H. Riedinger

Re-oxygenation of hypoxic simian virus 40 (SV40)-infected CV1 cells causes distinct changes of SV40 minichromosome-associated replication proteins.

H. Riedinger, M. van Betteraey-Nikoleit, H. Probst, 2002, European journal of biochemistry.

Fast Control of DNA Replication in Response to Hypoxia and to Inhibited Protein Synthesis in CCRF-CEM and HeLa Cells

H. Riedinger, H. Probst, G. Probst, 1999, Biological chemistry.

Hypoxia Blocks In Vivo Initiation of Simian Virus 40 Replication at a Stage Preceding Origin Unwinding

H. Riedinger, H. Probst, Maria van Betteraey, 1999, Journal of Virology.

Oxygen-dependent Regulation of in VivoReplication of Simian Virus 40 DNA Is Modulated by Glucose*

H. Riedinger, H. Probst, K. Eisele, 2001, The Journal of Biological Chemistry.

Reversible shutdown of replicon initiation by transient hypoxia in Ehrlich ascites cells. Dependence of initiation on short-lived protein.

V. Gekeler, H. Riedinger, H. Probst, 1992, European journal of biochemistry.

Role of ribonucleotide reductase and deoxynucleotide pools in the oxygen-dependent control of DNA replication in Ehrlich ascites cells.

H. Riedinger, H. Probst, K. Brischwein, 1997, European journal of biochemistry.

Replication of simian virus 40 (SV40) DNA in virus-infected CV1 cells selectively permeabilized for small molecules by Staphylococcus aureus alpha-toxin: involvement of mitochondria in the fast O2-dependent regulation of SV40 DNA replication.

K. Trummler, H. Riedinger, H. Probst, 2005, The Biochemical journal.

No significant overreplication occurs in Ehrlich ascites cells during and after reversal of hypoxia.

V. Gekeler, H. Riedinger, H. Probst, 1989, Experimental cell research.