O. Stoler

发表

Dynamic Gain Analysis Reveals Encoding Deficiencies in Cortical Neurons That Recover from Hypoxia-Induced Spreading Depolarizations

M. Gutnick, F. Wolf, I. Fleidervish, 2019, The Journal of Neuroscience.

Functional implications of axon initial segment cytoskeletal disruption in stroke

I. Fleidervish, O. Stoler, 2015, Acta Pharmacologica Sinica.

Subcellular Distribution of Persistent Sodium Conductance in Cortical Pyramidal Neurons

I. Fleidervish, O. Stoler, I. Melamed, 2021, The Journal of Neuroscience.

Mitochondrial activity is crucial for the plasticity of central synapses, but how the firing

I. Fleidervish, I. Sekler, Grace E. Stutzmann, 2022 .

Aberrant activity of mitochondrial NCLX is linked to impaired synaptic transmission and is associated with mental retardation

Y. Amitai, H. Prokisch, I. Fleidervish, 2021, Communications biology.

CDH2 mutation affecting N-cadherin function causes attention-deficit hyperactivity disorder in humans and mice

O. Birk, B. Rotblat, G. Meiri, 2021, Nature Communications.

Author Correction: Aberrant activity of mitochondrial NCLX is linked to impaired synaptic transmission and is associated with mental retardation

Y. Amitai, H. Prokisch, I. Fleidervish, 2021, Communications biology.

Role of sodium channel subtype in action potential generation by neocortical pyramidal neurons

M. Gutnick, F. Wolf, I. Fleidervish, 2018, Proceedings of the National Academy of Sciences.

Mitochondria decode firing frequency and coincidences of postsynaptic APs and EPSPs

I. Fleidervish, I. Sekler, Grace E. Stutzmann, 2021, bioRxiv.

Frequency- and spike-timing-dependent mitochondrial Ca2+ signaling regulates the metabolic rate and synaptic efficacy in cortical neurons

I. Fleidervish, I. Sekler, O. Stoler, 2022, eLife.