Da-zhong Xu

发表

HIF-1 mediates pathogenic inflammatory responses to intestinal ischemia-reperfusion injury.

G. Semenza, R. Feinman, E. Deitch, 2010, American journal of physiology. Gastrointestinal and liver physiology.

Adenosine A2A receptor activation reduces lung injury in trauma/hemorrhagic shock*

E. Deitch, Z. Nemeth, G. Haskó, 2006, Critical care medicine.

TRAUMA-SHOCK-INDUCED GUT INJURY AND THE PRODUCTION OF BIOLOGICALLY ACTIVE INTESTINAL LYMPH IS ABROGATED BY CASTRATION IN A LARGE ANIMAL PORCINE MODEL

E. Deitch, M. Senthil, A. Watkins, 2007, Shock.

PERSISTENT HIF-1α ACTIVATION IN GUT ISCHEMIA/REPERFUSION INJURY: POTENTIAL ROLE OF BACTERIA AND LIPOPOLYSACCHARIDE

R. Feinman, E. Deitch, M. Condon, 2004, Shock.

Hypoxia-inducible factor plays a gut-injurious role in intestinal ischemia reperfusion injury.

G. Semenza, D. Palange, R. Feinman, 2011, American journal of physiology. Gastrointestinal and liver physiology.

Gut lymph hypothesis of early shock and trauma-induced multiple organ dysfunction syndrome: A new look at gut origin sepsis

E. Deitch, Q. Lu, Da-zhong Xu, 2006 .

Relationship between disruption of the unstirred mucus layer and intestinal restitution in loss of gut barrier function after trauma hemorrhagic shock.

L. Magnotti, E. Deitch, A. Watkins, 2007, Surgery.

Na+,K+-ATPase activity is inhibited in cultured intestinal epithelial cells by endotoxin or nitric oxide.

C. Szabó, E. Deitch, G. Haskó, 2005, International journal of molecular medicine.

Trauma-hemorrhagic shock-induced pulmonary epithelial and endothelial cell injury utilizes different programmed cell death signaling pathways.

R. Feinman, E. Deitch, A. Watkins, 2009, American journal of physiology. Lung cellular and molecular physiology.

Intravenous Injection of Trauma-Hemorrhagic Shock Mesenteric Lymph Causes Lung Injury That Is Dependent Upon Activation of the Inducible Nitric Oxide Synthase Pathway

R. Feinman, E. Deitch, M. Senthil, 2007, Annals of surgery.

Loss of the Intestinal Mucus Layer in the Normal Rat Causes Gut Injury But Not Toxic Mesenteric Lymph nor Lung Injury

D. Palange, E. Deitch, Q. Lu, 2010, Shock.

Albumin peptide: A molecular marker for trauma/hemorrhagic-shock in rat mesenteric lymph

E. Deitch, M. Senthil, Z. Sifri, 2005, Peptides.

Trauma/Hemorrhagic Shock Mesenteric Lymph Upregulates Adhesion Molecule Expression and IL-6 Production in Human Umbilical Vein Endothelial Cells

E. Deitch, J. Sambol, G. Haskó, 2002, Shock.

Hemorrhagic shock induces endothelial cell apoptosis, which is mediated by factors contained in mesenteric lymph

E. Deitch, G. Haskó, Q. Lu, 2004, Critical care medicine.

A study of the biologic activity of trauma-hemorrhagic shock mesenteric lymph over time and the relative role of cytokines.

E. Deitch, Z. Nemeth, A. Osband, 2004, Surgery.

MESENTERIC LYMPH DUCT LIGATION IMPROVES SURVIVAL IN A LETHAL SHOCK MODEL

R. Feinman, E. Deitch, M. Senthil, 2008, Shock.

The influence of the type of resuscitation fluid on gut injury and distant organ injury in a rat model of trauma/hemorrhagic shock.

E. Deitch, A. Watkins, F. Caputo, 2008, The Journal of trauma.

RESISTANCE OF THE FEMALE, AS OPPOSED TO THE MALE, INTESTINE TO I/R-MEDIATED INJURY IS ASSOCIATED WITH INCREASED RESISTANCE TO GUT-INDUCED DISTANT ORGAN INJURY

E. Deitch, D. Livingston, C. Hauser, 2007, Shock.

Mesenteric lymph duct ligation attenuates lung injury and neutrophil activation after intraperitoneal injection of endotoxin in rats.

E. Deitch, A. Watkins, F. Caputo, 2008, The Journal of trauma.

Amiloride combined with small-volume resuscitation with hypertonic saline is superior in ameliorating trauma-hemorrhagic shock-induced lung injury in rats to the administration of either agent alone

E. Deitch, G. Haskó, S. Zaets, 2005, Critical care medicine.

TRAUMA-HEMORRHAGIC SHOCK MESENTERIC LYMPH FROM RAT CONTAINS A MODIFIED FORM OF ALBUMIN THAT IS IMPLICATED IN ENDOTHELIAL CELL TOXICITY

E. Deitch, S. Zaets, Z. Sifri, 2005, Shock.

Pancreatic Duct Ligation Reduces Lung Injury Following Trauma and Hemorrhagic Shock

L. Magnotti, E. Deitch, S. Zaets, 2004, Annals of surgery.

THE ROLE OF LYMPH FACTORS IN LUNG INJURY, BONE MARROW SUPPRESSION, AND ENDOTHELIAL CELL DYSFUNCTION IN A PRIMATE MODEL OF TRAUMA-HEMORRHAGIC SHOCK

R. Forsythe, H. Redl, E. Deitch, 2004, Shock.

Albumin Protects Against Gut-Induced Lung Injury In Vitro and In Vivo

E. Deitch, C. Hauser, A. Osband, 2004, Annals of surgery.

Trauma-Hemorrhagic Shock Mesenteric Lymph Induces Endothelial Apoptosis That Involves Both Caspase-Dependent and Caspase-Independent Mechanisms

E. Deitch, Z. Nemeth, L. Gaspers, 2004, Annals of surgery.

Trauma-hemorrhagic shock-induced up-regulation of endothelial cell adhesion molecules is blunted by mesenteric lymph duct ligation

E. Deitch, A. Issekutz, Q. Lu, 2004, Critical care medicine.

The Role of Oxidant-Mediated Pathways in the Cytotoxicity of Endothelial Cells Exposed to Mesenteric Lymph from Rats Subjected to Trauma-Hemorrhagic Shock

E. Deitch, A. Osband, S. Zaets, 2003, Shock.

Serine Proteases Are Involved in the Pathogenesis of Trauma-Hemorrhagic Shock-Induced Gut and Lung Injury

E. Deitch, H. Shi, Q. Lu, 2003, Shock.

Hypertonic Saline Resuscitation Limits Neutrophil Activation After Trauma-Hemorrhagic Shock

E. Deitch, C. Hauser, H. Shi, 2003, Shock.

Mesenteric lymph duct ligation prevents shock-induced RBC deformability and shape changes.

E. Deitch, S. Zaets, T. Berezina, 2003, The Journal of surgical research.

Trauma-Hemorrhage-Induced Neutrophil Priming Is Prevented by Mesenteric Lymph Duct Ligation

John M. Adams, E. Deitch, J. Sambol, 2002, Shock.

Hypertonic Saline Improves Intestinal Mucosa Barrier Function and Lung Injury After Trauma-Hemorrhagic Shock

E. Deitch, C. Hauser, H. Shi, 2002, Shock.

Entry of gut lymph into the circulation primes rat neutrophil respiratory burst in hemorrhagic shock

John M. Adams, E. Deitch, D. Livingston, 2001, Critical care medicine.

Hematopoietic failure after hemorrhagic shock is mediated partially through mesenteric lymph

R. Forsythe, P. Rameshwar, E. Deitch, 2001, Critical care medicine.

LYMPH FROM A PRIMATE BABOON TRAUMA HEMORRHAGIC SHOCK MODEL ACTIVATES HUMAN NEUTROPHILS

John M. Adams, R. Forsythe, H. Redl, 2006, Shock.

CIRBP promotes ferroptosis by interacting with ELAVL1 and activating ferritinophagy during renal ischaemia‐reperfusion injury

You‐hua Zhu, Wen‐yu Zhao, Lei Zhang, 2021, Journal of cellular and molecular medicine.

Trauma Hemorrhagic Shock-Induced Lung Injury Involves a Gut-Lymph-Induced TLR4 Pathway in Mice

D. Palange, R. Feinman, E. Deitch, 2011, PLoS ONE.

The female intestine is more resistant than the male intestine to gut injury and inflammation when subjected to conditions associated with shock states.

R. Feinman, E. Deitch, Q. Lu, 2005, American journal of physiology. Gastrointestinal and liver physiology.

Acute Lung Injury after Hemorrhagic Shock is Dependent on Gut Injury and Sex

Da-zhong Xu, C. Adams, Magnotti Louis J., 2000, The American surgeon.

Recombinant factor XIII mitigates hemorrhagic shock-induced organ dysfunction.

E. Deitch, S. Zaets, T. Berezina, 2011, The Journal of surgical research.

Neutrophil activation is modulated by sex hormones after trauma-hemorrhagic shock and burn injuries.

E. Deitch, C. Hauser, P. Ananthakrishnan, 2006, American journal of physiology. Heart and circulatory physiology.

Does Recombinant Factor XIII Eliminate Early Manifestations of Multiple-Organ Injury After Experimental Burn Similarly to Gut Ischemia-Reperfusion Injury or Trauma-Hemorrhagic Shock?

E. Deitch, S. Zaets, T. Berezina, 2014, Journal of burn care & research : official publication of the American Burn Association.

Vagal nerve stimulation modulates gut injury and lung permeability in trauma-hemorrhagic shock

D. Palange, E. Deitch, L. Ulloa, 2012, The journal of trauma and acute care surgery.

Amiloride moderates increased gut permeability and diminishes mesenteric lymph-mediated priming of neutrophils in trauma/hemorrhagic shock.

E. Deitch, G. Haskó, Q. Lu, 2006, Surgery.

Sex hormones modulate distant organ injury in both a trauma/hemorrhagic shock model and a burn model.

E. Deitch, P. Ananthakrishnan, Q. Lu, 2005, Surgery.

Female sex hormones protect red blood cells from damage after trauma-hemorrhagic shock.

E. Deitch, S. Zaets, T. Berezina, 2004, Surgical infections.

Mesenteric lymph from burned rats induces endothelial cell injury and activates neutrophils

J. Skurnick, E. Deitch, H. Shi, 2004, Critical care medicine.

Sexual dimorphism in the activation of neutrophils by shock mesenteric lymph.

John M. Adams, E. Deitch, D. Livingston, 2003, Surgical infections.

Small volume resuscitation with hypertonic saline is more effective in ameliorating trauma-hemorrhagic shock-induced lung injury, neutrophil activation and red blood cell dysfunction than pancreatitic protease inhibition.

E. Deitch, S. Zaets, T. Berezina, 2005, The Journal of trauma.

Trauma-hemorrhagic shock-induced red blood cell damage leads to decreased microcirculatory blood flow*

E. Deitch, Z. Spolarics, S. Zaets, 2009, Critical care medicine.

Effect of Trauma-Hemorrhagic Shock on Red Blood Cell Deformability and Shape

E. Deitch, Z. Spolarics, S. Zaets, 2003, Shock.

Hemorrhagic shock induced up-regulation of P-selectin expression is mediated by factors in mesenteric lymph and blunted by mesenteric lymph duct interruption.

E. Deitch, J. Sambol, D. Granger, 2001, The Journal of trauma.

Gut-derived mesenteric lymph but not portal blood increases endothelial cell permeability and promotes lung injury after hemorrhagic shock.

L. Magnotti, E. Deitch, J. Upperman, 1998, Annals of surgery.

The effect of hypoxia/reoxygenation on the cellular function of intestinal epithelial cells.

E. Deitch, Q. Lu, Da-zhong Xu, 1999, The Journal of trauma.

A time course study of the protective effect of mesenteric lymph duct ligation on hemorrhagic shock-induced pulmonary injury and the toxic effects of lymph from shocked rats on endothelial cell monolayer permeability.

E. Deitch, Q. Lu, Da-zhong Xu, 2001, Surgery.

Elemental diet-induced immune suppression is caused by both bacterial and dietary factors.

R. Berg, E. Deitch, Da-zhong Xu, 1993, JPEN. Journal of parenteral and enteral nutrition.

Hypoxia-inducible factor plays a gut-injurious role in intestinal ischemia reperfusion injury.

G. Semenza, D. Palange, R. Feinman, 2011, American journal of physiology. Gastrointestinal and liver physiology.

Acute Lung Injury after Hemorrhagic Shock is Dependent on Gut Injury and Sex

Da-zhong Xu, C. Adams, Magnotti Louis J., 2000, The American surgeon.

Calcium and phospholipase A2 appear to be involved in the pathogenesis of hemorrhagic shock-induced mucosal injury and bacterial translocation.

E. Deitch, Q. Lu, Da-zhong Xu, 1995, Critical care medicine.

Mesenteric lymph duct ligation provides long term protection against hemorrhagic shock-induced lung injury.

L. Magnotti, E. Deitch, J. Sambol, 2000, Shock.

Effect of heat shock and endotoxin stress on enterocyte viability apoptosis and function varies based on whether the cells are exposed to heat shock or endotoxin first.

E. Deitch, Q. Lu, Da-zhong Xu, 1996, Archives of surgery.