N. H. Smith

Intrauterine origins of metabolic disease

S. Ozanne, N. H. Smith, 2006 .

Poor maternal nutrition leads to alterations in oxidative stress, antioxidant defense capacity, and markers of fibrosis in rat islets: potential underlying mechanisms for development of the diabetic phenotype in later life

Richard H. Jones, S. Ozanne, J. Tarry-Adkins, 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

The imprinted Mir483 is a growth suppressor and metabolic regulator functioning through IGF1

K. Siddle, I. Sandovici, D. Cimadomo, 2022, bioRxiv.

Adverse effects of reduced oxygen tension on the proliferative capacity of rat kidney and insulin-secreting cell lines involve DNA damage and stress responses

S. Ozanne, J. Tarry-Adkins, R. H. Jones, 2008, Experimental cell research.

Maternal diet and aging alter the epigenetic control of a promoter–enhancer interaction at the Hnf4a gene in rat pancreatic islets

Yoko Ito, Yoko Ito, C. Ling, 2011, Proceedings of the National Academy of Sciences.

Ageing is associated with molecular signatures of inflammation and type 2 diabetes in rat pancreatic islets

I. Sandovici, S. Andrews, G. Yeo, 2015, Diabetologia.

Overexpression of Igf2-derived Mir483 inhibits Igf1 expression and leads to developmental growth restriction and metabolic dysfunction in mice.

I. Sandovici, D. Ferland-McCollough, D. Fernandez‐Twinn, 2024, Cell reports.