Melissa X. Shi

Robust autoactivation for apoptosis by BAK but not BAX highlights BAK as an important therapeutic target

Rachel T. Uren, Jerry M. Adams, G. Dewson, 2020, Cell Death & Disease.

BH3 mimetic drugs cooperate with Temozolomide, JQ1 and inducers of ferroptosis in killing glioblastoma multiforme cells

A. Strasser, A. Bush, R. Kluck, 2022, Cell Death & Differentiation.

Intact TP53 function is essential for sustaining durable responses to BH3-mimetic drugs in leukemias.

A. Strasser, A. Roberts, A. Wei, 2021, Blood.

Robust autoactivation for apoptosis by BAK but not BAX highlights BAK as an important therapeutic target

Rachel T. Uren, Jerry M. Adams, G. Dewson, 2020, Cell Death & Disease.

Ensemble Properties of Bax Determine Its Function.

P. Colman, J. Sandow, R. Kluck, 2018, Structure.

Disordered clusters of Bak dimers rupture mitochondria during apoptosis

Rachel T. Uren, G. Dewson, R. Kluck, 2016, bioRxiv.

Structure of the BAK-activating antibody 7D10 bound to BAK reveals an unexpected role for the α1-α2 loop in BAK activation

Michelle S. Miller, Brian J. Smith, P. Colman, 2022, Cell Death & Differentiation.

Detection of differentially expressed genes in lymphomas using cDNA arrays: identification of clusterin as a new diagnostic marker for anaplastic large-cell lymphomas.

E. Jaffe, A. Strasser, M. Raffeld, 2000, Blood.