N. Pore

发表

Lipid perturbation by corticosteroids: an anisotropic study

A. Ghosh, R. Basu, P. Nandy, 1996 .

Blocking ADAM17 Function with a Monoclonal Antibody Improves Sepsis Survival in a Murine Model of Polymicrobial Sepsis

Robert Hullsiek, B. Walcheck, H. Mishra, 2020, International journal of molecular sciences.

Regulation of glut1 mRNA by Hypoxia-inducible Factor-1

F. Ismail-Beigi, A. Maity, N. Pore, 2001, The Journal of Biological Chemistry.

M-CSF Signals through the MAPK/ERK Pathway via Sp1 to Induce VEGF Production and Induces Angiogenesis In Vivo

A. Maity, Yijie Wang, C. Marsh, 2008, PloS one.

Expression of the antibacterial peptide, cecropin, in cultured mammalian cells

S. Pal, N. Pore, 2004, Biotechnology Letters.

Anti-ADAM17 monoclonal antibody MEDI3622 increases IFNγ production by human NK cells in the presence of antibody-bound tumor cells

B. Walcheck, H. Mishra, N. Pore, 2018, Cancer Immunology, Immunotherapy.

Activation of ADAM17 by IL-15 Limits Human NK Cell Proliferation

Jeffrey S. Miller, B. Walcheck, H. Mishra, 2021, Frontiers in Immunology.

PTEN mutation and epidermal growth factor receptor activation regulate vascular endothelial growth factor (VEGF) mRNA expression in human glioblastoma cells by transactivating the proximal VEGF promoter.

Amit Maity, D. O’Rourke, A. Maity, 2003, Cancer research.

InVivo Loss of Function Screening Reveals Carbonic Anhydrase IX as a Key Modulator of Tumor Initiating Potential in Primary Pancreatic Tumors

A. Scarpa, R. Hollingsworth, B. Higgs, 2015, Neoplasia.

Akt1 Activation Can Augment Hypoxia-Inducible Factor-1α Expression by Increasing Protein Translation through a Mammalian Target of Rapamycin–Independent Pathway

E. Bernhard, A. Maity, G. Kao, 2006, Molecular Cancer Research.

Sp1 is involved in Akt-mediated induction of VEGF expression through an HIF-1-independent mechanism.

D. O’Rourke, D. Stokoe, G. Pagès, 2004, Molecular biology of the cell.

Phosphatase and tensin homologue deficiency in glioblastoma confers resistance to radiation and temozolomide that is reversed by the protease inhibitor nelfinavir.

S. Hahn, E. Bernhard, A. Maity, 2007, Cancer research.

HIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cells.

A. Maity, G. Cerniglia, Anjali K. Gupta, 2006, Neoplasia.

Nelfinavir down-regulates hypoxia-inducible factor 1alpha and VEGF expression and increases tumor oxygenation: implications for radiotherapy.

S. Hahn, E. Bernhard, A. Maity, 2006, Cancer research.

In Vivo Loss of Function Screening Reveals Carbonic Anhydrase IX as a Key Modulator of Tumor Initiating Potential in Primary Pancreatic

A. Scarpa, R. Hollingsworth, B. Higgs, 2015 .

Epidermal growth factor receptor transcriptionally up-regulates vascular endothelial growth factor expression in human glioblastoma cells via a pathway involving phosphatidylinositol 3'-kinase and distinct from that induced by hypoxia.

D. O’Rourke, A. Maity, D. Solomon, 2000, Cancer research.

Epidermal Growth Factor Receptor Transcriptionally UpRegulates Vascular Endothelial Growth Factor Expression in Human Glioblastoma Cells via a Pathway Involving Phosphatidylinositol 3 *-Kinase and Distinct from That Induced by Hypoxia 1

D. O’Rourke, A. Maity, D. Solomon, 2000 .

Resistance to durvalumab and durvalumab plus tremelimumab is associated with functional STK11 mutations in non-small-cell lung cancer patients and is reversed by STAT3 knockdown.

Ricardo J. Miragaia, Y. Shrestha, Song Wu, 2021, Cancer discovery.

Resistance to Durvalumab and Durvalumab plus Tremelimumab Is Associated with Functional STK11 Mutations in Patients with Non–Small Cell Lung Cancer and Is Reversed by STAT3 Knockdown

Ricardo J. Miragaia, Y. Shrestha, J. Barrett, 2021 .

EGFR tyrosine kinase inhibitors decrease VEGF expression by both hypoxia-inducible factor (HIF)-1-independent and HIF-1-dependent mechanisms.

A. Maity, G. Kao, G. Cerniglia, 2006, Cancer research.

Regulation of histone deacetylase 4 expression by the SP family of transcription factors.

A. Maity, G. Kao, M. Dowling, 2005, Molecular biology of the cell.

Regulation of histone deacetylase 4 ( HDAC 4 ) expression by the SP family of transcription factors

A. Maity, G. Kao, M. Dowling, 2005 .

Phase 1, First-in-Human Study of MEDI2228, a BCMA-Targeted ADC in Patients with Relapsed/Refractory Multiple Myeloma

M. Dimopoulos, Shaji K. Kumar, A. Yee, 2020 .

HIV Protease Inhibitors Decrease VEGF / HIF-1 A Expression and Angiogenesis in Glioblastoma Cells 1

A. Maity, G. Cerniglia, Anjali K. Gupta, 2006 .

Epidermal Growth Factor Receptor Inhibition Modulates the Microenvironment by Vascular Normalization to Improve Chemotherapy and Radiotherapy Efficacy

Arjun G. Yodh, Chandra M. Sehgal, Regine Choe, 2009, PloS one.

Implications for Radiotherapy VEGF Expression and Increases Tumor Oxygenation : and α Nelfinavir Down-regulates Hypoxia-Inducible Factor 1

S. Hahn, E. Bernhard, A. Maity, 2006 .

Vascular endothelial growth factor and tumor immune microenvironment

N. Pore, 2021 .

Discovery and Development of MEDI7247, a Novel Pyrrolobenzodiazepine (PBD)-Based Antibody Drug Conjugate Targeting ASCT2, for Treating Hematological Cancers

E. Hurt, M. Rebelatto, R. Tammali, 2018, Blood.

The chemokine receptor CXCR4: A homing device for hypoxic cancer cells?

A. Maity, N. Pore, 2006, Cancer biology & therapy.

Preclinical radiolabeling, in vivo biodistribution and positron emission tomography of a novel pyrrolobenzodiazepine (PBD)-based antibody drug conjugate targeting ASCT2.

S. Coats, S. Gong, H. Gan, 2023, Nuclear medicine and biology.