology.

Selenium deficiency alters the lipoxygenase pathway and mitogenic response in bovine lymphocytes.

A. Mastro, C. C. Reddy, R. W. Scholz, 1992, The Journal of nutrition.

Aspirin-triggered 15-epi-lipoxin A4 and stable analogs on lipoxin A4 are potent inhibitors of acute inflammation. Receptors and pathways.

T. Takano, C. Serhan, J. Maddox, 1999, Advances in experimental medicine and biology.

Adverse hepatic drug reactions: inflammatory episodes as consequence and contributor.

J. Luyendyk, P. Ganey, R. Roth, 2004, Chemico-biological interactions.

Acute exposure to ozone exacerbates acetaminophen-induced liver injury in mice.

P. Ganey, R. Roth, J. Harkema, 2010, Toxicological sciences : an official journal of the Society of Toxicology.

The Role of Hypoxia-Inducible Factor-1α in Acetaminophen Hepatotoxicity

B. Copple, J. Luyendyk, P. Ganey, 2011, Journal of Pharmacology and Experimental Therapeutics.

Modest Inflammation Enhances Diclofenac Hepatotoxicity in Rats: Role of Neutrophils and Bacterial Translocation

Michael J Liguori, Eric A G Blomme, J. Waring, 2006, Journal of Pharmacology and Experimental Therapeutics.

Coagulation-Dependent Gene Expression and Liver Injury in Rats Given Lipopolysaccharide with Ranitidine but Not with Famotidine

Lois D Lehman-McKeeman, J. Luyendyk, P. Ganey, 2006, Journal of Pharmacology and Experimental Therapeutics.

Application of high-throughput Fourier-transform infrared spectroscopy in toxicology studies: contribution to a study on the development of an animal model for idiosyncratic toxicity.

Royston Goodacre, George G Harrigan, R. Goodacre, 2004, Toxicology letters.

Increased 15-HPETE production decreases prostacyclin synthase activity during oxidant stress in aortic endothelial cells.

L. Sordillo, J. Maddox, I. K. Mullarky, 2001, Free radical biology & medicine.

Inflammation and Drug Idiosyncrasy—Is There a Connection?

J. Luyendyk, P. Ganey, R. Roth, 2003, Journal of Pharmacology and Experimental Therapeutics.

Comparison of TNFα to Lipopolysaccharide as an Inflammagen to Characterize the Idiosyncratic Hepatotoxicity Potential of Drugs: Trovafloxacin as an Example

J. Waring, J. Luyendyk, E. Blomme, 2010, International journal of molecular sciences.

Microarray Analysis of Lipopolysaccharide Potentiation of Trovafloxacin-Induced Liver Injury in Rats Suggests a Role for Proinflammatory Chemokines and Neutrophils

J. Waring, J. Luyendyk, E. Blomme, 2006, Journal of Pharmacology and Experimental Therapeutics.

Increased 12-HETE production in bovine lymphocytes during selenium deficiency.

A. Mastro, C. C. Reddy, R. W. Scholz, 1991, Biochemical and biophysical research communications.

Gene expression analysis points to hemostasis in livers of rats cotreated with lipopolysaccharide and ranitidine.

Lyle D Burgoon, Timothy R Zacharewski, William B Mattes, 2004, Toxicological sciences : an official journal of the Society of Toxicology.