Jun Chen

发表

The mechanism of the G0/G1 cell cycle phase arrest induced by activation of PXR in human cells.

Chun Meng, Hang Wang, Huahua Feng, 2011, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.

Phospholipid scramblase 3 is the mitochondrial target of protein kinase C delta-induced apoptosis.

Q. Dai, Jihua Liu, Jun Chen, 2003, Cancer research.

The Endoplasmic Reticulum Adaptor Protein ERAdP Initiates NK Cell Activation via the Ubc13-Mediated NF-κB Pathway

B. Long, P. Zhu, B. Ye, 2015, The Journal of Immunology.

Ubc 13-Mediated NF-ERAdP Initiates NK Cell Activation via the The Endoplasmic Reticulum Adaptor Protein

B. Long, Yanyan Du, B. Ye, 2014 .

Enhancement of metabolic oxidative stress-induced cytotoxicity by the thioredoxin inhibitor 1-methylpropyl 2-imidazolyl disulfide is mediated through the ASK1-SEK1-JNK1 pathway.

Yong J. Lee, Jae-Joon Song, Jun Chen, 2002, Molecular pharmacology.

Activation of Nrf2 by cadmium and its role in protection against cadmium-induced apoptosis in rat kidney cells.

Jun Chen, Z. Shaikh, 2009, Toxicology and applied pharmacology.

Different signaling responses to anti‐proliferative agents in human aortic and venous smooth muscle cells

D. Blumenthal, A. Cheung, Jihua Liu, 2006, Journal of cellular biochemistry.

Valosin-Containing Protein Cleavage by Granzyme K Accelerates an Endoplasmic Reticulum Stress Leading to Caspase-Independent Cytotoxicity of Target Tumor Cells

Z. Fan, Lei Shi, Yuming Guo, 2010, The Journal of Immunology.

Cytotoxicity of Target Tumor Cells Leading to Caspase-Independent Endoplasmic Reticulum Stress by Granzyme K Accelerates an Valosin-Containing Protein Cleavage

Z. Fan, Lei Shi, Yuming Guo, 2010 .

Tumor Cells Caspase-Independent Cytotoxicity of Target Reticulum Stress Leading to Granzyme K Accelerates an Endoplasmic Valosin-Containing Protein Cleavage

Jun Chen, 2010 .

Mitochondrial ceramide increases in UV-irradiated HeLa cells and is mainly derived from hydrolysis of sphingomyelin

Q. Dai, Jihua Liu, T. McIntyre, 2004, Oncogene.

An acute lytic cell death induced by xanthohumol obstructed ROS detecting in HL-60 cells.

Yue Meng, Li Zhou, Jun Chen, 2020, Toxicology in vitro : an international journal published in association with BIBRA.

Xanthohumol Induces ROS through NADPH Oxidase, Causes Cell Cycle Arrest and Apoptosis

Zhen Li, Jing Zhao, Jun Chen, 2021, Oxidative Medicine and Cellular Longevity.

Potent and Selective Disruption of Protein Kinase D Functionality by a Benzoxoloazepinolone*

P. Wipf, J. Lazo, E. Sharlow, 2008, Journal of Biological Chemistry.

Phospholipid scramblase 3 controls mitochondrial structure, function, and apoptotic response.

D. Grossman, Q. Dai, Jihua Liu, 2003, Molecular cancer research : MCR.

The apoptotic effect of HA14-1, a Bcl-2-interacting small molecular compound, requires Bax translocation and is enhanced by PK11195.

Ruey-min Lee, Q. Dai, Jihua Liu, 2002, Molecular cancer therapeutics.

T-cell Immunoglobulin and ITIM Domain (TIGIT) Receptor/Poliovirus Receptor (PVR) Ligand Engagement Suppresses Interferon-γ Production of Natural Killer Cells via β-Arrestin 2-mediated Negative Signaling*

Xuan Cheng, N. Hou, Xiao Yang, 2014, The Journal of Biological Chemistry.

Granzyme K degrades the redox/DNA repair enzyme Ape1 to trigger oxidative stress of target cells leading to cytotoxicity.

Z. Fan, Yuming Guo, Jun Chen, 2008, Molecular immunology.

Cancer Therapy : Preclinical BCMab 1 , A Monoclonal Antibody against Aberrantly Glycosylated Integrin a 3 b 1 , Has Potent Antitumor Activity of Bladder Cancer In Vivo

Zhao Yang, Chong Li, Deqing Hu, 2014 .

Protein Kinase C-Independent Effects of Protein Kinase D3 in Glucose Transport in L6 Myotubes

G. Lu, Q. J. Wang, Jun Chen, 2005, Molecular Pharmacology.

BCMab1, A Monoclonal Antibody against Aberrantly Glycosylated Integrin α3β1, Has Potent Antitumor Activity of Bladder Cancer In Vivo

Zhao Yang, Chong Li, Deqing Hu, 2014, Clinical Cancer Research.

Protein kinase D 3 is localized in vesicular structures and interacts with vesicle-associated membrane protein 2.

S. Garfield, G. Lu, A. Huppler, 2007, Cellular signalling.

Protein kinase D3 (PKD3) contributes to prostate cancer cell growth and survival through a PKCepsilon/PKD3 pathway downstream of Akt and ERK 1/2.

Shivendra V. Singh, F. Deng, Q. Wang, 2008, Cancer research.

A protein kinase C/protein kinase D pathway protects LNCaP prostate cancer cells from phorbol ester-induced apoptosis by promoting ERK1/2 and NF-{kappa}B activities.

Liyong Zhang, Q. J. Wang, Jun Chen, 2011, Carcinogenesis.

Novel protein kinase D inhibitors cause potent arrest in prostate cancer cell growth and motility

P. Wipf, J. Lazo, E. Sharlow, 2010, BMC chemical biology.

A protein kinase C/protein kinase D pathway protects LNCaP prostate cancer cells from phorbol ester-induced apoptosis by promoting ERK1/2 and NF- k B activities

Liyong Zhang, Q. J. Wang, Jun Chen, 2011 .

Eriodictyol regulated ferroptosis, mitochondrial dysfunction, and cell viability via Nrf2/HO‐1/NQO1 signaling pathway in ovarian cancer cells

Jun Chen, W. Tian, Qiufang Li, 2023, Journal of biochemical and molecular toxicology.

Individual C1 domains of PKD3 in phorbol ester-induced plasma membrane translocation of PKD3 in intact cells.

Q. J. Wang, Jun Chen, Gulsum Anderson, 2005, Cellular signalling.

Selective binding of phorbol esters and diacylglycerol by individual C1 domains of the PKD family.

F. Deng, Q. J. Wang, Jun Li, 2008, The Biochemical journal.

Bcl-2 family proteins are essential for platelet survival

C. Tse, M. L. Smith, S. Rosenberg, 2007, Cell Death and Differentiation.

Xuan Cheng, N. Hou, Xiao Yang, 2014, The Journal of Biological Chemistry.

Phospholipid scramblase 3 controls mitochondrial structure, function, and apoptotic response.

D. Grossman, Q. Dai, Jihua Liu, 2003, Molecular cancer research : MCR.

Potent and Selective Disruption of Protein Kinase D Functionality by a Benzoxoloazepinolone*

P. Wipf, J. Lazo, E. Sharlow, 2008, Journal of Biological Chemistry.