Han Li

发表

Deletion of Individual Ku Subunits in Mice Causes an NHEJ-Independent Phenotype Potentially by Altering Apurinic/Apyrimidinic Site Repair

Y. Choi, E. Reiling, P. Hasty, 2014, PloS one.

Ku80-deleted cells are defective at base excision repair.

P. Hasty, T. Marple, Han Li, 2013, Mutation research.

Non-homologous end joining, but not homologous recombination, enables survival for cells exposed to a histone deacetylase inhibitor

P. Hasty, T. Marple, Han Li, 2005, Nucleic acids research.

A genotoxic screen: rapid analysis of cellular dose-response to a wide range of agents that either damage DNA or alter genome maintenance pathways.

P. Hasty, T. Marple, Han Li, 2004, Mutation research.

As3MT-mediated SAM consumption, which inhibits the methylation of histones and LINE1, is involved in arsenic-induced male reproductive damage.

F. Chen, Qizhan Liu, Yongyue Wei, 2022, Environmental pollution.

Deletion of Ku70, Ku80, or Both Causes Early Aging without Substantially Increased Cancer

H. Vogel, P. Hasty, V. Holcomb, 2007, Molecular and Cellular Biology.

DNA double-strand breaks: A potential causative factor for mammalian aging?

P. Hasty, J. Mitchell, Han Li, 2008, Mechanisms of Ageing and Development.

SIRT1/PGC-1α is involved in arsenic-induced male reproductive damage through mitochondrial dysfunction, which is blocked by the antioxidative effect of zinc.

F. Chen, Qizhan Liu, Yongyue Wei, 2023, Environmental pollution.

Trex2 Enables Spontaneous Sister Chromatid Exchanges Without Facilitating DNA Double-Strand Break Repair

Jeremy M. Stark, J. Stark, P. Hasty, 2011, Genetics.

DNA double-strand breaks: A potential causative factor for mammalian aging?

P. Hasty, Han Li, James R. Mitchell, 2008, Mechanisms of Ageing and Development.