B. Lawrence

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A Birth Cohort Study of Maternal and Infant Serum PCB-153 and DDE Concentrations and Responses to Infant Tuberculosis Vaccination

I. Hertz-Picciotto, S. Schwartz, T. Jusko, 2015, Environmental health perspectives.

Conditional deletion of Ahr alters gene expression profiles in hematopoietic stem cells

S. Welle, T. Gasiewicz, B. Lawrence, 2018, PloS one.

Increased mortality associated with TCDD exposure in mice infected with influenza A virus is not due to severity of lung injury or alterations in Clara cell protein content.

K. Harrod, B. Lawrence, A. Bohn, 2005, Chemico-biological interactions.

Developmental Exposure to the Potent Aryl Hydrocarbon Receptor Agonist 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Impairs the Cell-Mediated Immune Response to Infection with Influenza A Virus, but Enhances Elements of Innate Immunity

Jennifer A Cundiff, B. Lawrence, B. Vorderstrasse, 2004, Journal of immunotoxicology.

Genome-Wide Transcriptional Analysis Reveals Novel AhR Targets That Regulate Dendritic Cell Function during Influenza A Virus Infection

Jason R. Myers, J. Myers, D. Shepherd, 2019, ImmunoHorizons.

Early life exposures shape the CD4+ T cell transcriptome, influencing proliferation, differentiation, and mitochondrial dynamics later in life

Jason R. Myers, P. Brookes, J. Myers, 2019, Scientific Reports.

Influence of Early-Life Environmental Exposures on Immune Function Across the Life Span

B. Lawrence, L. Boulé, Lisbeth A. Boulé, 2016 .

Activation of the aryl hydrocarbon receptor during development enhances the pulmonary CD4+ T-cell response to viral infection.

M. Pavelka, D. Topham, Bethany N Winans, 2015, American journal of physiology. Lung cellular and molecular physiology.

New insights into the aryl hydrocarbon receptor as a modulator of host responses to infection

B. Lawrence, B. Vorderstrasse, B. A. Vorderstrasse, 2013, Seminars in Immunopathology.

Maternal and early postnatal polychlorinated biphenyl exposure in relation to total serum immunoglobulin concentrations in 6-month-old infants

I. Hertz-Picciotto, S. Schwartz, T. Jusko, 2011, Journal of immunotoxicology.

The aryl hydrocarbon receptor affects distinct tissue compartments during ontogeny of the immune system.

Amanda J. Moore, B. Lawrence, J. Hogaboam, 2008, Toxicological sciences : an official journal of the Society of Toxicology.

Exacerbation of Lung Radiation Injury by Viral Infection: The Role of Clara Cells and Clara Cell Secretory Protein

Jacqueline P. Williams, J. Finkelstein, C. Johnston, 2013, Radiation research.

Negative effects of low dose atrazine exposure on the development of effective immunity to FV3 in Xenopus laevis.

J. Robert, L. Grayfer, B. Lawrence, 2014, Developmental and comparative immunology.

Aryl hydrocarbon receptor activation during pregnancy, and in adult nulliparous mice, delays the subsequent development of DMBA‐induced mammary tumors

V. Arlt, D. Medina, S. Fenton, 2011, International journal of cancer.

Neonatal hyperoxia alters the host response to influenza A virus infection in adult mice through multiple pathways.

M. O’Reilly, Min Yee, B. Lawrence, 2013, American journal of physiology. Lung cellular and molecular physiology.

Activation of the aryl hydrocarbon receptor is essential for mediating the anti-inflammatory effects of a novel low-molecular-weight compound.

M. Denison, M. Woisetschläger, N. Harrer, 2008, Blood.

Developmental Activation of the AHR Increases Effector CD4+ T Cells and Exacerbates Symptoms in Autoimmune Disease-Prone Gnaq+/- Mice.

B. Fenton, T. Jusko, K. Thevenet‐Morrison, 2015, Toxicological sciences : an official journal of the Society of Toxicology.

The Ancestral Environment Shapes Antiviral CD8+ T cell Responses across Generations

Bethany N Winans, B. Lawrence, L. Boulé, 2019, iScience.

Neonatal hyperoxia enhances the inflammatory response in adult mice infected with influenza A virus.

M. O’Reilly, S. McGrath-Morrow, Min Yee, 2008, American journal of respiratory and critical care medicine.

Role of glutathione and reactive oxygen intermediates in 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced immune suppression in C57Bl/6 mice.

N. Kerkvliet, D. J. Reed, B. Lawrence, 1999, Toxicological sciences : an official journal of the Society of Toxicology.

Exposure to a mixture of 23 chemicals associated with unconventional oil and gas operations alters immune response to challenge in adult mice

S. Georas, J. Robert, B. Lawrence, 2021, Journal of immunotoxicology.

Long term effects of carbaryl exposure on antiviral immune responses in Xenopus laevis.

J. Robert, B. Lawrence, F. De Jesús Andino, 2017, Chemosphere.

The effects of maternal exposure to bisphenol A on allergic lung inflammation into adulthood.

S. Georas, T. Chapman, A. Roy, 2012, Toxicological sciences : an official journal of the Society of Toxicology.

The Aryl Hydrocarbon Receptor Modulates T Follicular Helper Cell Responses to Influenza Virus Infection in Mice

B. Lawrence, Cassandra L. Houser, 2022, The Journal of Immunology.

Examining the relationship between impaired host resistance and altered immune function in mice treated with TCDD.

B. Lawrence, A. Bohn, B. A. Vorderstrasse, 2003, Toxicology.

2,3,7,8-Tetrachlorodibenzo-p-dioxin induction of cytochrome P450-dependent arachidonic acid metabolism in mouse liver microsomes: evidence for species-specific differences in responses.

N. Kerkvliet, B. Lawrence, A. Rifkind, 1998, Toxicology and applied pharmacology.

Activation of the Aryl Hydrocarbon Receptor During Pregnancy in the Mouse Alters Mammary Development Through Direct Effects on Stromal and Epithelial Tissues1

B. Lawrence, Ravikumar Manickam, B. Lew, 2011, Biology of reproduction.

Aryl hydrocarbon receptor targets pathways extrinsic to bone marrow cells to enhance neutrophil recruitment during influenza virus infection.

B. Lawrence, A. Bohn, S. Teske, 2008, Toxicological sciences : an official journal of the Society of Toxicology.

Aryl Hydrocarbon Receptor Activation during Influenza Virus Infection Unveils a Novel Pathway of IFN-γ Production by Phagocytic Cells1

T. Bushnell, Haley D Neff-Laford, B. Lawrence, 2007, The Journal of Immunology.

Role of altered arachidonic acid metabolism in 2,3,7, 8-tetrachlorodibenzo-p-dioxin-induced immune suppression in C57Bl/6 mice.

N. Kerkvliet, B. Lawrence, 1998, Toxicological sciences : an official journal of the Society of Toxicology.

Dung biomass smoke exposure impairs resolution of inflammatory responses to influenza infection.

T. Thatcher, P. Sime, A. Nogales, 2022, Toxicology and applied pharmacology.

Differential consequences of two distinct AhR ligands on innate and adaptive immune responses to influenza A virus.

B. Lawrence, K. Martin, J. Wheeler, 2014, Toxicological sciences : an official journal of the Society of Toxicology.

Distribution and behavior of the Ah receptor in murine T lymphocytes.

N. Kerkvliet, M. Leid, B. Lawrence, 1996, Toxicology and applied pharmacology.

Developmental Exposure to a Mixture of 23 Chemicals Associated With Unconventional Oil and Gas Operations Alters the Immune System of Mice

S. Georas, J. Robert, S. Nagel, 2018, Toxicological sciences : an official journal of the Society of Toxicology.

Impact of the aryl hydrocarbon receptor on Aurora A kinase and the G2/M phase pathway in hematopoietic stem and progenitor cells

S. Bhattacharya, T. Gasiewicz, B. Lawrence, 2022, bioRxiv.

Aryl hydrocarbon receptor signaling modulates antiviral immune responses: ligand metabolism rather than chemical source is the stronger predictor of outcome

B. Lawrence, L. Boulé, G. Jin, 2018, Scientific Reports.

Novel Cellular Targets of AhR Underlie Alterations in Neutrophilic Inflammation and Inducible Nitric Oxide Synthase Expression during Influenza Virus Infection

B. Lawrence, K. Martin, J. Wheeler, 2012, The Journal of Immunology.

Production by g Novel Pathway of IFN-during Influenza Virus Infection Unveils a Aryl Hydrocarbon Receptor Activation

T. Bushnell, Haley D Neff-Laford, B. Lawrence, 2007 .

Sex-specific enhanced behavioral toxicity induced by maternal exposure to a mixture of low dose endocrine-disrupting chemicals.

D. Cory-Slechta, J. L. Allen, M. Sobolewski, 2014, Neurotoxicology.

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V. Arlt, S. Fenton, B. Brewer, 2013 .

Neonatal oxygen increases sensitivity to influenza A virus infection in adult mice by suppressing epithelial expression of Ear1.

S. Welle, M. O’Reilly, P. Keng, 2012, The American journal of pathology.

Neonatal hyperoxia increases sensitivity of adult mice to bleomycin-induced lung fibrosis.

M. O’Reilly, Min Yee, B. Lawrence, 2013, American journal of respiratory cell and molecular biology.

Lung irradiation increases mortality after influenza A virus challenge occurring late after exposure.

Jacqueline P. Williams, J. Finkelstein, C. Johnston, 2013, International journal of radiation oncology, biology, physics.

Developmental Exposure to Bisphenol A Modulates Innate but Not Adaptive Immune Responses to Influenza A Virus Infection

A. Roy, S. Bauer, B. Lawrence, 2012, PloS one.

Demographic, Reproductive, and Dietary Determinants of Perfluorooctane Sulfonic (PFOS) and Perfluorooctanoic Acid (PFOA) Concentrations in Human Colostrum.

I. Hertz-Picciotto, M. Lamoree, T. Jusko, 2016, Environmental science & technology.

Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus

M. O’Reilly, T. Stevens, Min Yee, 2015, Pediatric pulmonology.

Environmental Lead Exposure and Influenza and Respiratory Syncytial Virus Diagnoses in Young Children: A Test-Negative Case-Control Study

T. Jusko, K. Thevenet‐Morrison, E. van Wijngaarden, 2020, International journal of environmental research and public health.

Neither direct nor developmental exposure to bisphenol A alters the severity of experimental inflammatory colitis in mice

A. Roy, L. Saubermann, W. Cao, 2013, Journal of immunotoxicology.

T cell receptor transgenic mice provide novel insights into understanding cellular targets of TCDD: suppression of antibody production, but not the response of CD8(+) T cells, during infection with influenza virus.

B. Lawrence, K. Mitchell, 2003, Toxicology and applied pharmacology.

Consensus on the Key Characteristics of Immunotoxic Agents as a Basis for Hazard Identification

Andrew A. Rooney, L. Zeise, C. McHale, 2022, Environmental health perspectives.

You AhR what you eat?

D. Sherr, B. Lawrence, 2012, Nature Immunology.

Water Contaminants Associated With Unconventional Oil and Gas Extraction Cause Immunotoxicity to Amphibian Tadpoles

J. Robert, S. Nagel, S. J. Price, 2018, Toxicological sciences : an official journal of the Society of Toxicology.

The aryl hydrocarbon receptor modulates murine hematopoietic stem cell homeostasis and influences lineage-biased stem and progenitor cells.

B. Lawrence, A. Franchini, Keegan Vaughan, 2021, Stem cells and development.

Old Receptor, New Tricks—The Ever-Expanding Universe of Aryl Hydrocarbon Receptor Functions. Report from the 4th AHR Meeting, 29–31 August 2018 in Paris, France

X. Coumoul, R. Barouki, C. Esser, 2018, International journal of molecular sciences.

Environmental toxins as modulators of antiviral immune responses.

B. Lawrence, 2007, Viral immunology.

Activation of the aryl hydrocarbon receptor diminishes the memory response to homotypic influenza virus infection but does not impair host resistance.

B. Lawrence, B. A. Vorderstrasse, 2004, Toxicological sciences : an official journal of the Society of Toxicology.

Exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) suppresses the humoral and cell-mediated immune responses to influenza A virus without affecting cytolytic activity in the lung.

Travis K Warren, B. Lawrence, K. Mitchell, 2000, Toxicological sciences : an official journal of the Society of Toxicology.

Linking the Aryl Hydrocarbon Receptor with Altered DNA Methylation Patterns and Developmentally Induced Aberrant Antiviral CD8+ T Cell Responses

A. Puga, W. Kraus, Anusha Nagari, 2015, The Journal of Immunology.

Environmental toxicants and the developing immune system: a missing link in the global battle against infectious disease?

M. Humble, Bethany N Winans, B. Lawrence, 2011, Reproductive toxicology.

Activation of the aryl hydrocarbon receptor during different critical windows in pregnancy alters mammary epithelial cell proliferation and differentiation.

M. O’Reilly, B. Lawrence, L. Collins, 2009, Toxicological sciences : an official journal of the Society of Toxicology.

New insights into the aryl hydrocarbon receptor as a modulator of host responses to infection

B. Lawrence, B. A. Vorderstrasse, B. P. Lawrence, 2013, Seminars in Immunopathology.

A novel effect of dioxin: exposure during pregnancy severely impairs mammary gland differentiation.

Jennifer A Cundiff, S. Fenton, B. Lawrence, 2004, Toxicological sciences : an official journal of the Society of Toxicology.

Aryl Hydrocarbon Receptor Activation Impairs the Priming but Not the Recall of Influenza Virus-Specific CD8+ T Cells in the Lung1

Jennifer A Cundiff, Alan D. Roberts, D. Woodland, 2006, The Journal of Immunology.

Lung T Cells in the + Influenza Virus-Specific CD 8 Impairs the Priming but Not the Recall of Aryl Hydrocarbon Receptor Activation

Jennifer A Cundiff, Alan D. Roberts, D. Woodland, 2006 .

The aryl hydrocarbon receptor is a modulator of anti-viral immunity.

B. Lawrence, J. Head, 2009, Biochemical pharmacology.

Aryl hydrocarbon receptor activation reduces dendritic cell function during influenza virus infection.

Joshua J. Neumiller, Amanda J. Moore, B. Lawrence, 2010, Toxicological sciences : an official journal of the Society of Toxicology.

Blood Lead Concentrations and Antibody Levels to Measles, Mumps, and Rubella among U.S. Children

T. Jusko, K. Thevenet‐Morrison, R. Wright, 2019, International journal of environmental research and public health.

Aryl hydrocarbon receptor signaling modulates antiviral immune responses: ligand metabolism rather than chemical source is the stronger predictor of outcome

B. Lawrence, L. Boulé, G. Jin, 2018, Scientific Reports.

Immunotoxicology of Halogenated Aromatic Hydrocarbons

B. Lawrence, J. L. Meyers, 2018 .

Neonatal hyperoxia leads to persistent alterations in NK responses to influenza A virus infection.

M. O’Reilly, Min Yee, B. Lawrence, 2015, American journal of physiology. Lung cellular and molecular physiology.

Measuring the Activity of Cytolytic Lymphocytes

B. Lawrence, 2004, Current protocols in toxicology.

Exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) renders influenza virus-specific CD8+ T cells hyporesponsive to antigen.

B. Lawrence, K. Mitchell, 2003, Toxicological sciences : an official journal of the Society of Toxicology.

Assessment of recent developmental immunotoxicity studies with bisphenol A in the context of the 2015 EFSA t-TDI.

Andrew A. Rooney, A. Piersma, H. van Loveren, 2016, Reproductive toxicology.

Activation of the aryl hydrocarbon receptor increases pulmonary neutrophilia and diminishes host resistance to influenza A virus.

Joshua J. Neumiller, B. Lawrence, J. Regal, 2005, American journal of physiology. Lung cellular and molecular physiology.

Effects of Developmental Activation of the AhR on CD4+ T-Cell Responses to Influenza Virus Infection in Adult Mice

Bethany N Winans, B. Lawrence, L. Boulé, 2014, Environmental health perspectives.

Developmental exposure to a mixture of unconventional oil and gas chemicals: A review of experimental effects on adult health, behavior, and disease

J. Robert, S. Nagel, C. Kassotis, 2020, Molecular and Cellular Endocrinology.

The role of polycyclic aromatic hydrocarbon metabolism in dimethylbenz[a]anthracene-induced pre-B lymphocyte apoptosis.

N. Kerkvliet, M. E. Hahn, D. Sherr, 1999, Toxicology and applied pharmacology.

Invited Perspective: Environmental Chemical-Sensing AHR Remains an Enigmatic Key Player in Toxicology

B. Lawrence, B. Kaplan, Barbara L F Kaplan, 2023, Environmental health perspectives.

Refinement of coding SNPs in the human aryl hydrocarbon receptor gene using ISNPranker: An integrative-SNP ranking web-tool

Majid Khalili, Zahra Khoshkam, Younes Aftabi, 2020, Comput. Biol. Chem..

New insights into the role of the aryl hydrocarbon receptor in the function of CD11c+ cells during respiratory viral infection

Bethany N Winans, B. Lawrence, K. Martin, 2014, European journal of immunology.

Memory CD8+ T Cells Are Sufficient To Alleviate Impaired Host Resistance to Influenza A Virus Infection Caused by Neonatal Oxygen Supplementation

M. O’Reilly, Min Yee, B. Lawrence, 2012, Clinical and Vaccine Immunology.

DNA Methylation Patterns in CD4+ T Cells of Naïve and Influenza A Virus-Infected Mice Developmentally Exposed to an Aryl Hydrocarbon Receptor Ligand

Jason R. Myers, B. Lawrence, L. Boulé, 2021, Environmental health perspectives.

The Oxygen Environment at Birth Specifies the Population of Alveolar Epithelial Stem Cells in the Adult Lung

T. Mariani, M. O’Reilly, R. Gelein, 2016, Stem cells.

Neonatal oxygen exposure alters airway hyper‐responsiveness but not the response to allergen challenge in adult mice

M. O’Reilly, B. Lawrence, J. Regal, 2014, Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology.

Fewer CTL, not enhanced NK cells, are sufficient for viral clearance from the lungs of immunocompromised mice.

Haley D Neff-Laford, B. Lawrence, B. A. Vorderstrasse, 2003, Cellular immunology.

Environmental exposures are hidden modifiers of anti-viral immunity.

B. Lawrence, A. Franchini, B. P. Lawrence, 2018, Current opinion in toxicology.

Fewer T lymphocytes and decreased pulmonary influenza virus burden in mice exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD).

B. Lawrence, H. Luong, T. Warren, 2000, Journal of toxicology and environmental health. Part A.

Protection against Lethal Challenge with Streptococcus pneumoniae Is Conferred by Aryl Hydrocarbon Receptor Activation but Is Not Associated with an Enhanced Inflammatory Response

B. Lawrence, B. A. Vorderstrasse, 2006, Infection and Immunity.

TCDD exposure disrupts mammary epithelial cell differentiation and function.

B. Lawrence, L. Collins, B. Lew, 2009, Reproductive toxicology.

Environmental cues received during development shape dendritic cell responses later in life

S. Gerber, Bethany N Winans, B. Lawrence, 2018, PloS one.

Thyroid Disrupting Chemicals in mixture Perturb Thymocyte Differentiation in Xenopus laevis tadpoles.

J. Robert, B. Lawrence, Connor C. McGuire, 2021, Toxicological sciences : an official journal of the Society of Toxicology.

Influenza A virus-dependent remodeling of pulmonary clock function in a mouse model of COPD

I. Rahman, I. Sundar, T. Ahmad, 2015, Scientific Reports.

Diverse gene expression pattern during 5-fluorouridine-induced apoptosis.

Thomas D. Schmittgen, M. Lerner, D. Brackett, 2005, International journal of oncology.

Immune Modulation by TCDD and Related Polyhalogenated Aromatic Hydrocarbons

N. Kerkvliet, B. Lawrence, 2006 .

A cohort study of developmental polychlorinated biphenyl (PCB) exposure in relation to post-vaccination antibody response at 6-months of age.

I. Hertz-Picciotto, S. Schwartz, T. Kavanagh, 2010, Environmental research.

Developmental exposure to chemicals associated with unconventional oil and gas extraction alters immune homeostasis and viral immunity of the amphibian Xenopus.

J. Robert, S. Nagel, B. Lawrence, 2019, The Science of the total environment.

New insights into the role of the aryl hydrocarbon receptor in the function of CD11c+ cells during respiratory viral infection

Bethany N Winans, B. Lawrence, K. Martin, 2014, European journal of immunology.

Environmental toxicants and the developing immune system: a missing link in the global battle against infectious disease?

M. Humble, Bethany N Winans, B. Lawrence, 2011, Reproductive toxicology.

A cohort study of developmental polychlorinated biphenyl (PCB) exposure in relation to post-vaccination antibody response at 6-months of age.

I. Hertz-Picciotto, S. Schwartz, T. Kavanagh, 2010, Environmental Research.

Life-Long Implications of Developmental Exposure to Environmental Stressors: New Perspectives.

F. Perera, J. Toppari, C. Junien, 2015, Endocrinology.