Kazuo Sakamoto

发表

The direction and role of phenotypic transition between podocytes and parietal epithelial cells in focal segmental glomerulosclerosis.

I. Pastan, M. Nagata, T. Matsusaka, 2014, American journal of physiology. Renal physiology.

Long Dialysis Time is the More Important Factor of Erythropoietin Response in Hemodialysis Patients with Diabetes than Kt/V

T. Kitazono, K. Tsuruya, Kazuo Sakamoto, 2014 .

Biphasic MIF and SDF1 expression during podocyte injury promote CD44-mediated glomerular parietal cell migration in focal segmental glomerulosclerosis.

M. Nagata, T. Matsusaka, Kazuo Sakamoto, 2020, American journal of physiology. Renal physiology.

Glomerular filtrate affects the dynamics of podocyte detachment in a model of diffuse toxic podocytopathy.

M. Nagata, T. Matsusaka, Kazuo Sakamoto, 2021, Kidney international.

Podocyte injury-driven intracapillary plasminogen activator inhibitor type 1 accelerates podocyte loss via uPAR-mediated β1-integrin endocytosis.

I. Pastan, T. Miyata, M. Nagata, 2015, American journal of physiology. Renal physiology.

Podocyte injury-driven lipid peroxidation accelerates the infiltration of glomerular foam cells in focal segmental glomerulosclerosis.

I. Pastan, A. Fukamizu, M. Nagata, 2015, The American journal of pathology.