W. Dillmann

发表

Age-induced decreases in the messenger RNA coding for the sarcoplasmic reticulum Ca2(+)-ATPase of the rat heart.

R. Polikar, D. Rohrer, L. M. Maciel, 1990, Circulation research.

Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network.

D. M. Foster, H. Lal, W. Dillmann, 2013, International journal of cardiology.

Inhibition of HDAC9 increases T regulatory cell function and prevents colitis in mice.

H. Sai, W. Hancock, Liqing Wang, 2010, Gastroenterology.

Results Differing Effects of Pan-HDACi and Class I-Specific HDACi Therapy on DSS Colitis

H. Sai, W. Hancock, Liqing Wang, 2022 .

Cardiac-Directed Expression of Adenylyl Cyclase Catalytic Domain Reverses Cardiac Dysfunction Caused by Sustained Beta-Adrenergic Receptor Stimulation

M. Gao, T. Guo, H. Hammond, 2016, JACC. Basic to translational science.

Thyroid hormone induced changes in cardiac proteins and mRNAs.

D. Rohrer, B. Popovich, W. Dillmann, 1987, Hormone and metabolic research. Supplement series.

Hyperthermia: from diagnostic and treatments to new discoveries.

M. Wacker, C. Touchberry, M. Brotto, 2012, Recent patents on biotechnology.

Overexpression of PHGPx and HSP60/10 protects against ischemia/reoxygenation injury.

J. Hollander, W. Dillmann, Kurt M. Lin, 2003, Free radical biology & medicine.

Specific heat shock proteins protect microtubules during simulated ischemia in cardiac myocytes.

Jody L. Martin, R. Mestril, W. Bluhm, 1998, American journal of physiology. Heart and circulatory physiology.

Small heat shock proteins and protection against ischemic injury in cardiac myocytes.

L. Brunton, Jody L. Martin, R. Mestril, 1997, Circulation.

Ischemia of the dog heart induces the appearance of a cardiac mRNA coding for a protein with migration characteristics similar to heat-shock/stress protein 71.

J. Ross, H. Mehta, W. Dillmann, 1986, Circulation research.

Influences of Increased Expression of the Ca2+ ATPase of the Sarcoplasmic Reticulum by a Transgenic Approach on Cardiac Contractility

W. Dillmann, 1998, Annals of the New York Academy of Sciences.

Transcription of the rat sarcoplasmic reticulum Ca2+ adenosine triphosphatase gene is increased by 3,5,3'-triiodothyronine receptor isoform-specific interactions with the myocyte-specific enhancer factor-2a.

A. Moriscot, M. R. Sayen, W. Dillmann, 1997, Endocrinology.

Leukemia Inhibitory Factor and Interleukin-6 downregulate sarcoplasmic reticulum Ca2+ ATPase (SERCA2) in cardiac myocytes

F. Villarreal, W. Dillmann, S. Villegas, 2000, Basic Research in Cardiology.

Protein Kinase G Activation Reverses Oxidative Stress and Restores Osteoblast Function and Bone Formation in Male Mice With Type 1 Diabetes

R. Sah, R. Pilz, D. Casteel, 2018, Diabetes.

Altered cardiac phenotype in transgenic mice carrying the delta337 threonine thyroid hormone receptor beta mutant derived from the S family.

B. Gloss, M. R. Sayen, W. Bluhm, 1999, Endocrinology.

Cardiac ion channel expression and contractile function in mice with deletion of thyroid hormone receptor alpha or beta.

W. Giles, B. Gloss, J. Samarut, 2001, Endocrinology.

Cardiac Expression and Function of Thyroid Hormone Receptor β and Its PV Mutant

B. Gloss, Sheue-yann Cheng, D. Belke, 2003 .

Cardiac expression and function of thyroid hormone receptor beta and its PV mutant.

B. Gloss, Sheue-yann Cheng, D. Belke, 2003, Endocrinology.

Overexpression of heat shock protein 72 in transgenic mice decreases infarct size in vivo.

R. Mestril, R. Sievers, C. Wolfe, 1996, Circulation.

A recombinant antibody increases cardiac contractility by mimicking phospholamban phosphorylation

T. Dieterle, G. Silverman, D. Belke, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Effects of mutant and antisense RNA of phospholamban on SR Ca(2+)-ATPase activity and cardiac myocyte contractility.

W. Lew, J. Martín, W. Dillmann, 1999, Circulation.

Heat shock proteins in myocardial stress.

R. Mestril, W. Dillmann, 1995, Zeitschrift fur Kardiologie.

Small Heat Shock Proteins and Protection against Injury

W. Dillmann, 1999, Annals of the New York Academy of Sciences.

Heat shock proteins and protection against ischemic injury.

W. Dillmann, 1999 .

Transgene overexpression of αB crystallin confers simultaneous protection against cardiomyocyte apoptosis and necrosis during myocardial ischemia and reperfusion

D. Das, H. Otani, Jody L. Martin, 2001 .

Non-genomic thyroid hormone signaling through NO/cGMP/PKGII

J. Frangos, Shunhui Zhuang, R. Pilz, 2013, BMC Pharmacology and Toxicology.

p75 neurotrophin receptor regulates agonist-induced pulmonary vasoconstriction.

Benjamin D. Sachs, K. Akassoglou, W. Yao, 2008, American journal of physiology. Heart and circulatory physiology.

Sarco/endoplasmic Reticulum Calcium ATPase-2 Expression Is Regulated by ATF6 during the Endoplasmic Reticulum Stress Response

H. Hoover, D. Thuerauf, C. Glembotski, 2001, The Journal of Biological Chemistry.

BRIEF COMMUNICATION Mice Overexpressing 70-kDa Heat Shock Protein Show Increased Resistance to Malonate and 3-Nitropropionic Acid

O. Andreassen, M. Beal, R. Ferrante, 2002 .

Trophic effects of angiotensin II on neonatal rat cardiac myocytes are mediated by cardiac fibroblasts.

N. Kim, Noel N Kim, M. Printz, 1995, The American journal of physiology.

Diabetic Cardiomyopathy: What Is It and Can It Be Fixed?

W. Dillmann, 2019, Circulation research.

Transgenic overexpression of the sarcoplasmic reticulum Ca2+ ATPase improves reticular Ca2+ handling in normal and diabetic rat hearts

K. Wagner, J. Günther, C. Tschöpe, 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Identification of Functional Angiotensin II Receptors on Rat Cardiac Fibroblasts

Noel N Kim, M. Printz, F. Villarreal, 1993, Circulation.

Early Short-Term Treatment With Doxycycline Modulates Postinfarction Left Ventricular Remodeling

J. Omens, J. Covell, F. Villarreal, 2003, Circulation.

Hsp70 inhibits aminoglycoside-induced hearing loss and cochlear hair cell death

F. Lee, K. Harris, L. Cunningham, 2009, Cell Stress and Chaperones.

Ncor2/PPARα-Dependent Upregulation of MCUb in the Type 2 Diabetic Heart Impacts Cardiac Metabolic Flexibility and Function

C. Benner, S. Heinz, D. Casteel, 2020, Diabetes.

Selective reduction of creatine kinase subunit mRNAs in striated muscle of diabetic rats.

M. Payne, A. Strauss, W. Dillmann, 1992, The American journal of physiology.

Insulin responsiveness of CK-M and CK-B mRNA in the diabetic rat heart.

M. R. Sayen, B. Popovich, W. Dillmann, 1991, The American journal of physiology.

In vivo selective expression of thyroid hormone receptor α1 in endothelial cells attenuates myocardial injury in experimental myocardial infarction in mice.

A. Makino, J. Brown, J. Díaz-Juárez, 2014, American journal of physiology. Regulatory, integrative and comparative physiology.

Thyroid hormone inhibits ERK phosphorylation in pressure overload-induced hypertrophied mouse hearts through a receptor-mediated mechanism.

W. Dillmann, B. Scott, Jorge A. Suarez, 2010, American journal of physiology. Cell physiology.

In vivo gene delivery of HSP70i by adenovirus and adeno-associated virus preserves contractile function in mouse heart following ischemia-reperfusion.

B. Gloss, H. Duplain, J. Hollander, 2006, American journal of physiology. Heart and circulatory physiology.

Skeletal Muscle Ischemia and Heat Shock Proteins.

W. Dillmann, 1996 .

Unique Hexosaminidase Reduces Metabolic Survival Signal and Sensitizes Cardiac Myocytes to Hypoxia/Reoxygenation Injury

Steven P Jones, N. Zachara, T. Hamid, 2008, Circulation research.

Common clinical indices of thyroid hormone action: relationships to serum free 3,5,3'-triiodothyronine concentration and estimated nuclear occupancy.

G. Runger, J. Bantle, J. Oppenheimer, 1980, The Journal of clinical endocrinology and metabolism.

Overexpression of p53 due to excess protein O-GlcNAcylation is associated with coronary microvascular disease in type 2 diabetes.

J. Yuan, A. Makino, Makiko Watanabe, 2019, Cardiovascular research.

O-GlcNAcase overexpression reverses coronary endothelial cell dysfunction in type 1 diabetic mice.

A. Makino, W. Dillmann, Ying Han, 2015, American journal of physiology. Cell physiology.

Hsp70 Inhibits Aminoglycoside-Induced Hair Cell Death and is Necessary for the Protective Effect of Heat Shock

M. I. Lomax, F. Lee, L. Cunningham, 2008, Journal of the Association for Research in Otolaryngology.

Thyroid hormone markedly increases the mRNA coding for sarcoplasmic reticulum Ca2+-ATPase in the rat heart.

D. Rohrer, W. Dillmann, Daniel K. Rohrer, 1988, The Journal of biological chemistry.

Thyroid hormone receptor-beta is associated with coronary angiogenesis during pathological cardiac hypertrophy.

A. Makino, D. Belke, W. Dillmann, 2009, Endocrinology.

Early degradation and serum appearance of type I collagen fragments after myocardial infarction.

J. Omens, J. Covell, J. Risteli, 2004, Journal of molecular and cellular cardiology.

Brief Communication Early degradation and serum appearance of type I collagen fragments after myocardial infarction

J. Omens, J. Covell, J. Risteli, 2004 .

Role for high-glucose-induced protein O-GlcNAcylation in stimulating cardiac fibroblast collagen synthesis.

S. Ihm, Nakon Aroonsakool, F. Villarreal, 2014, American journal of physiology. Cell physiology.

Effect of lifelong overexpression of HSP70 in skeletal muscle on age‐related oxidative stress and adaptation after nondamaging contractile activity

M. Jackson, R. Mestril, A. McArdle, 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

The MKK6-p38 MAPK pathway prolongs the cardiac contractile calcium transient, downregulates SERCA2, and activates NF-AT.

C. Glembotski, W. Dillmann, P. McDonough, 2003, Cardiovascular research.

Ischemia-Reoxygenation Protects Mitochondrial Function and Prevents Apoptotic Cell Deaths Induced by Simulated Combined and Individual Mitochondrial HSP 60 and HSP 10 Expression in Cardiac Myocytes

I. Scheffler, R. Mestril, Ian Y. Lian, 2001 .

Triiodothyronine-stimulated formation of poly(A)-containing nuclear RNA and mRNA in rat liver.

J. Oppenheimer, W. Dillmann, H. Schwartz, 1978, Endocrinology.

Dilated cardiomyopathy in transgenic mice expressing a mutant A subunit of protein phosphatase 2A.

G. Walter, H. Rockman, A. Rapacciuolo, 2000, American journal of physiology. Heart and circulatory physiology.

Heat shock protein 70 kDa over-expression and 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced nigrostriatal degeneration in mice

J. López-Barneo, J. Díaz-Martín, W. Dillmann, 2011, Neuroscience.

Mitochondrial 8-oxoguanine glycosylase decreases mitochondrial fragmentation and improves mitochondrial function in H9C2 cells under oxidative stress conditions.

T. Gawlowski, Heidi E. Kocalis, W. Dillmann, 2014, American journal of physiology. Cell physiology.

CHIP and Hsp70 regulate tau ubiquitination, degradation and aggregation

John Hardy, Dennis W. Dickson, Andrew Grover, 2004 .

Using genetically engineered mice to study myocardial ischemia-reperfusion injury.

B. Gloss, Y. Ho, D. Das, 2002, Methods in enzymology.

Deletion of the Inducible 70-kDa Heat Shock Protein Genes in Mice Impairs Cardiac Contractile Function and Calcium Handling Associated With Hypertrophy

D. Dix, C. Boer, Ying Hu, 2006, Circulation.

Transgene overexpression of alphaB crystallin confers simultaneous protection against cardiomyocyte apoptosis and necrosis during myocardial ischemia and reperfusion.

D. Das, H. Otani, W. Dillmann, 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Molecular mechanisms at the tissue level in hyperthyroidism.

J. Oppenheimer, W. Dillmann, 1978, Clinics in endocrinology and metabolism.

Diabetes mellitus-induced changes in the concentration of specific mRNAs and proteins.

W. Dillmann, 1988, Diabetes/metabolism reviews.

Comparison of creatine kinase M and B subunit, mRNAs and isoenzyme activity in ischemicdog myocardium

B. Popovich, W. Dillmann, H. Metha, 1987 .

Basic Science and Experimental Studies The Na/Ca 2 Exchanger/SR Ca 2 ATPase Transport Capacity Regulates the Contractility of Normal and Hypertrophied Feline Ventricular Myocytes

S. Houser, J. Gaughan, B. McGowan, 2005 .

Functional effects of enhancing or silencing adenosine A2b receptors in cardiac fibroblasts.

F. Villarreal, B. Ito, J. Suárez, 2004, American journal of physiology. Heart and circulatory physiology.

Endothelin downregulates SERCA2 gene and protein expression in adult rat ventricular myocytes: regulation by pertussis toxin-sensitive Gi protein and cAMP.

L. Brunton, Jody L. Martin, W. Dillmann, 2009, American journal of physiology. Heart and circulatory physiology.

Mitochondria protection from hypoxia/reoxygenation injury with mitochondria heat shock protein 70 overexpression.

J. Hollander, W. Dillmann, Courtney L. Williamson, 2008, American journal of physiology. Heart and circulatory physiology.

Overexpression of HSP10 in skeletal muscle of transgenic mice prevents the age-related fall in maximum tetanic force generation and muscle Cross-Sectional Area.

M. Jackson, R. Mestril, A. McArdle, 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

Increased expression of SERCA in the hearts of transgenic mice results in increased oxidation of glucose.

A. Stenbit, D. Belke, W. Dillmann, 2007, American journal of physiology. Heart and circulatory physiology.

Regulation of expression of cardiac sarcoplasmic reticulum proteins under pathophysiological conditions

W. Dillmann, 1996, Molecular and Cellular Biochemistry.

Compromised Myocardial Energetics in Hypertrophied Mouse Hearts Diminish the Beneficial Effect of Overexpressing SERCA2a

J. Ingwall, R. Tian, D. Belke, 2011, The Journal of Biological Chemistry.

Influence of Transgenic Expression of Sarcoplasmic Reticulum Ca2+ATPase on Reticular Ca2+ Transport in Rat Hearts

K. Wagner, J. Günther, M. Paul, 2003 .

Adenovirus-mediated overexpression of human transforming growth factor-beta 1 in rat cardiac fibroblasts, myocytes and smooth muscle cells.

A. A. Lee, F. Villarreal, F. Giordano, 1996, Journal of molecular and cellular cardiology.

Iodide Induces Transforming Growth Factor Beta1 (TGF-β1) mRNA in Sheep Thyroid Cells

J. Meinkoth, W. Dillmann, G. Burrow, 1992 .

Influence of thyroid hormone and retinoic acid on slow sarcoplasmic reticulum Ca2+ ATPase and myosin heavy chain alpha gene expression in cardiac myocytes. Delineation of cis-active DNA elements that confer responsiveness to thyroid hormone but not to retinoic acid.

D. Rohrer, W. Dillmann, R. Hartong, 1991, The Journal of biological chemistry.

Calcium regulatory proteins and their alteration by transgenic approaches.

W. Dillmann, 1999, The American journal of cardiology.

Phospholamban-to-SERCA2 ratio controls the force-frequency relationship.

W. Lew, S. Post, W. Bluhm, 1999, American journal of physiology. Heart and circulatory physiology.

Thyroid hormone response of slow and fast sarcoplasmic reticulum Ca2+ ATPase mRNA in striated muscle

M. R. Sayen, D. Rohrer, W. Dillmann, 1992, Molecular and Cellular Endocrinology.

Preserved Cardiac Function despite Marked Impairment of cAMP Generation

Hong Wang, M. Gao, T. Tang, 2013, PloS one.

Alterations in mitochondrial function and cytosolic calcium induced by hyperglycemia are restored by mitochondrial transcription factor A in cardiomyocytes.

A. Makino, W. Dillmann, Hong Wang, 2008, American journal of physiology. Cell physiology.

Anthracycline cardiotoxicity in transgenic mice overexpressing SR Ca2+-ATPase.

R. Olson, B. Cusack, B. Burke, 2003, Biochemical and biophysical research communications.

Effects of cytokine treatment on angiotensin II type 1A receptor transcription and splicing in rat cardiac fibroblasts.

B. Greenberg, W. Dillmann, D. Gurantz, 2005, American journal of physiology. Heart and circulatory physiology.

Transcription Factor NF-κB Is Necessary for Up-regulation of Type 1 Angiotensin II Receptor mRNA in Rat Cardiac Fibroblasts Treated with Tumor Necrosis Factor-α or Interleukin-1β*

B. Greenberg, W. Dillmann, D. Gurantz, 2002, The Journal of Biological Chemistry.

Mechanism of action of thyroid hormones.

W. Dillmann, 1985, The Medical clinics of North America.

Comparison of the metabolism and distribution of L-triiodothyronine and triiodothyroacetic acid in the rat: a possible explanation of differential hormonal potency.

M. Surks, J. Oppenheimer, B. Goslings, 1976, Endocrinology.

The Raf-MEK-ERK Cascade Represents a Common Pathway for Alteration of Intracellular Calcium by Ras and Protein Kinase C in Cardiac Myocytes*

D. Zechner, C. Glembotski, W. Dillmann, 1998, The Journal of Biological Chemistry.

Nuclear receptors and the initiation of thyroid hormone action.

M. Surks, J. Oppenheimer, W. Dillmann, 1976, Recent progress in hormone research.

Selective alterations in hepatic enzyme response after reduction of nuclear triiodothyronine receptor sites by partial hepatectomy and starvation.

J. Oppenheimer, W. Dillmann, H. Schwartz, 1978, Biochemical and biophysical research communications.

Glucagon administration decreases hepatic nuclear triiodothyronine binding capacity.

J. Oppenheimer, W. Dillmann, R. A. Bonner, 1978, Endocrinology.

Cellular action of thyroid hormone on the heart.

W. Dillmann, 2002, Thyroid : official journal of the American Thyroid Association.

Adeno-associated virus-mediated expression of thyroid hormone receptor isoforms-alpha1 and -beta1 improves contractile function in pressure overload-induced cardiac hypertrophy.

B. Gloss, D. Belke, W. Dillmann, 2007, Endocrinology.

The Thyroid Hormone Receptor-β-Selective Agonist GC-1 Differentially Affects Plasma Lipids and Cardiac Activity.

B. Gloss, J. Baxter, G. Grover, 2000, Endocrinology.

Constitutive protein kinase G activation exacerbates stress‐induced cardiomyopathy

Yusu Gu, N. Dalton, G. Boss, 2021, British journal of pharmacology.

Phospholamban-to-SERCA2 ratio controls the force-frequency relationship.

W. Lew, S. Post, W. Bluhm, 1999, The American journal of physiology.

Mutation of COOH-terminal lysines in overexpressed alpha B-crystallin abrogates ischemic protection in cardiomyocytes.

Jody L. Martin, R. Mestril, W. Bluhm, 2002, American journal of physiology. Heart and circulatory physiology.

cAMP‐ and Ca2+/calmodulin‐dependent protein kinases mediate inotropic, lusitropic and arrhythmogenic effects of urocortin 2 in mouse ventricular myocytes

H. Mächler, B. Pieske, J. Spiess, 2011, British journal of pharmacology.

Downregulation of Connexin40 Is Associated with Coronary Endothelial Cell Dysfunction in Streptozotocin-induced Diabetic Mice Cx-mimetic Peptides 40 Gap27 (srpteknvfiv) and Control Scram

J. Yuan, A. Makino, O. Platoshyn, 2022 .

O-GlcNAcylation of 8-Oxoguanine DNA Glycosylase (Ogg1) Impairs Oxidative Mitochondrial DNA Lesion Repair in Diabetic Hearts*

D. Casteel, Darren E. Casteel, F. Cividini, 2016, The Journal of Biological Chemistry.

Effects of hyperthyroidism on delayed rectifier K+ currents in left and right murine atria.

Ying Hu, W. Dillmann, S. V. Jones, 2005, American journal of physiology. Heart and circulatory physiology.

Effects of hyperthyroidism on delayed rectifier potassium currents in left and right murine atrium

Ying Hu, W. Dillmann, S. V. Jones, 2005 .

α1-Adrenergic Stimulation Inhibits 3,5,3'-Triiodothyronine-Induced Expression of the Rat Heart Sarcoplasmic Reticulum Ca2+ Adenosine Triphosphatase Gene1.

K. Knowlton, A. Moriscot, W. Dillmann, 1997, Endocrinology.

Thyroid hormone receptor-α and vascular function.

J. Yuan, A. Makino, W. Dillmann, 2012, American journal of physiology. Cell physiology.

Thyroid hormone receptor- (cid:1) and vascular function

J. Yuan, A. Makino, W. Dillmann, 2012 .

Nuclear Receptors for Triiodothyronine: A Physiological Perspective

J. Oppenheimer, W. Dillmann, 1978 .

Editorial: thyroid hormone action and cardiac contractility - a complex affair.

W. Dillmann, 1996, Endocrinology.

Diabetes decreases creatine kinase enzyme activity and mRNA level in the rat heart.

K. Boheler, B. Popovich, W. Dillmann, 1989, The American journal of physiology.

Heat shock proteins and protection against ischemic injury.

W. Dillmann, 2001, Infectious diseases in obstetrics and gynecology.

Time course of response of individual messenger RNAs in the rat heart to T3.

Ravi Moysore Shanker, W. Dillmann, W. E. Neeley, 1987, Journal of molecular and cellular cardiology.

Expression of the mitochondrial calcium uniporter in cardiac myocytes improves impaired mitochondrial calcium handling and metabolism in simulated hyperglycemia.

F. Cividini, J. Díaz-Juárez, W. Dillmann, 2016, American journal of physiology. Cell physiology.

Conditional increase in SERCA2a protein is able to reverse contractile dysfunction and abnormal calcium flux in established diabetic cardiomyopathy.

W. Dillmann, B. Scott, Jorge A. Suarez, 2008, American journal of physiology. Regulatory, integrative and comparative physiology.

Influence of thyroid hormone on myosin heavy chain mRNA and other messenger RNAs in the rat heart.

Ravi Moysore Shanker, W. Dillmann, A. Barrieux, 1989, Endocrine research.

Mice overexpressing rat heat shock protein 70 are protected against cerebral infarction

F R Sharp, P. Weinstein, F. Sharp, 2000, Annals of neurology.

Phorbol myristate acetate-induced hypertrophy of neonatal rat cardiac myocytes is associated with decreased sarcoplasmic reticulum Ca2+ ATPase (SERCA2) gene expression and calcium reuptake.

F. Villarreal, F. Giordano, W. Dillmann, 1996, Journal of molecular and cellular cardiology.

Diabetes Mellitus Induces Changes in Cardiac Myosin of the Rat

W. Dillmann, 1980, Diabetes.

IN CARDIAC FUNCTION AND THEIR MOLECULAR BASIS

W. Dillmann, 1989 .

Changing the cardiac calcium transient: SERCA2 overexpression versus phospholamban inhibition

W. Dillmann, 2000 .

Diabetes and thyroid-hormone-induced changes in cardiac function and their molecular basis.

W. Dillmann, 1989, Annual review of medicine.

Overexpression of Wild-Type Heat Shock Protein 27 and a Nonphosphorylatable Heat Shock Protein 27 Mutant Protects Against Ischemia/Reperfusion Injury in a Transgenic Mouse Model

Jody L. Martin, J. Hollander, D. Belke, 2004, Circulation.

Overexpression of the rat sarcoplasmic reticulum Ca2+ ATPase gene in the heart of transgenic mice accelerates calcium transients and cardiac relaxation.

H. Rockman, D. Choi, M. R. Sayen, 1997, The Journal of clinical investigation.

The Cardiac-specific Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Induces Hypertrophy and Dilated Cardiomyopathy Associated with Increased Protein Phosphatase 2A Activity*

Tong Zhang, John Ross, H. Schulman, 2002, The Journal of Biological Chemistry.

Nitrosyl-Cobinamide, a New and Direct Nitric Oxide–Releasing Drug Effective In Vivo

Virgil L. Woods, Gerry R Boss, A. Makino, 2007, Experimental biology and medicine.

The effects of amiodarone on serum thyroid hormones and hepatic thyroxine 5'-monodeiodination in rats.

J. Hershman, A. Reed, W. Dillmann, 1983, Endocrinology.

Nongenomic Thyroid Hormone Signaling Occurs Through a Plasma Membrane–Localized Receptor

J. Frangos, G. Boss, Shunhui Zhuang, 2014, Science Signaling.

Diabetes and the Accompanying Hyperglycemia Impairs Cardiomyocyte Calcium Cycling through Increased Nuclear O-GlcNAcylation*

M. Fukuda, Misa Suzuki, W. Dillmann, 2003, Journal of Biological Chemistry.

Cardiac hypertrophy and thyroid hormone signaling

W. Dillmann, 2009, Heart Failure Reviews.

Influence of thyroid hormone on the in vitro translational activity of specific mRNAs in the rat heart.

P. Contreras, W. Dillmann, A. Barrieux, 1983, Journal of Biological Chemistry.

Myofibroblasts revert to an inactive phenotype during regression of liver fibrosis

Christopher K. Glass, Min Cong, David A. Brenner, 2012, Proceedings of the National Academy of Sciences.

Ischemia Induces Changes in the Level of mRNAs Coding for Stress Protein 71 and Creatine Kinase M

W. Dillmann, H. Mehta, Brett K. Popovich, 1988, Circulation research.

Cardiac Response to Pressure Overload in the Rat: The Selective Alteration of In Vitro Directed RNA Translation Products

K. Boheler, W. Dillmann, 1988, Circulation research.

Diabetes-induced alterations in the translational activity of specific messenger ribonucleic acids isolated from rat hearts.

W. Dillmann, A. Barrieux, W. E. Neeley, 1985, Circulation research.

Hormonal influences on cardiac myosin ATPase activity and myosin isoenzyme distribution

W. Dillmann, 1984, Molecular and Cellular Endocrinology.

Glucagon influences the expression of thyroid hormone action: discrepancy between nuclear triiodothyronine receptor number and enzyme responses.

J. Oppenheimer, W. Dillmann, 1979, Endocrinology.

Selective modulation of thyroid hormone receptor action

Paul Webb, R. Fletterick, J. Baxter, 2001, The Journal of Steroid Biochemistry and Molecular Biology.

Prospective study of perioperative factors predicting hypocalcemia after thyroid and parathyroid surgery.

L. Orloff, D. Tieu, R. Weisman, 2006, Archives of otolaryngology--head & neck surgery.

Mitochondrial fragmentation and superoxide anion production in coronary endothelial cells from a mouse model of type 1 diabetes

A. Makino, W. Dillmann, B. Scott, 2010, Diabetologia.

Intracoronary gene transfer of fibroblast growth factor–5 increases blood flow and contractile function in an ischemic region of the heart

Peipei Ping, P. Ping, A. DeMaria, 1996, Nature Medicine.

Thyroid hormone action in the heart.

G. Kahaly, Wolfgang H Dillmann, George J Kahaly, 2005, Endocrine reviews.

Modulation of Dynamin-related Protein 1 (DRP1) Function by Increased O-linked-β-N-acetylglucosamine Modification (O-GlcNAc) in Cardiac Myocytes*

J. Yates, T. Gawlowski, R. Schwappacher, 2012, The Journal of Biological Chemistry.

Increased Enzymatic O-GlcNAcylation of Mitochondrial Proteins Impairs Mitochondrial Function in Cardiac Myocytes Exposed to High Glucose*

S. Trauger, W. Dillmann, B. Scott, 2009, Journal of Biological Chemistry.

Mitochondrial Calcium Handling and Heart Disease in Diabetes Mellitus.

J. Díaz-Juárez, W. Dillmann, Jorge A. Suarez, 2020, Biochimica et biophysica acta. Molecular basis of disease.

Induction of HSP70 in Cultured Rat Neonatal Cardiomyocytes by Hypoxia and Metabolic Stress

R. Mestril, K Iwaki, S H Chi, 1993, Circulation.

Thyroid hormone-induced stimulation of the sarcoplasmic reticulum Ca(2+) ATPase gene is inhibited by LIF and IL-6.

B. Gloss, F. Villarreal, A. Moriscot, 2000, American journal of physiology. Endocrinology and metabolism.

Proteomic comparison of human and great ape blood plasma reveals conserved glycosylation and differences in thyroid hormone metabolism.

A. Varki, R. Parekh, Sandra Díaz, 2001, American journal of physical anthropology.

Regulation of mitochondrial morphology and function by O-GlcNAcylation in neonatal cardiac myocytes.

A. Makino, T. Gawlowski, W. Dillmann, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

P3-228 Chip and HSP70 regulate tau ubiquitination, degradation and aggregation

L. Petrucelli, M. Hutton, T. Dawson, 2004, Neurobiology of Aging.

Delineation of three different thyroid hormone-response elements in promoter of rat sarcoplasmic reticulum Ca2+ATPase gene. Demonstration that retinoid X receptor binds 5' to thyroid hormone receptor in response element 1.

C. Glass, M. Lazar, R. Kurokawa, 1994, The Journal of biological chemistry.

Over-expression of inducible HSP70 chaperone suppresses neuropathology and improves motor function in SCA1 mice.

H. Zoghbi, H. Orr, R. Mestril, 2001, Human molecular genetics.

Role of Myotonic Dystrophy Protein Kinase (DMPK) in Glucose Homeostasis and Muscle Insulin Action

D. Catalucci, M. Palacín, A. Zorzano, 2007, PloS one.

Diabetes mellitus and hypothyroidism induce changes in myosin isoenzyme distribution in the rat heart--do alterations in fuel flux mediate these changes?

W. Dillmann, 1986, Advances in experimental medicine and biology.

Myosin isoenzyme distribution and Ca+2-activated myosin ATPase activity in the rat heart is influenced by fructose feeding and triiodothyronine.

W. Dillmann, 1985, Endocrinology.

Fructose feeding increases Ca++-activated myosin ATPase activity and changes myosin isoenzyme distribution in the diabetic rat heart.

W. Dillmann, 1984, Endocrinology.

A physiological dose of triiodothyronine normalizes cardiac myosin adenosine triphosphatase activity and changes myosin isoenzyme distribution in semistarved rats.

W. Dillmann, N. Alexander, S. Berry, 1983, Endocrinology.

Influence of thyroid hormone administration on myosin ATPase activity and myosin isoenzyme distribution in the heart of diabetic rats.

W. Dillmann, 1982, Metabolism: clinical and experimental.

Nuclear receptors and thyroid hormone action: a progress report.

H. Towle, J. Oppenheimer, W. Dillmann, 1979, Federation proceedings.

Hsp72 Overexpression Accelerates the Recovery from Caerulein-Induced Pancreatitis

M. Wagner, M. Lunova, P. Strnad, 2012, PloS one.

Phospholamban: a major determinant of the cardiac force-frequency relationship.

E. Kranias, W F Bluhm, M Meyer, 2000, American journal of physiology. Heart and circulatory physiology.

Characterization of agonist-induced vasoconstriction in mouse pulmonary artery.

K. Akassoglou, J. Yuan, C. V. Remillard, 2008, American journal of physiology. Heart and circulatory physiology.

The Na+/Ca2+ exchanger/SR Ca2+ ATPase transport capacity regulates the contractility of normal and hypertrophied feline ventricular myocytes.

S. Houser, J. Gaughan, B. McGowan, 2005, Journal of cardiac failure.

Cardiac function in thyroid disease: clinical features and management considerations.

W. Dillmann, 1993, The Annals of thoracic surgery.

Intravenous AAV8 Encoding Urocortin-2 Increases Function of the Failing Heart in Mice

A. Miyanohara, M. Gao, T. Guo, 2015, Human gene therapy.

Intravenous adeno-associated virus serotype 8 encoding urocortin-2 provides sustained augmentation of left ventricular function in mice.

N. Dalton, A. Miyanohara, H. Patel, 2013, Human gene therapy.

The rat as a model for cardiovascular disease

W. Dillmann, 2008 .

Biochemical basis of thyroid hormone action in the heart.

W. Dillmann, 1990, The American journal of medicine.

Conditional Up-Regulation of SERCA2a Exacerbates RyR2-Dependent Ventricular and Atrial Arrhythmias

A. A. Armoundas, S. Györke, Bin Liu, 2020, International journal of molecular sciences.

Microbial DNA Enrichment Promotes Adrenomedullary Inflammation, Catecholamine Secretion, and Hypertension in Obese Mice

K. Tang, S. Mahata, W. Dillmann, 2022, Journal of the American Heart Association.

Increased FOG-2 in Failing Myocardium Disrupts Thyroid Hormone–Dependent SERCA2 Gene Transcription

Jing Wu, R. Patten, E. Svensson, 2008, Circulation research.

Cardiac Ion Channel Expression and Contractile Function in Mice with Deletion of Thyroid Hormone Receptor α or β1

W. Giles, B. Gloss, J. Samarut, 2001 .

Surgical management of the thyroid nodule: Patient selection based on the results of fine‐needle aspiration cytology

A. Nahum, M. Bouvet, K. Robbins, 1992, The Laryngoscope.

Integrins protect cardiomyocytes from ischemia/reperfusion injury.

Susan S. Taylor, G. Perkins, J. Copps, 2013, The Journal of clinical investigation.

Rapid effects of insulin on in vitro translational activity of specific mRNA in diabetic rat heart.

Ravi Moysore Shanker, W. Dillmann, W. E. Neeley, 1986, The American journal of physiology.

Messenger RNA content and complexity of euthyroid and hypothyroid rat liver.

H. Towle, J. Oppenheimer, W. Dillmann, 1979, The Journal of biological chemistry.

Effect of diabetes and hypothyroidism on the predominance of cardiac myosin heavy chains synthesized in vivo or in a cell-free system.

W. Dillmann, A. Barrieux, G. S. Reese, 1984, The Journal of biological chemistry.

Protection against myocardial dysfunction after a brief ischemic period in transgenic mice expressing inducible heat shock protein 70.

J. Omens, J. Covell, R. Mestril, 1998, The Journal of clinical investigation.

Diabetes-induced changes of proteins synthesized by adult cardiac myocytes are partially reversed by insulin.

E. Cagliero, W. Dillmann, A. Barrieux, 1988, Journal of molecular and cellular cardiology.

Transgenic Expression of Sarcoplasmic Reticulum Ca2+ ATPase Modifies the Transition From Hypertrophy to Early Heart Failure

W. Manning, B. Lorell, Kenta Ito, 2001, Circulation research.

Overexpression of the sarcoplasmic reticulum Ca(2+)-ATPase improves myocardial contractility in diabetic cardiomyopathy.

D. Belke, W. Bluhm, W. Dillmann, 2002, Diabetes.

Morphine induces physiological, structural, and molecular benefits in the diabetic myocardium

S. Mahata, J. Peart, H. Patel, 2021, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Maintaining Myocardial Glucose Utilization in Diabetic Cardiomyopathy Accelerates Mitochondrial Dysfunction

Chae-Myeong Ha, E. Abel, L. Dell’Italia, 2020, Diabetes.

Integrative and Translational Physiology : Integrative Aspects of Energy Homeostasis and Metabolic Diseases Excess protein O-GlcNAcylation and the progression of diabetic cardiomyopathy

Indroneal Banerjee, S. Ihm, F. Villarreal, 2012 .

Decreased sarcoplasmic reticulum activity and contractility in diabetic db/db mouse heart.

D. Belke, W. Dillmann, E. Swanson, 2004, Diabetes.

Restoring mitochondrial calcium uniporter expression in diabetic mouse heart improves mitochondrial calcium handling and cardiac function

Mohit M. Jain, F. Cividini, J. Díaz-Juárez, 2018, The Journal of Biological Chemistry.

Angiotensin II stimulates the autocrine production of transforming growth factor-β1 in adult rat cardiac fibroblasts

A. McCulloch, F. Villarreal, A. A. Lee, 1995 .

Doxycycline inducible expression of SERCA2a improves calcium handling and reverts cardiac dysfunction in pressure overload-induced cardiac hypertrophy.

B. Gloss, T. Dieterle, J. McDonald, 2004, American journal of physiology. Heart and circulatory physiology.

ISCHEMIA, INFARCTION AND HSP70

R. Mestril, W. Dillmann, 1997 .

Heat shock proteins and protection against myocardial ischemia.

R. Mestril, W. Dillmann, 1995, Journal of molecular and cellular cardiology.

protein 70 transgenic animals with upregulated heat shock Functional properties of skeletal muscle from

J. Zweier, P. Kuppusamy, M. Brotto, 2015 .

Functional properties of skeletal muscle from transgenic animals with upregulated heat shock protein 70.

M. Brotto, R. Mestril, T. Nosek, 2000, Physiological genomics.

Angiotensin II stimulates the autocrine production of transforming growth factor-beta 1 in adult rat cardiac fibroblasts.

A. McCulloch, A. A. Lee, F. Villarreal, 1995, Journal of molecular and cellular cardiology.

Cardiac uptake of minocycline and mechanisms for in vivo cardioprotection.

F. Villarreal, Michael O. Griffin, W. Dillmann, 2008, Journal of the American College of Cardiology.

Combined and Individual Mitochondrial HSP60 and HSP10 Expression in Cardiac Myocytes Protects Mitochondrial Function and Prevents Apoptotic Cell Deaths Induced by Simulated Ischemia-Reoxygenation

I. Scheffler, R. Mestril, Ian Y. Lian, 2001, Circulation.

Overexpression of sarcoplasmic reticulum Ca(2+)-ATPase improves cardiac contractile function in hypothyroid mice.

W F Bluhm, M Meyer, M R Sayen, 1999, Cardiovascular research.

Adenovirus-Mediated Overexpression of O-GlcNAcase Improves Contractile Function in the Diabetic Heart

M. Hoshijima, Ying Hu, D. Belke, 2005, Circulation research.

Glucose Regulation of Load‐Induced mTOR Signaling and ER Stress in Mammalian Heart

M. Entman, D. Glover, M. Gambello, 2013, Journal of the American Heart Association.

The thyroid hormone receptor-beta-selective agonist GC-1 differentially affects plasma lipids and cardiac activity.

B. Gloss, J. Baxter, G. Grover, 2000, Endocrinology.

Heat shock and adaptive response to ischemia.

R. Mestril, W H Dillmann, R Mestril, 1991, Trends in cardiovascular medicine.

Mice Overexpressing 70-kDa Heat Shock Protein Show Increased Resistance to Malonate and 3-Nitropropionic Acid

O. Andreassen, M. Beal, R. Ferrante, 2002, Experimental Neurology.

Thermodynamic limitation for Ca2+ handling contributes to decreased contractile reserve in rat hearts.

J. Halow, V. Figueredo, S. A. Camacho, 1998, American journal of physiology. Heart and circulatory physiology.

Cardiac hypertrophy-induced changes in mRNA levels for TGF-beta 1, fibronectin, and collagen.

F. Villarreal, W. Dillmann, 1992, The American journal of physiology.

Effect of Thyroid Hormone on the Ploidy of Rat Liver Nuclei as Determined by Flow-Cytometry

L. Koss, R. Wolley, J. Oppenheimer, 1978, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

Mechanism of Action of Thyroid Hormone on the Cardiac Vascular System

W. Dillmann, 2009 .

Myocardial remodeling in hypertensive Ren-2 transgenic rats.

J. Omens, D. Mackenna, F. Villarreal, 1995, Hypertension.

Mutant Protects Against Ischemia / Reperfusion Injury in a Transgenic Overexpression of Wild-Type Heat Shock Protein 27 and a Nonphosphorylatable Heat

Jody L. Martin, J. Hollander, D. Belke, 2004 .

Influence of phosphorylation and oligomerization on the protective role of the small heat shock protein 27 in rat adult cardiomyocytes.

R. Mestril, L. Weber, W. Dillmann, 1999, Gene expression.

Specific heat shock proteins protect microtubules during simulated ischemia in cardiac myocytes.

R. Mestril, W. Bluhm, W. Dillmann, 1998, The American journal of physiology.

Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transition.

Steven P Jones, Lewis J. Watson, H. Facundo, 2008, Journal of molecular and cellular cardiology.

Impaired sarcoplasmic reticulum function leads to contractile dysfunction and cardiac hypertrophy.

H. Knot, W. Bluhm, W. Dillmann, 2001, American journal of physiology. Heart and circulatory physiology.

Different configurations of specific thyroid hormone response elements mediate opposite effects of thyroid hormone and GC-1 on gene expression.

B. Gloss, J. Baxter, T. Scanlan, 2005, Endocrinology.

Neuronal Na+ Channels Are Integral Components of Pro-Arrhythmic Na+/Ca2+ Signaling Nanodomain That Promotes Cardiac Arrhythmias During β-Adrenergic Stimulation

Sathya D. Unudurthi, A. A. Armoundas, S. Priori, 2016, JACC. Basic to translational science.

Transcription factor NF-kappa B is necessary for up-regulation of type 1 angiotensin II receptor mRNA in rat cardiac fibroblasts treated with tumor necrosis factor-alpha or interleukin-1 beta.

B. Greenberg, W. Dillmann, D. Gurantz, 2002, The Journal of biological chemistry.

Hexokinase-1 mitochondrial dissociation and protein O-GlcNAcylation drive heart failure with preserved ejection fraction

M. Liu, H. Ardehali, J. Shapiro, 2023 .

A Role for HSP27 in Sensory Neuron Survival

R. Coggeshall, C. Woolf, M. Costigan, 1999, The Journal of Neuroscience.

Sorcin modulates mitochondrial Ca(2+) handling and reduces apoptosis in neonatal rat cardiac myocytes.

W. Dillmann, Hong Wang, P. McDonough, 2013, American journal of physiology. Cell physiology.

In vivo adenoviral transfer of sorcin reverses cardiac contractile abnormalities of diabetic cardiomyopathy.

B. Gloss, L. Brunton, T. Dieterle, 2004, American journal of physiology. Heart and circulatory physiology.

Myocyte protection by 10 kD heat shock protein (Hsp10) involves the mobile loop and attenuation of the Ras GTP‐ase pathway

V. Kao, J. Hollander, W. Dillmann, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

The role of thyroid hormone receptors in the heart.

B. Gloss, W. Dillmann, 2002, Methods in molecular biology.

Altered Cardiac Phenotype in Transgenic Mice Carrying the Δ337 Threonine Thyroid Hormone Receptor β Mutant Derived from the S Family.

B. Gloss, M. R. Sayen, W. Bluhm, 1999, Endocrinology.

Altered cardiac calcium handling in diabetes

D. Belke, W. Dillmann, 2004, Current hypertension reports.

Induction of heat shock proteins by tyrosine kinase inhibitors in rat cardiomyocytes and myogenic cells confers protection against simulated ischemia.

S. Lau, R. Mestril, W. Dillmann, 1997, Journal of molecular and cellular cardiology.

Adenovirus-mediated gene transfer reconstitutes depressed sarcoplasmic reticulum Ca2+-ATPase levels and shortens prolonged cardiac myocyte Ca2+ transients.

M. R. Sayen, F. Giordano, W. Dillmann, 1997, Circulation.

SERCA2a upregulation ameliorates cellular alternans induced by metabolic inhibition.

A. A. Armoundas, M. Scherrer-Crosbie, D. Puppala, 2016, Journal of applied physiology.

Overexpression of the rat inducible 70-kD heat stress protein in a transgenic mouse increases the resistance of the heart to ischemic injury.

D. Yellon, M. Marber, R. Mestril, 1995, The Journal of clinical investigation.

Transcription of the Rat Sarcoplasmic Reticulum Ca2+ Adenosine Triphosphatase Gene Is Increased by 3,5,3′-Triiodothyronine Receptor Isoform-Specific Interactions with the Myocyte-Specific Enhancer Factor-2a1

A. Moriscot, M. R. Sayen, W. Dillmann, 1997 .

Transcription of the Rat Sarcoplasmic Reticulum Ca Adenosine Triphosphatase Gene Is Increased by 3,5,3 (cid:42) Triiodothyronine Receptor Isoform-Specific Interactions with the Myocyte-Specific Enhancer Factor-2a*

A. Moriscot, M. R. Sayen, W. Dillmann, 1996 .

Restoration of coronary microvascular function by OGA overexpression in a high-fat diet with low-dose streptozotocin-induced type 2 diabetic mice

J. Yuan, A. Makino, W. Dillmann, 2023, Diabetes & vascular disease research.

Isolation of a novel inducible rat heat-shock protein (HSP70) gene and its expression during ischaemia/hypoxia and heat shock.

R. Mestril, M. R. Sayen, W. Dillmann, 1994, The Biochemical journal.

Gut lumen-leaked microbial DNA causes myocardial inflammation and impairs cardiac contractility in ageing mouse heart

S. Mahata, W. Dillmann, Wei Ying, 2023, Frontiers in immunology.

Gene transfer of a phospholamban-targeted antibody improves calcium handling and cardiac function in heart failure.

J. Ross, Yusu Gu, K. Peterson, 2005, Cardiovascular research.

Alpha-amanitin administration results in a temporary inhibition of hepatic enzyme induction by triiodothyronine: further evidence favoring a long-lived mediator of thyroid hormone action.

M. Surks, J. Oppenheimer, W. Dillmann, 1977, Endocrinology.

Evaluation of left ventricular mechanical work and energetics of normal hearts in SERCA2a transgenic rats

K. Obata, M. Hirabayashi, Guo-Xing Zhang, 2012, The Journal of Physiological Sciences.

Heat Shock Proteins and the Cardiovascular System

A. Knowlton, W. Dillmann, 1997, Developments in Cardiovascular Medicine.

Methyl palmoxirate increases Ca2+-myosin ATPase activity and changes myosin isoenzyme distribution in the diabetic rat heart.

W. Dillmann, 1985, The American journal of physiology.

cAMP‐ and Ca2+/calmodulin‐dependent protein kinases mediate inotropic, lusitropic and arrhythmogenic effects of urocortin 2 in mouse ventricular myocytes

H. Mächler, B. Pieske, J. Spiess, 2011, British journal of pharmacology.

Regulation of mitochondrial morphology and function by O-GlcNAcylation in neonatal cardiac myocytes.

A. Makino, T. Gawlowski, W. Dillmann, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

Expression of inducible stress protein 70 in rat heart myogenic cells confers protection against simulated ischemia-induced injury.

R. Mestril, M. R. Sayen, W. Dillmann, 1994, The Journal of clinical investigation.

The Thyroid Hormone Receptor-β-Selective Agonist GC-1 Differentially Affects Plasma Lipids and Cardiac Activity.

B. Gloss, T. Scanlan, W. Dillmann, 2000, Endocrinology.

Protection against myocardial dysfunction after a brief ischemic period in transgenic mice expressing inducible heat shock protein 70.

J. Omens, R. Mestril, W. Dillmann, 1998, The Journal of clinical investigation.

Overexpression of inducible heat shock protein (HSP)72 in transgenic mice decreases infarct size

R. Sievers, C. Wolfe, W. Dillmann, 1996 .

Downregulation of connexin40 is associated with coronary endothelial cell dysfunction in streptozotocin-induced diabetic mice.

J. Yuan, A. Makino, O. Platoshyn, 2008, American journal of physiology. Cell physiology.

CALL FOR PAPERS Integrative and Translational Physiology: Integrative Aspects of Energy Homeostasis and Metabolic Diseases Excess protein O-GlcNAcylation and the progression of diabetic cardiomyopathy

Indroneal Banerjee, S. Ihm, W. Dillmann, 2012 .

Thermodynamic limitation for Ca2+ handling contributes to decreased contractile reserve in rat hearts.

R. Tian, W. Dillmann, M. Meyer, 1998, American journal of physiology. Heart and circulatory physiology.

Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network.

D. M. Foster, W. Dillmann, L. Watson, 2013, International journal of cardiology.

CALL FOR PAPERS Cellular Mechanisms of Tissue Fibrosis Role for high-glucose-induced protein O-GlcNAcylation in stimulating cardiac fibroblast collagen synthesis

S. Ihm, Nakon Aroonsakool, F. Villarreal, 2014 .

Sarco/endoplasmic Reticulum Calcium ATPase-2 Expression Is Regulated by ATF6 during the Endoplasmic Reticulum Stress Response

H. Hoover, D. Thuerauf, C. Glembotski, 2001, The Journal of Biological Chemistry.

Sarcoplasmic reticulum Ca2+-ATPase overexpression by adenovirus mediated gene transfer and in transgenic mice

W. Dillmann, M. Meyer, 1998 .

Sarcoplasmic reticulum Ca(2+)-ATPase overexpression by adenovirus mediated gene transfer and in transgenic mice.

W. Dillmann, M. Meyer, 1998, Cardiovascular research.

Endothelin downregulates SERCA2 gene and protein expression in adult rat ventricular myocytes: regulation by pertussis toxin-sensitive Gi protein and cAMP.

L. Brunton, Jody L. Martin, W. Dillmann, 2009, American Journal of Physiology. Heart and Circulatory Physiology.

p75 neurotrophin receptor regulates agonist-induced pulmonary vasoconstriction.

Benjamin D. Sachs, K. Akassoglou, W. Yao, 2008, American journal of physiology. Heart and circulatory physiology.