Akiko Hayashi

Expression of hypoxia-inducible factor 1alpha, hypoxia-inducible factor 2alpha, and von Hippel-Lindau protein in epithelial ovarian neoplasms and allelic loss of von Hippel-Lindau gene: nuclear expression of hypoxia-inducible factor 1alpha is an independent prognostic factor in ovarian carcinoma.

G. Melillo, I. Konishi, Norihiko Kikuchi, 2007, Human pathology.

Changes in the expression of E-cadherin repressors, Snail, Slug, SIP1, and Twist, in the development and progression of ovarian carcinoma: the important role of Snail in ovarian tumorigenesis and progression

T. Shiozawa, I. Konishi, Norihiko Kikuchi, 2009, Medical Molecular Morphology.

Overexpression of RhoA enhances peritoneal dissemination: RhoA suppression with Lovastatin may be useful for ovarian cancer

T. Nikaido, I. Konishi, Norihiko Kikuchi, 2008, Cancer science.

Type‐specific roles of histone deacetylase (HDAC) overexpression in ovarian carcinoma: HDAC1 enhances cell proliferation and HDAC3 stimulates cell migration with downregulation of E‐cadherin

T. Shiozawa, A. Suzuki, I. Konishi, 2010, International journal of cancer.

A Recurrent Mucinous Neoplasm Originating From an Ovarian Mucinous Cystadenoma After an Adnexectomy As the First Procedure: A Case Report

Akiko Hayashi, S. Ohira, Kyoko Tanaka, 2022, Cureus.

Hypoxia upregulates ovarian cancer invasiveness via the binding of HIF‐1α to a hypoxia‐induced, methylation‐free hypoxia response element of S100A4 gene

T. Shiozawa, I. Konishi, Norihiko Kikuchi, 2012, International journal of cancer.

Involvement of pelvic inflammation-related mismatch repair abnormalities and microsatellite instability in the malignant transformation of ovarian endometriosis.

T. Shiozawa, T. Miyamoto, A. Suzuki, 2012, Human pathology.

Type‐specific roles of histone deacetylase (HDAC) overexpression in ovarian carcinoma: HDAC1 enhances cell proliferation and HDAC3 stimulates cell migration with downregulation of E‐cadherin

T. Shiozawa, A. Suzuki, I. Konishi, 2010, International journal of cancer.