B. Moore

发表

IL-10 Alters Immunoproteostasis in APP Mice, Increasing Plaque Burden and Worsening Cognitive Behavior

Nathan D. Price, James A. Eddy, Pritam Das, 2015, Neuron.

TLR5 decoy receptor as a novel anti-amyloid therapeutic for Alzheimer’s disease

Cory C. Funk, N. Price, D. Dickson, 2018, The Journal of experimental medicine.

A novel panel of α-synuclein antibodies reveal distinctive staining profiles in synucleinopathies

T. Golde, Paramita Chakrabarty, B. Giasson, 2017, PloS one.

BRI2 (ITM2b) Inhibits Aβ Deposition In Vivo

D. Dickson, S. Younkin, F. Zou, 2008, The Journal of Neuroscience.

Organotypic brain slice cultures to model neurodegenerative proteinopathies

Hunter S. Futch, T. Golde, C. Croft, 2019, Molecular Neurodegeneration.

Generation and characterization of new monoclonal antibodies targeting the PHF1 and AT8 epitopes on human tau

T. Golde, Paramita Chakrabarty, Y. Levites, 2017, Acta Neuropathologica Communications.

Aß40 mutants protect Drosophila against Aß42 toxicity despite their amyloidogenic properties in the non-transgenic mouse brain

Michael A. Smith, T. Golde, P. Cruz, 2020 .

Steroids as γ‐secretase modulators

T. Golde, E. Koo, K. Felsenstein, 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Rationally designed dehydroalanine (ΔAla)‐containing peptides inhibit amyloid‐β (Aβ) peptide aggregation

T. Rosenberry, Vijayaraghavan Rangachari, Ghulam M. Maharvi, 2009, Biopolymers.

Cytosolic proteins lose solubility as amyloid deposits in a transgenic mouse model of Alzheimer-type amyloidosis.

D. Borchelt, S. Stevens, B. Moore, 2013, Human molecular genetics.

Microglia show differential transcriptomic response to Aβ peptide aggregates ex vivo and in vivo

Cory C. Funk, M. Allen, Xue Wang, 2021, Life Science Alliance.

The 5th International Conference on Molecular Neurodegeneration: Overlapping Pathologies and Common Mechanisms

Cory C. Funk, Hunter S. Futch, A. Levey, 2019, Molecular Neurodegeneration.

Reversible Pathologic and Cognitive Phenotypes in an Inducible Model of Alzheimer-Amyloidosis

D. Borchelt, A. Savonenko, T. Golde, 2013, The Journal of Neuroscience.

An anti-CRF antibody suppresses the HPA axis and reverses stress-induced phenotypes

Hunter S. Futch, T. Golde, P. Cruz, 2019, The Journal of experimental medicine.

Substrate-targeting γ-secretase modulators

M. Wolfe, D. Walsh, T. Golde, 2008, Nature.

IL-10 Alters Immunoprote ostasis in APP Mice , Increasing Plaque Burden and Worsening Cognitive Behavior Highlights

James A. Eddy, Cory C. Funk, N. Price, 2015 .

Retention in Endoplasmic Reticulum 1 (RER1) Modulates Amyloid-β (Aβ) Production by Altering Trafficking of γ-Secretase and Amyloid Precursor Protein (APP)*

T. Golde, K. Felsenstein, Hyo-Jin Park, 2012, The Journal of Biological Chemistry.

Short Aβ peptides attenuate Aβ42 toxicity in vivo

D. Borchelt, T. Golde, P. Cruz, 2018, The Journal of experimental medicine.

Remodeling Alzheimer-amyloidosis models by seeding

D. Borchelt, B. Moore, B. Ulm, 2021, Molecular neurodegeneration.

Overlapping profiles of Aβ peptides in the Alzheimer's disease and pathological aging brains

D. Dickson, T. Golde, Paramita Chakrabarty, 2012, Alzheimer's Research & Therapy.

Divergent effects of the H50Q and G51D SNCA mutations on the aggregation of α‐synuclein

T. Golde, B. Giasson, N. Rutherford, 2014, Journal of neurochemistry.

Soluble brain homogenates from diverse human and mouse sources preferentially seed diffuse Aβ plaque pathology when injected into newborn mouse hosts.

Hunter S. Futch, D. Borchelt, T. Golde, 2021, Free neuropathology.

Biophysical analyses of synthetic amyloid-beta(1-42) aggregates before and after covalent cross-linking. Implications for deducing the structure of endogenous amyloid-beta oligomers.

T. Rosenberry, N. Milkovic, Vijayaraghavan Rangachari, 2009, Biochemistry.

Overlapping profiles of Aβ peptides in the Alzheimer's disease and pathological aging brains

D. Dickson, T. Golde, Paramita Chakrabarty, 2012, Alzheimer's Research & Therapy.

Aß40 displays amyloidogenic properties in the non-transgenic mouse brain but does not exacerbate Aß42 toxicity in Drosophila

Michael A. Smith, T. Golde, P. Cruz, 2020, Alzheimer's Research & Therapy.

Secondary Structure and Interfacial Aggregation of Amyloid- â(1 40) on Sodium Dodecyl Sulfate Micelles

T. Rosenberry, Vijayaraghavan Rangachari, B. Moore, 2022 .

Amyloid-beta(1-42) rapidly forms protofibrils and oligomers by distinct pathways in low concentrations of sodium dodecylsulfate.

T. Rosenberry, Vijayaraghavan Rangachari, D. Hartigan, 2007, Biochemistry.

Organotypic brain slice cultures to model neurodegenerative proteinopathies

T. Golde, H. Futch, B. Moore, 2019, Molecular Neurodegeneration.

Generation and characterization of new monoclonal antibodies targeting the PHF1 and AT8 epitopes on human tau

T. Golde, Paramita Chakrabarty, Y. Levites, 2017, Acta neuropathologica communications.

Lysine 624 of the Amyloid Precursor Protein (APP) Is a Critical Determinant of Amyloid β Peptide Length

T. Golde, B. Cottrell, E. Koo, 2011, The Journal of Biological Chemistry.

BRI2 (ITM2b) Inhibits Aβ Deposition In Vivo

D. Dickson, S. Younkin, F. Zou, 2008, Journal of Neuroscience.

Secondary structure and interfacial aggregation of amyloid-beta(1-40) on sodium dodecyl sulfate micelles.

T. Rosenberry, Vijayaraghavan Rangachari, B. Moore, 2006, Biochemistry.

Novel monoclonal antibodies targeting the microtubule-binding domain of human tau

T. Golde, Paramita Chakrabarty, Y. Levites, 2018, PloS one.