M. Soleimani

M. Soleimani, 2015, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

SLC26 Cl−/HCO3− Exchangers in the Kidney: Roles in Health and Disease

M. Soleimani, 2013, Kidney international.

Double Knockout of Carbonic Anhydrase II (CAII) and Na+-Cl- Cotransporter (NCC) Causes Salt Wasting and Volume Depletion

M. Soleimani, Sharon Barone, Jie Xu, 2013, Cellular Physiology and Biochemistry.

A novel target for diuretic therapy.

M. Soleimani, 2012, Iranian journal of kidney diseases.

A Novel Diuretic Regimen — Soleimani 420 Iranian

M. Soleimani, 2012 .

Double knockout of pendrin and Na-Cl cotransporter (NCC) causes severe salt wasting, volume depletion, and renal failure

G. Shull, M. Soleimani, H. Amlal, 2012, Proceedings of the National Academy of Sciences.

Deletion of the Cl-/HCO3- exchanger pendrin downregulates calcium-absorbing proteins in the kidney and causes calcium wasting.

M. Soleimani, H. Amlal, Sharon Barone, 2012, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

Slc26a11, a chloride transporter, localizes with the vacuolar H(+)-ATPase of A-intercalated cells of the kidney.

M. Soleimani, Sharon Barone, Hong C. Li, 2011, Kidney international.

Deletion of the anion exchanger Slc26a4 (pendrin) decreases apical Cl(-)/HCO3(-) exchanger activity and impairs bicarbonate secretion in kidney collecting duct.

S. Petrovic, Xuming Sun, M. Soleimani, 2010, American journal of physiology. Cell physiology.

Deletion of the chloride transporter Slc26a9 causes loss of tubulovesicles in parietal cells and impairs acid secretion in the stomach

S. Alper, P. Song, G. Shull, 2008, Proceedings of the National Academy of Sciences.

Sodium and chloride absorptive defects in the small intestine in Slc26a6 null mice

M. Manns, M. Wiemann, U. Seidler, 2007, Pflügers Archiv - European Journal of Physiology.

Slc26a6 (PAT1) Deletion Downregulates the Apical Na+/H+ Exchanger in the Straight Segment of the Proximal Tubule

A. McDonough, S. Petrovic, M. Soleimani, 2007, American Journal of Nephrology.

SLC 26 A 9 is expressed in gastric surface epithelial cells , mediates Cl / HCO 3 exchange , and is inhibited by NH 4

D. Witte, G. Shull, J. G. Forte, 2005 .

SLC26A9 is expressed in gastric surface epithelial cells, mediates Cl-/HCO3- exchange, and is inhibited by NH4+.

D. Witte, G. Shull, J. G. Forte, 2005, American journal of physiology. Cell physiology.

Renal and intestinal transport defects in Slc26a6-null mice.

J. Lorenz, P. Aronson, Tong Wang, 2005, American journal of physiology. Cell physiology.

Kidney and Intestine Transport Defects in Slc 26 a 6 Null Mice

J. Lorenz, P. Aronson, Tong Wang, 2004 .

Differential regulation of basolateral Cl-/HCO3- exchangers SLC26A7 and AE1 in kidney outer medullary collecting duct.

J. Kere, S. Petrovic, M. Soleimani, 2004, Journal of the American Society of Nephrology : JASN.

A Basis of Enhanced Fluid and Electrolyte Reabsorption By Formate in the Kidney Proximal Tubule

A. Weinstein, S. Petrovic, M. Soleimani, 2004 .

Activation of the apical Na+/H+ exchanger NHE3 by formate: a basis of enhanced fluid and electrolyte reabsorption by formate in the kidney.

A. Weinstein, S. Petrovic, M. Soleimani, 2004, American journal of physiology. Renal physiology.

Regulation of the apical Cl-/HCO-3 exchanger pendrin in rat cortical collecting duct in metabolic acidosis.

S. Petrovic, M. Soleimani, Zhaohui Wang, 2003, American journal of physiology. Renal physiology.

Identification of an apical \batchmode \documentclass[fleqn,10pt,legalpaper]{article} \usepackage{amssymb} \usepackage{amsfonts} \usepackage{amsmath} \pagestyle{empty} \begin{document} \(\mathrm{Cl}^{-}{/}\mathrm{HCO}_{3}^{-}\) \end{document} exchanger in rat kidney proximal tubule

S. Petrovic, M. Soleimani, Zhaohui Wang, 2003 .

Identification of an apical Cl-/HCO3- exchanger in gastric surface mucous and duodenal villus cells.

G. Shull, P. Dudeja, K. Ramaswamy, 2003, American journal of physiology. Gastrointestinal and liver physiology.

Identification of an apical Cl-/HCO-3 exchanger in rat kidney proximal tubule.

S. Petrovic, M. Soleimani, Zhaohui Wang, 2003, American journal of physiology. Cell physiology.

Identification of a basolateral Cl−/HCO 3 − exchanger specific to gastric parietal cells

J. Kere, S. Alper, H. Lohi, 2003 .

Identification of a basolateral Cl-/HCO3- exchanger specific to gastric parietal cells.

J. Kere, S. Alper, H. Lohi, 2003, American journal of physiology. Gastrointestinal and liver physiology.

Colocalization of the apical Cl-/HCO3- exchanger PAT1 and gastric H-K-ATPase in stomach parietal cells.

G. Shull, J. G. Forte, U. Seidler, 2002, American journal of physiology. Gastrointestinal and liver physiology.

Identification of an apical Cl(-)/HCO3(-) exchanger in the small intestine.

E. Mann, S. Petrovic, M. Soleimani, 2002, American journal of physiology. Gastrointestinal and liver physiology.

special communication Colocalization of the apical Cl/HCO 3 exchanger PAT1 and gastric H-K-ATPase in stomach parietal cells

G. Shull, J. G. Forte, U. Seidler, 2002 .

Pendrin: an apical Cl-/OH-/HCO3- exchanger in the kidney cortex.

P. Kopp, S. Petrovic, P. Kopp, 2001, American journal of physiology. Renal physiology.

Impaired pancreatic ductal bicarbonate secretion in cystic fibrosis.

M. Soleimani, Manoocher Soleimani, 2001, JOP : Journal of the pancreas.

Molecular physiology of the renal chloride-formate exchanger

M. Soleimani, 2001, Current opinion in nephrology and hypertension.

Downregulated in adenoma and putative anion transporter are regulated by CFTR in cultured pancreatic duct cells.

M. Soleimani, C. Schweinfest, Zhaohui Wang, 2001, American journal of physiology. Gastrointestinal and liver physiology.

Renal salt wasting in mice lacking NHE3 Na+/H+ exchanger but not in mice lacking NHE2.

J. Lorenz, G. Shull, P. Schultheis, 2001, American journal of physiology. Renal physiology.

Urine Ammonium, Metabolic Acidosis and Progression of Chronic Kidney Disease

N. Pourafshar, M. Soleimani, Shirin Pourafshar, 2017, Nephron.

Short-term exposure to a high-protein diet differentially affects glomerular filtration rate but not Acid-base balance in older compared to younger adults.

G. Falciglia, M. Soleimani, H. Amlal, 2007, Journal of the American Dietetic Association.

Regulation of the basolateral chloride/base exchangers AE1 and SLC26A7 in the kidney collecting duct in potassium depletion.

J. Kere, A. Blanchard, F. Karet, 2007, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

The non-diuretic hypotensive effects of thiazides are enhanced during volume depletion states

Jack Rubinstein, J. Lorenz, Min Jiang, 2017, PloS one.

Distinct and sequential upregulation of genes regulating cell growth and cell cycle progression during hepatic ischemia-reperfusion injury.

A. Lentsch, R. Casero, S. Petrovic, 2005, American journal of physiology. Cell physiology.

Homozygous loss-of-function mutations in SLC26A7 cause goitrous congenital hypothyroidism.

C. Anderson, M. Ojaniemi, J. Kero, 2018, JCI insight.

Activation of H+-ATPase by hypotonicity: a novel regulatory mechanism for H+ secretion in IMCD cells.

M. Soleimani, H. Amlal, A. Goel, 1998, American journal of physiology. Renal physiology.

Activation of H+-ATPase by hypotonicity: a novel regulatory mechanism for H+ secretion in IMCD cells.

M. Soleimani, H. Amlal, A. Goel, 1998, The American journal of physiology.

ZIP8, Member of the Solute-Carrier-39 (SLC39) Metal-Transporter Family: Characterization of Transporter Properties

D. Nebert, Lei He, T. Dalton, 2006, Molecular Pharmacology.

Use of the pH-sensitive dye BCECF to study pH regulation in cultured human kidney proximal tubule cells

A. Evan, J. Mcateer, M. Soleimani, 1991 .

Impaired mucus-bicarbonate barrier in Helicobacter pylori-infected mice.

L. Engstrand, M. Phillipson, L. Holm, 2006, American journal of physiology. Gastrointestinal and liver physiology.

Regulation of Acid-Base Transporters by Vasopressin in the Kidney Collecting Duct of Brattleboro Rat

S. Petrovic, S. Sheriff, M. Soleimani, 2006, American Journal of Nephrology.

Ablation of polyamine catabolic enzymes provokes Purkinje cell damage, neuroinflammation, and severe ataxia

J. Cleveland, N. Rahmati, D. Lindquist, 2020, Journal of neuroinflammation.

List of papers

G. Shull, J. G. Forte, M. Phillipson, 2005 .

Kidney intercalated cells and the transcription factor FOXi1 drive cystogenesis in tuberous sclerosis complex

E. Henske, J. Bissler, E. Zhang, 2021, Proceedings of the National Academy of Sciences.

Identification of IQGAP1 as a SLC26A4 (Pendrin)-Binding Protein in the Kidney

L. Holliday, M. Soleimani, Sharon Barone, 2022, Frontiers in Molecular Biosciences.

Modulation of polyamine metabolic flux in adipose tissue alters the accumulation of body fat by affecting glucose homeostasis

S. Merali, C. Porter, Chunli Liu, 2013, Amino Acids.

Slc2a5 (Glut5) Is Essential for the Absorption of Fructose in the Intestine and Generation of Fructose-induced Hypertension*

Jian Zuo, U. Seidler, S. L. Fussell, 2009, Journal of Biological Chemistry.

Molecular and functional characterization of sodium--hydrogen exchanger in skin as well as cultured keratinocytes and melanocytes.

R. Boissy, R. Sarangarajan, M. Soleimani, 2001, Biochimica et biophysica acta.

Glucocorticoids enhance the expression of the basolateral Na+:HCO3- cotransporter in renal proximal tubules.

M. Soleimani, H. Amlal, C. Burnham, 2000, Kidney international.

[Formula: see text] reabsorption in renal collecting duct of NHE-3-deficient mouse: a compensatory response.

G. Shull, P. Schultheis, M. Soleimani, 1999, American journal of physiology. Renal physiology.

HCO-3 reabsorption in renal collecting duct of NHE-3-deficient mouse: a compensatory response.

G. Shull, S. Nakamura, P. Schultheis, 1999, The American journal of physiology.

Cloning, renal distribution, and regulation of the rat Na+-[Formula: see text]cotransporter.

G. Shull, M. Flagella, M. Soleimani, 1998, American journal of physiology. Renal physiology.

NHE-1 is the sodium-hydrogen exchanger isoform present in erythroid cells.

R. Sarangarajan, C. Joiner, M. Soleimani, 1998, Biochimica et biophysica acta.

Functional Characterization of a Cloned Human Kidney Na+:HCO3 − Cotransporter*

M. Soleimani, H. Amlal, C. Burnham, 1998, The Journal of Biological Chemistry.

Cloning , renal distribution , and regulation of the rat Na 1-HCO 3 2 cotransporter

G. Shull, M. Flagella, M. Soleimani, 1998 .

Glycosylation of the Na+/H+ exchanger isoform NHE-3 is species specific.

E. Chang, M. Rao, M. Soleimani, 1996, The Journal of laboratory and clinical medicine.

The effect of lethal acid stress on Na+/H+ exchanger isoforms in cultured inner medullary collecting duct cells: deletion of NHE-2 and over expression of NHE-1.

J. Mcateer, M. Soleimani, G. Singh, 1995, Biochimica et biophysica acta.

Localization of the Na+/H+ exchanger isoform NHE-3 in rabbit and canine kidney.

E. Chang, M. Musch, M. Rao, 1994, Biochimica et biophysica acta.

Studies of renal injury IV: The GLUT1 gene protects renal cells from cyclosporine A toxicity.

J. Dominguez, M. Soleimani, T. Batiuk, 2002, Kidney international.

Pathways for HCO3-reabsorption in mouse medullary collecting duct segments.

S. Nakamura, M. Soleimani, H. Amlal, 2000, The Journal of laboratory and clinical medicine.

Differential expression of ileal Na(+)/H(+) exchanger isoforms after enterectomy.

R. Falcone, B. Warner, C. Erwin, 1999, The Journal of surgical research.

K+ depletion increases HCO3- reabsorption in OMCD by activation of colonic H(+)-K(+)-ATPase.

S. Nakamura, M. Soleimani, J. Galla, 1998, The American journal of physiology.

Colonic H+-K+-ATPase is induced and mediates increased HCO3- reabsorption in inner medullary collecting duct in potassium depletion.

S. Nakamura, M. Soleimani, H. Amlal, 1998, Kidney international.

depletion increases HCO 3 (cid:50) reabsorption in OMCD by activation of colonic H (cid:49) -K (cid:49) -ATPase

M. Soleimani, Suguru Nakamura, J. Galla, 1998 .

A possible molecular basis of natriuresis during ischemic-reperfusion injury in the kidney.

H. Rabb, G. Shull, M. Soleimani, 1998, Journal of the American Society of Nephrology : JASN.

Ischemic-reperfusion injury in the kidney: overexpression of colonic H+-K+-ATPase and suppression of NHE-3.

H. Rabb, G. Shull, T. Craig, 1997, Kidney international.

Functional upregulation of H+-ATPase by lethal acid stress in cultured inner medullary collecting duct cells.

M. Soleimani, H. Amlal, Zhaohui Wang, 1997, American journal of physiology. Cell physiology.

Inhibition of glycosylation decreases Na+/H+ exchange activity, blocks NHE-3 transport to the membrane, and increases NHE-3 mRNA expression in LLC-PK1 cells.

J. Dominguez, E. Chang, M. Rao, 1996, The Journal of laboratory and clinical medicine.

Long-term high osmolality activates Na(+)-H+ exchange and protein kinase C in aortic smooth muscle cells.

J. Dominguez, M. Soleimani, G. Singh, 1995, Circulation research.

SLC26A7 Constitutes the SCN--selective Anion Conductance of the Basolateral Membrane of the Retinal Pigment Epithelium.

B. Hughes, M. Soleimani, Xu Cao, 2020, American journal of physiology. Cell physiology.

SLC26A7 constitutes the thiocyanate-selective anion conductance of the basolateral membrane of the retinal pigment epithelium

M. Soleimani, Xu Cao, X. B. A. Hughes, 2020 .

Ischemic and non-ischemic acute kidney injury cause hepatic damage.

R. Schuster, A. Lentsch, M. Hedayati, 2009, Kidney international.

THE Slc 2 a 5 ( Glut 5 ) IS ESSENTIAL FOR THE ABSORPTION OF FRUCTOSE IN THE INTESTINE AND GENERATION OF FRUCTOSE-INDUCED HYPERTENSION

Jian Zuo, U. Seidler, S. L. Fussell, 2008 .

Vasopressin induces expression of the Cl-/HCO3- exchanger SLC26A7 in kidney medullary collecting ducts of Brattleboro rats.

F. Karet, S. Petrovic, Xuming Sun, 2006, American journal of physiology. Renal physiology.

Vasopressin-independent regulation of collecting duct aquaporin-2 in food deprivation.

S. Sheriff, M. Soleimani, H. Amlal, 2005, Kidney international.

Identification of stathmin as a novel marker of cell proliferation in the recovery phase of acute ischemic renal failure.

H. Rabb, N. Yokota, S. Petrovic, 2004, American journal of physiology. Cell physiology.

Overexpression of SSAT in kidney cells recapitulates various phenotypic aspects of kidney ischemia-reperfusion injury.

H. Rabb, K. Matlin, R. Casero, 2004, Journal of the American Society of Nephrology : JASN.

Novel Schering and ouabain-insensitive potassium-dependent proton secretion in the mouse cortical collecting duct.

G. Shull, S. Petrovic, Z. Spicer, 2002, American journal of physiology. Renal physiology.

Coordinated down-regulation of NBC-1 and NHE-3 in sodium and bicarbonate loading.

L. Pavelić, M. Soleimani, H. Amlal, 2001, Kidney international.

Fasting downregulates renal water channel AQP2 and causes polyuria.

M. Soleimani, H. Amlal, Z. Wang, 2001, American journal of physiology. Renal physiology.

CFTR drives Na+-[Formula: see text]cotransport in pancreatic duct cells: a basis for defective[Formula: see text] secretion in CF.

R. Frizzell, C. Ulrich, M. Soleimani, 1999, American journal of physiology. Cell physiology.

CFTR upregulates the expression of the basolateral Na(+)-K(+)-2Cl(-) cotransporter in cultured pancreatic duct cells.

M. Soleimani, H. Shumaker, 1999, The American journal of physiology.

Characterization of Na+/[Formula: see text]cotransporter isoform NBC-3.

M. Soleimani, H. Amlal, C. Burnham, 1999, American journal of physiology. Renal physiology.

CFTR drives Na+-cotransport in pancreatic duct cells: a basis for defective secretion in CF

R. Frizzell, C. Ulrich, M. Soleimani, 1999 .

Potassium depletion downregulates chloride-absorbing transporters in rat kidney.

M. Soleimani, H. Amlal, Zhaohui Wang, 1998, The Journal of clinical investigation.

Spermidine/spermine N1-acetyltransferase overexpression in kidney epithelial cells disrupts polyamine homeostasis, leads to DNA damage, and causes G2 arrest.

P. Diegelman, C. Porter, N. Kisiel, 2007, American journal of physiology. Cell physiology.

Spermidine/spermine N1-acetyltransferase overexpression in kidney epithelial cells disrupts polyamine homeostasis, leads to DNA damage, and causes G2 arrest.

P. Diegelman, C. Porter, N. Kisiel, 2007, American journal of physiology. Cell physiology.

Insulin resistance and hypertension: new insights.

M. Soleimani, 2015, Kidney international.

Spermidine/spermine N1-acetyltransferase-mediated polyamine catabolism regulates beige adipocyte biogenesis.

Yuan Fang, Wei Gao, Can Zhao, 2018, Metabolism: clinical and experimental.

Slc26a3 deficiency is associated with loss of colonic HCO3− secretion, absence of a firm mucus layer and barrier impairment in mice

W. Xia, M. Montrose, M. Johansson, 2014, Acta physiologica.

The Role of Salt in the Pathogenesis of Fructose-Induced Hypertension

M. Soleimani, Pooneh Alborzi, 2011, International journal of nephrology.

cAMP-dependent and cholinergic regulation of the electrogenic intestinal / pancreatic Na + / HCO 3-cotransporter pNBC 1 in human embryonic kidney ( HEK 293 ) cells

M. Manns, O. Bachmann, U. Seidler, 2015 .

Leukocyte Involvement in Renal Reperfusion-Induced Liver Damage

B. Seifi, H. Sadeghipour, J. Hadjati, 2011, Renal failure.

Liver Oxidative Stress after Renal Ischemia-Reperfusion Injury is Leukocyte Dependent in Inbred Mice

B. Seifi, H. Sadeghipour, J. Hadjati, 2011, Iranian journal of basic medical sciences.

Effects of different periods of renal ischemia on liver as a remote organ.

M. Hedayati, S. Ostad, M. Kadkhodaee, 2009, World journal of gastroenterology.

Slc4a8 in the Kidney: Expression, Subcellular Localization and Role in Salt Reabsorption

M. Soleimani, Sharon Barone, K. Zahedi, 2018, Cellular Physiology and Biochemistry.

Na+:HCO3- cotransporters (NBC): expression and regulation in the kidney.

M. Soleimani, 2002, Journal of nephrology.

Characterization of Na+/HCO-3 cotransporter isoform NBC-3.

M. Soleimani, H. Amlal, C. Burnham, 1999, The American journal of physiology.

Effect of Renal Tubule-Specific Knockdown of the Na+/H+ Exchanger NHE3 in Akita Diabetic Mice.

Yiling Fu, V. Vallon, K. Sharma, 2019, American journal of physiology. Renal physiology.

ANG II controls Na+-K+([Formula: see text])-2Cl-cotransport via 20-HETE and PKC in medullary thick ascending limb.

M. Paillard, M. Soleimani, H. Amlal, 1998, American journal of physiology. Cell physiology.

Polyamine Catabolism in Acute Kidney Injury

M. Soleimani, Sharon Barone, K. Zahedi, 2019, International journal of molecular sciences.

The chloride channel/transporter Slc26a9 regulates the systemic arterial pressure and renal chloride excretion

M. Soleimani, H. Amlal, Sharon Barone, 2012, Journal of Molecular Medicine.

Interaction of Membrane Skeletal Protein, Protein 4.1B and p55, and Sodium Bicarbonate Cotransporter1 in Mouse Renal S1-S2 Proximal Tubules

M. Komada, G. Seki, Tatsuo Suzuki, 2007, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

CaSR signaling down‐regulates AQP2 expression via a novel microRNA pathway in pendrin and NaCl cotransporter knockout mice

Annarita Di Mise, G. Tamma, M. Ranieri, 2017, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Oral Cadmium in Mice Carrying 5 Versus 2 Copies of the Slc39a8 Gene

Bin Wang, D. Nebert, Zhiwei Liu, 2014, International journal of toxicology.

Epithelial Anion Transporter Pendrin Contributes to Inflammatory Lung Pathology in Mouse Models of Bordetella pertussis Infection

N. Carbonetti, S. Wall, M. Soleimani, 2014, Infection and Immunity.

The switch of intestinal Slc26 exchangers from anion absorptive to HCOFormula secretory mode is dependent on CFTR anion channel function.

O. Nylander, U. Seidler, M. Soleimani, 2010, American journal of physiology. Cell physiology.

Mechanism of Thiazide Diuretic Arterial Pressure Reduction: The Search Continues

R. Rapoport, M. Soleimani, 2019, Front. Pharmacol..

ANG II controls Na+-K+([Formula: see text])-2Cl-cotransport via 20-HETE and PKC in medullary thick ascending limb.

M. Paillard, M. Soleimani, H. Amlal, 1998, American journal of physiology. Cell physiology.

H+-K+-ATPases: regulation and role in pathophysiological states.

R. Silver, M. Soleimani, 1999, American journal of physiology. Renal physiology.

AFLUID June 45/6

R. Silver, M. Soleimani, 1999 .

H+-K+-ATPases: regulation and role in pathophysiological states.

R. Silver, M. Soleimani, 1999, The American journal of physiology.

Proximal Tubule Epithelial Cell Specific Ablation of the Spermidine/Spermine N1-Acetyltransferase Gene Reduces the Severity of Renal Ischemia/Reperfusion Injury

T. Murray-Stewart, R. Casero, M. Soleimani, 2014, PloS one.

Immunolocalization of phospho-Arg-directed protein kinase-substrate in hypoxic kidneys using in vivo cryotechnique

S. Saitoh, Y. Saitoh, N. Ohno, 2009, Medical Molecular Morphology.

Loss of Slc26a9 anion transporter alters intestinal electrolyte and HCO3- transport and reduces survival in CFTR-deficient mice

U. Seidler, H. Lenzen, Taolang Li, 2014, Pflügers Archiv - European Journal of Physiology.

Absence of Slc26a9 results in altered tracheobronchial anion transport and high mortality in neonate mice (1181.7)

P. Anagnostopoulou, U. Seidler, M. Mall, 2014 .

Hepatocyte-specific ablation of spermine/spermidine-N1-acetyltransferase gene reduces the severity of CCl4-induced acute liver injury.

R. Schuster, A. Lentsch, R. Casero, 2012, American journal of physiology. Gastrointestinal and liver physiology.

Hepatocyte-speciﬁc ablation of spermine/spermidine- N 1 -acetyltransferase gene reduces the severity of CCl 4 -induced acute liver injury

R. Schuster, A. Lentsch, R. Casero, 2012 .

Spermidine/spermine-N1-acetyltransferase ablation protects against liver and kidney ischemia-reperfusion injury in mice.

L. Alhonen, D. Witte, A. Lentsch, 2009, American journal of physiology. Gastrointestinal and liver physiology.

Spermidine / spermine-N 1-acetyltransferase ablation protects against liver and kidney ischemia-reperfusion injury in mice

L. Alhonen, D. Witte, A. Lentsch, 2009 .

Expression of SSAT, a novel biomarker of tubular cell damage, increases in kidney ischemia-reperfusion injury.

H. Rabb, C. Porter, R. Casero, 2003, American journal of physiology. Renal physiology.

Identification of a Novel Biomarker of Tubular Cell Injury in IRI By Suppression Subtractive Hybridization: SSAT Expression Increases in Renal Ischemia Reperfusion Injury

H. Rabb, C. Porter, R. Casero, 2003 .

Chloride/bicarbonate exchanger SLC26A7 is localized in endosomes in medullary collecting duct cells and is targeted to the basolateral membrane in hypertonicity and potassium depletion.

S. Petrovic, R. Worrell, M. Soleimani, 2006, Journal of the American Society of Nephrology : JASN.

ZIP14 and ZIP8 zinc/bicarbonate symporters in Xenopus oocytes: characterization of metal uptake and inhibition.

D. Nebert, Marina Gálvez-Peralta, Lei He, 2012, Metallomics : integrated biometal science.

Missense mutations in Na+:HCO3- cotransporter NBC1 show abnormal trafficking in polarized kidney cells: a basis of proximal renal tubular acidosis.

J. Matthews, L. Conforti, R. Worrell, 2005, American journal of physiology. Renal physiology.

Stoichiometry of Na+-HCO-3 cotransport in basolateral membrane vesicles isolated from rabbit renal cortex.

P. Aronson, M. Soleimani, S. M. Grassi, 1987, The Journal of clinical investigation.

Potassium depletion increases luminal Na+/H+ exchange and basolateral Na+:CO3=:HCO3- cotransport in rat renal cortex.

M. Soleimani, T. Mckinney, M. Hosford, 1990, The Journal of clinical investigation.

Cloning and Functional Expression of a Human Kidney Na+:HCO3 −Cotransporter*

G. Shull, M. Soleimani, H. Amlal, 1997, The Journal of Biological Chemistry.

Effects of acetazolamide on Na+-HCO-3 cotransport in basolateral membrane vesicles isolated from rabbit renal cortex.

P. Aronson, M. Soleimani, 1989, The Journal of clinical investigation.

Prostaglandin-E2 Mediated Increase in Calcium and Phosphate Excretion in a Mouse Model of Distal Nephron Salt Wasting

F. McCormack, M. Soleimani, Sharon Barone, 2016, PloS one.

Carbonic anhydrase II does not regulate nitrite‐dependent nitric oxide formation and vasodilation

M. Gladwin, D. Kim‐Shapiro, Qinzi Xu, 2020, British journal of pharmacology.

Properties of the renal Na(+)-HCO3- cotransporter.

P. Aronson, S. Grassl, M. Soleimani, 1991, Seminars in nephrology.

How cystic fibrosis affects pancreatic ductal bicarbonate secretion.

C. Ulrich, M. Soleimani, 2000, The Medical clinics of North America.

Putative anion transporter-1 (Pat-1, Slc26a6) contributes to intracellular pH regulation during H+-dipeptide transport in duodenal villous epithelium.

C. Supuran, L. Clarke, M. Soleimani, 2010, American journal of physiology. Gastrointestinal and liver physiology.

CFTR drives Na+-[Formula: see text]cotransport in pancreatic duct cells: a basis for defective[Formula: see text] secretion in CF.

R. Frizzell, C. Ulrich, M. Soleimani, 1999, American journal of physiology. Cell physiology.

A pH modifier site regulates activity of the Na+:HCO3- cotransporter in basolateral membranes of kidney proximal tubules.

M. Soleimani, T. Mckinney, J. A. Bergman, 1991, The Journal of clinical investigation.

Probenecid Pre-treatment Downregulates the Kidney Cl-/HCO3- Exchanger (Pendrin) and Potentiates Hydrochlorothiazide-Induced Diuresis

M. Soleimani, Sharon Barone, K. Zahedi, 2018, Front. Physiol..

Potentiation of the Effect of Thiazide Derivatives by Carbonic Anhydrase Inhibitors: Molecular Mechanisms and Potential Clinical Implications

M. Soleimani, Sharon Barone, K. Zahedi, 2013, PloS one.

Generation and analyses of R8L barttin knockin mouse.

T. Okado, S. Sasaki, S. Bachmann, 2011, American journal of physiology. Renal physiology.

Decreased Expression of Slc26a4 (Pendrin) and Slc26a7 in the Kidneys of Carbonic Anhydrase II-Deficient Mice

S. Petrovic, Xuming Sun, M. Soleimani, 2008, Cellular Physiology and Biochemistry.

The Role of Epithelial Sodium Channel ENaC and the Apical Cl-/HCO3- Exchanger Pendrin in Compensatory Salt Reabsorption in the Setting of Na-Cl Cotransporter (NCC) Inactivation

M. Soleimani, Sharon Barone, Mina Patel-Chamberlin, 2016, PloS one.

Pendrin as a Novel Target for Diuretic Therapy

M. Soleimani, H. Amlal, 2011, Cellular Physiology and Biochemistry.

Vascular contractile reactivity in hypotension due to reduced renal reabsorption of Na+ and restricted dietary Na+

R. Rapoport, M. Soleimani, Saeed Alshahrani, 2017, Naunyn-Schmiedeberg's Archives of Pharmacology.

Pendrin: an apical Cl 2 /OH 2 /HCO 3 2 exchanger in the kidney cortex

S. Petrovic, P. Kopp, M. Soleimani, 2001 .

Differential Regulation of Basolateral Cl (cid:1) /HCO 3 (cid:1) Exchangers SLC26A7 and AE1 in Kidney Outer Medullary Collecting Duct

J. Kere, S. Petrovic, M. Soleimani, 2004 .

Spermidine/spermine-N¹-acetyltransferase in kidney ischemia reperfusion injury.

M. Soleimani, K. Zahedi, 2011, Methods in molecular biology.

Stathmin-deficient mice develop fibrosis and show delayed recovery from ischemic-reperfusion injury.

H. Rabb, J. Bissler, D. Citron, 2006, American journal of physiology. Renal physiology.

Renal Transcriptome and Metabolome in Mice with Principal Cell-Specific Ablation of the Tsc1 Gene: Derangements in Pathways Associated with Cell Metabolism, Growth and Acid Secretion

R. Casero, M. Soleimani, Sharon Barone, 2022, International journal of molecular sciences.

Thiazide Therapy in Chronic Kidney Disease: Renal and Extra Renal Targets.

Ashkan Karimi, N. Pourafshar, M. Soleimani, 2018, Current drug metabolism.

Slc39a14 Gene Encodes ZIP14, A Metal/Bicarbonate Symporter: Similarities to the ZIP8 Transporter

D. Nebert, Zhiwei Liu, Lei He, 2008, Molecular Pharmacology.

Cd2+ versus Zn2+ uptake by the ZIP8 HCO3--dependent symporter: kinetics, electrogenicity and trafficking.

D. Nebert, Zhiwei Liu, Lei He, 2008, Biochemical and biophysical research communications.

Potassium-Depletion Metabolic Alkalosis

M. Soleimani, 2005 .

Identification of the electrogenic 2Cl-/H+ exchanger, ClC5, as a chloride secreting transporter candidate in kidney cyst epithelium in tuberous sclerosis.

J. Bissler, L. Holliday, M. Soleimani, 2022, The American journal of pathology.

Molecular transport machinery involved in orchestrating luminal acid‐induced duodenal bicarbonate secretion in vivo

Ashutosh Kumar Singh, B. K. Thakur, U. Seidler, 2013, The Journal of physiology.

Functional activity of Pat‐1 (Slc26a6) Cl−/HCO3− exchange in the lower villus epithelium of murine duodenum

E. E. Hoover, L. Clarke, Jennifer M. Brazill, 2011, Acta physiologica.

Ileal oxalate absorption and urinary oxalate excretion are enhanced in Slc26a6 null mice.

Mike L Green, M. Soleimani, M. Hatch, 2006, American journal of physiology. Gastrointestinal and liver physiology.

Involvement of the anion exchanger SLC26A6 in prostaglandin E2- but not forskolin-stimulated duodenal HCO3- secretion.

W. Colledge, U. Seidler, M. Soleimani, 2006, Gastroenterology.

Polyamines and Their Metabolism: From the Maintenance of Physiological Homeostasis to the Mediation of Disease

M. Soleimani, Sharon Barone, K. Zahedi, 2022, Medical sciences.

K+/NH4+ antiporter: a unique ammonium carrying transporter in the kidney inner medulla.

M. Soleimani, H. Amlal, 1997, Biochimica et biophysica acta.

Activation of endoplasmic reticulum stress response by enhanced polyamine catabolism is important in the mediation of cisplatin-induced acute kidney injury

Mark A. Hall, J. Cleveland, R. Casero, 2017, PloS one.

mode is dependent on CFTR anion channel function 2

O. Nylander, U. Seidler, M. Soleimani, 2010 .

Slc39a14 Gene Encodes ZIP14, A Metal/Bicarbonate Symporter: Similarities to the ZIP8 Transporter

D. Nebert, Zhiwei Liu, Lei He, 2008, Molecular Pharmacology.

Slc26a11 is prominently expressed in the brain and functions as a chloride channel: expression in Purkinje cells and stimulation of V H+-ATPase

C. I. Zeeuw, N. Rahmati, K. Kunzelmann, 2013, Pflügers Archiv - European Journal of Physiology.

Deletion of Slc26a6 alters the stoichiometry of apical Cl-/HCO-3 exchange in mouse pancreatic duct.

H. Ishiguro, M. Steward, A. Yamamoto, 2012, American journal of physiology. Cell physiology.

Presence of chloride-formate exchange in vascular smooth muscle and cardiac cells.

M. Soleimani, R. Howard, 1994, Circulation research.

Receptor Protein Tyrosine Phosphatase γ, CO2 Sensing in Proximal Tubule and Acid Base Homeostasis.

M. Soleimani, 2016, Journal of the American Society of Nephrology : JASN.

Downregulation of renal AQP2 water channel and NKCC2 in mice lacking the apical Na+‐H+ exchanger NHE3

G. Shull, S. Sheriff, M. Soleimani, 2003, Journal of Physiology.

Hepatic changes during various periods of reperfusion after induction of renal ischemia in rats.

B. Seifi, M. Kadkhodaee, M. Soleimani, 2009, Transplantation proceedings.

Modulation of polyamine metabolic flux in adipose tissue alters the accumulation of body fat by affecting glucose homeostasis

S. Merali, C. Porter, Chunli Liu, 2014, Amino Acids.

Lack of thiazide diuretic inhibition of agonist constriction of mouse mesenteric arterioles ex vivo

A. LeBlanc, R. Rapoport, Jason E Beare, 2018, Naunyn-Schmiedeberg's Archives of Pharmacology.

Male susceptibility to hepatic damage in acute uremia in rats.

M. Kadkhodaee, M. Soleimani, F. Golab, 2011, Urology.

Mouse Na+: HCO3- cotransporter isoform NBC-3 (kNBC-3): cloning, expression, and renal distribution.

L. Conforti, S. Petrovic, M. Soleimani, 2001, Kidney international.

Identification of a novel signal in the cytoplasmic tail of the Na+:HCO3- cotransporter NBC1 that mediates basolateral targeting.

L. Conforti, M. Soleimani, Hong C. Li, 2007, American journal of physiology. Renal physiology.

Identification of a carboxyl-terminal motif essential for the targeting of Na+-HCO-3 cotransporter NBC1 to the basolateral membrane.

J. Matthews, L. Conforti, H. Husseinzadeh, 2004, The Journal of biological chemistry.

Na+:HCO3− Cotransporters (NBC): Cloning and Characterization

M. Soleimani, C. Burnham, 2001, The Journal of Membrane Biology.

Role of PDZK1 in membrane expression of renal brush border ion exchangers.

P. Aronson, Tong Wang, B. Thomson, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Identification of a Carboxyl-terminal Motif Essential for the Targeting of Na+-\batchmode \documentclass[fleqn,10pt,legalpaper]{article} \usepackage{amssymb} \usepackage{amsfonts} \usepackage{amsmath} \pagestyle{empty} \begin{document} \(\mathrm{HC}\mathrm{O}_{3}^{-}\) \end{document} Cotransporter N

J. Matthews, L. Conforti, H. Husseinzadeh, 2004, Journal of Biological Chemistry.

M1724 The Apical Chloride/Base Exchanger Slc26a6 (Pat1) and the Fructose-Absorbing Transporter Slc2a5 (Glut5) Play An Essential Role in the Pathogenesis of Fructose-Induced Hypertension

U. Seidler, Zi Zuo, M. Soleimani, 2008 .

Expression, regulation and the role of SLC26 Cl-/HCO3- exchangers in kidney and gastrointestinal tract.

M. Soleimani, 2006, Novartis Foundation symposium.

Concomitant Lung and Kidney Disorders in Critically Ill Patients: Core Curriculum 2022

M. Soleimani, U. Brewster, O. Ann, 2022 .

Pro-inflammatory cytokines of rat vasculature in DOCA-salt treatment

B. Seifi, M. Kadkhodaee, M. Soleimani, 2010, Molecular Biology Reports.

Physiologic and molecular aspects of the Na+:HCO3- cotransporter in health and disease processes.

M. Soleimani, C. Burnham, 2000, Kidney international.

Renal and intestinal absorptive defects in mice lacking the NHE3 Na +/H+ exchanger

G. Giebisch, T. Doetschman, P. Meneton, 1998, Nature Genetics.

SLC26A Gene Family Participate in pH Regulation during Enamel Maturation

M. Snead, M. Paine, R. Lacruz, 2015, PloS one.

Deletion of the Chloride Transporter Slc26a7 Causes Distal Renal Tubular Acidosis and Impairs Gastric Acid Secretion*

S. Alper, P. Song, Marian L Miller, 2009, The Journal of Biological Chemistry.

Upregulation of collecting duct aquaporin-2 by metabolic acidosis: role of vasopressin.

S. Sheriff, M. Soleimani, H. Amlal, 2004, American journal of physiology. Cell physiology.

Proximal Tubule–Specific Deletion of the NHE3 (Na+/H+ Exchanger 3) Promotes the Pressure-Natriuresis Response and Lowers Blood Pressure in Mice

X. Li, J. Zhuo, M. Tauc, 2018, Hypertension.

Isolation and cDNA Cloning of Ksp-cadherin, a Novel Kidney-specific Member of the Cadherin Multigene Family (*)

P. Igarashi, P. Aronson, A. Abu-Alfa, 1995, The Journal of Biological Chemistry.

Deletion of Slc26a1 and Slc26a7 Delays Enamel Mineralization in Mice

M. Paine, Wenting Lin, M. Soleimani, 2017, Front. Physiol..

Kidney Injury in COVID-19: Epidemiology, Molecular Mechanisms and Potential Therapeutic Targets

J. P. Teixeira, M. Soleimani, K. Zahedi, 2022, International journal of molecular sciences.

Effect of high osmolality on Na+/H+ exchange in renal proximal tubule cells.

J. Mcateer, E. Chang, M. Musch, 1994, The Journal of biological chemistry.

Apical ammonium inhibition of cAMP-stimulated secretion in T84 cells is bicarbonate dependent.

J. Matthews, R. Worrell, M. Soleimani, 2005, American journal of physiology. Gastrointestinal and liver physiology.

Regulation of aquaporin-2 by metabolic acidosis

S. Sheriff, M. Soleimani, 2004 .

The apical anion exchanger Slc26a6 promotes oxalate secretion by murine submandibular gland acinar cells

J. E. Melvin, G. Shull, C. Masaki, 2018, The Journal of Biological Chemistry.

PAT-1 (Slc26a6) is the predominant apical membrane Cl-/HCO3- exchanger in the upper villous epithelium of the murine duodenum.

G. Shull, L. Gawenis, L. Clarke, 2007, American journal of physiology. Gastrointestinal and liver physiology.

Effect of Slc26a6 deletion on apical Cl-/HCO3- exchanger activity and cAMP-stimulated bicarbonate secretion in pancreatic duct.

Min Goo Lee, H. Ishiguro, A. Yamamoto, 2007, American journal of physiology. Gastrointestinal and liver physiology.

Effect of in vitro metabolic acidosis on luminal Na+/H+ exchange and basolateral Na+:HCO3- cotransport in rabbit kidney proximal tubules.

M. Soleimani, G. L. Bizal, T. Mckinney, 1992, The Journal of clinical investigation.

CFTR upregulates the expression of the basolateral Na+-K+-2Cl-cotransporter in cultured pancreatic duct cells.

M. Soleimani, H. Shumaker, 1999, American journal of physiology. Cell physiology.

Metabolic modeling of single Th17 cells reveals regulators of autoimmunity

A. Regev, N. Yosef, Allon Wagner, 2021, Cell.

slc26a3 (dra)-deficient Mice Display Chloride-losing Diarrhea, Enhanced Colonic Proliferation, and Distinct Up-regulation of Ion Transporters in the Colon*

D. Spyropoulos, R. Worrell, M. Soleimani, 2006, Journal of Biological Chemistry.

Loss of Slc26a9 anion transporter alters intestinal electrolyte and HCO3- transport and reduces survival in CFTR-deficient mice

U. Seidler, H. Lenzen, Taolang Li, 2014, Pflügers Archiv - European Journal of Physiology.

Murine thyroid IL-4 expression worsens hypothyroidism under iodine restriction and mitigates Graves' disease development.

J. Dumont, F. Miot, M. Soleimani, 2022, Endocrinology.

Transient Expression of Na+/H+ Exchanger Isoform NHE-2 in LLC-PK1 Cells: Inhibition of Endogenous NHE-3 and Regulation by Hypertonicity

J. Orlowski, M. Soleimani, G. Singh, 1996, The Journal of Membrane Biology.

Enhanced cadmium-induced testicular necrosis and renal proximal tubule damage caused by gene-dose increase in a Slc39a8-transgenic mouse line.

D. Nebert, Nadine Dragin, T. Dalton, 2007, American journal of physiology. Cell physiology.

Tuberous sclerosis complex exhibits a new renal cystogenic mechanism

B. Siroky, J. Bissler, D. Bridges, 2019, Physiological reports.

Caffeine-induced diuresis and natriuresis is independent of renal tubular NHE3.

M. Busslinger, R. Fenton, T. Rieg, 2015, American journal of physiology. Renal physiology.

Wood muscle transport across the sarcolemma of trout white Carbonic anhydrase facilitates CO 2 and NH 3

J. Matthews, J. Xu, M. Soleimani, 1999 .

Erratum to: The distinct roles of anion transporters Slc26a3 (DRA) and Slc26a6 (PAT-1) in fluid and electrolyte absorption in the murine small intestine

Ashutosh Kumar Singh, P. Song, W. Xia, 2014, Pflügers Archiv - European Journal of Physiology.

CFTR induces the expression of DRA along with Cl(-)/HCO(3)(-) exchange activity in tracheal epithelial cells.

J. Yankaskas, G. Shull, M. Soleimani, 2000, American journal of physiology. Cell physiology.

Differential regulation of Na+/H+ exchange and H+ -ATPase by pH and HCO3− in kidney proximal tubules

E. Chang, M. Rao, M. Soleimani, 1995, The Journal of Membrane Biology.

Evaluation of Renal Oxidative Stress in the Development of DOCA-Salt Induced Hypertension and Its Renal Damage

B. Seifi, M. Kadkhodaee, M. Soleimani, 2010, Clinical and experimental hypertension.

cAMP potentiates HCO3− uptake via an electrogenically-driven Na+:3HCO3− cotransporter only in pancreatic duct epithelial cells expressing functional CFTR

E. Kopras, C. Ulrich, M. Soleimani, 1998 .

Identification of thrombospondin 1 (TSP-1) as a novel mediator of cell injury in kidney ischemia.

H. Rabb, C. Thakar, A. Lentsch, 2005, The Journal of clinical investigation.

Early polyuria and urinary concentrating defect in potassium deprivation.

Carissa M Krane, C. Krane, M. Soleimani, 2000, American journal of physiology. Renal physiology.

Ionic mechanism of Na+-HCO3- cotransport in rabbit renal basolateral membrane vesicles.

P. Aronson, M. Soleimani, 1989, The Journal of biological chemistry.

The role of spermidine/spermine N1-acetyltransferase in endotoxin-induced acute kidney injury.

L. Alhonen, D. Kramer, C. Porter, 2010, American journal of physiology. Cell physiology.

Characterization of Na+/cotransporter isoform NBC-3

M. Soleimani, H. Amlal, C. Burnham, 1999 .

Potassium deprivation upregulates expression of renal basolateral Na(+)-HCO(3)(-) cotransporter (NBC-1).

M. Soleimani, H. Amlal, K. Habo, 2000, American journal of physiology. Renal physiology.

Expression of the Na+-HCO-3 cotransporter NBC4 in rat kidney and characterization of a novel NBC4 variant.

M. Petrović, L. Conforti, S. Petrovic, 2003, American journal of physiology. Renal physiology.

Cloning, renal distribution, and regulation of the rat Na+-HCO3- cotransporter.

G. Shull, M. Flagella, M. Soleimani, 1998, The American journal of physiology.

kidney and characterization of a novel NBC4 variant -HCO cotransporter NBC4 in rat + Expression of the Na

M. Petrović, L. Conforti, S. Petrovic, 2016 .

Potassium depletion and acid-base transporters in rat kidney: differential effect of hypophysectomy.

M. Soleimani, Z. Wang, H. Shumaker, 1997, American Journal of Physiology.

Physiologic and molecular aspects of the Na+/H+ exchangers in health and disease processes.

M. Soleimani, G. Singh, 1995, Journal of investigative medicine : the official publication of the American Federation for Clinical Research.

Renal tubular NHE3 is required in the maintenance of water and sodium chloride homeostasis.

R. Fenton, T. Rieg, S. Poulsen, 2017, Kidney international.

Functional identity of a purified proximal tubule anion exchanger protein: mediation of chloride/formate and chloride/bicarbonate exchange.

M. Soleimani, G. L. Bizal, 1996, Kidney international.

A protein with anion exchange properties found in the kidney proximal tubule.

M. Soleimani, C. Anderson, G. L. Bizal, 1993, Kidney international.

Identification and covalent modification of a renal brush-border anion exchanger.

M. Soleimani, G. L. Bizal, Y. J. Hattabaugh, 1993, Biochimica et biophysica acta.

Metabolic Alkalosis Pathogenesis, Diagnosis, and Treatment: Core Curriculum 2022.

M. Soleimani, C. Do, Pamela Vásquez, 2022, American journal of kidney diseases : the official journal of the National Kidney Foundation.

Downregulation of the Cl-/HCO3-Exchanger Pendrin in Kidneys of Mice with Cystic Fibrosis: Role in the Pathogenesis of Metabolic Alkalosis

A. McDonough, M. Soleimani, K. Zahedi, 2018, Cellular Physiology and Biochemistry.

Pathophysiology of Renal Tubular Acidosis: Core Curriculum 2016.

M. Soleimani, A. Rastegar, 2016, American journal of kidney diseases : the official journal of the National Kidney Foundation.

Acute Regulation of Na + :HCO 3 - Cotransport System In Kidney Proximal Tubules

P. Aronson, M. Soleimani, G. L. Bizal, 1994 .

CFTR cotransport in pancreatic duct cells: a basis for defective HCO 3 (cid:50) secretion in CF

R. Frizzell, C. Ulrich, M. Soleimani, 1998 .

Slc26a7 Chloride Channel Activity and Localization in Mouse Reissner’s Membrane Epithelium

Daniel C. Marcus, Jie Xu, Philine Wangemann, 2014, PloS one.

New Insights into the Critical Importance of Intratubular Na+/H+ Exchanger 3 and Its Potential Therapeutic Implications in Hypertension

X. Li, J. Zhuo, M. Soleimani, 2021, Current Hypertension Reports.

Acute Kidney Injury in SARS-CoV-2 Infection: Direct Effect of Virus on Kidney Proximal Tubule Cells

M. Soleimani, 2020, International journal of molecular sciences.

Segmental differences in Slc26a3-dependent Cl− absorption and HCO3− secretion in the mouse large intestine in vitro in Ussing chambers

H. Nagai, M. Soleimani, H. Hayashi, 2021, The Journal of Physiological Sciences.

Pathogenesis of Hypertension in Metabolic Syndrome: The Role of Fructose and Salt

M. Soleimani, K. Zahedi, Sharon L. Barone, 2023, International journal of molecular sciences.

Na/K‐ATPase signaling tonically inhibits sodium reabsorption in the renal proximal tubule

M. Romero, S. Haller, S. Pierre, 2023, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Proximal tubule specific knockout of the Na+/H+ exchanger NHE3: effects on bicarbonate absorption and ammonium excretion

Tong Wang, A. McDonough, M. Tauc, 2013, Journal of Molecular Medicine.

SLC26A11 (KBAT) in Purkinje Cells Is Critical for Inhibitory Transmission and Contributes to Locomotor Coordination

Freek E. Hoebeek, Martijn Schonewille, Chris I. De Zeeuw, 2016, eNeuro.

Estrogen downregulates the proximal tubule type IIa sodium phosphate cotransporter causing phosphate wasting and hypophosphatemia.

M. Levi, M. Soleimani, H. Amlal, 2008, Kidney international.

Damage to the gastric epithelium activates cellular bicarbonate secretion via SLC26A9 Cl(-)/HCO(3)(-).

M. Montrose, M. Soleimani, E. Demitrack, 2010, American journal of physiology. Gastrointestinal and liver physiology.

Damage to the gastric epithelium activates cellular bicarbonate secretion via SLC26A9 Cl (cid:1) /HCO 3 (cid:1) exchange

M. Montrose, M. Soleimani, E. Demitrack, 2010 .

SLC26A9-mediated chloride secretion prevents mucus obstruction in airway inflammation.

Thomas Thum, Michael Kabesch, M. Kabesch, 2012, The Journal of clinical investigation.

ACELL December 46/6

M. Soleimani, H. Shumaker, 1999 .

Overexpression of miR-223 Tips the Balance of Pro- and Anti-hypertrophic Signaling Cascades toward Physiologic Cardiac Hypertrophy*

Xiaohong Wang, Yutian Li, Wei Huang, 2016, The Journal of Biological Chemistry.

Possible molecular basis for changes in potassium handling in acute renal failure.

H. Rabb, M. Soleimani, G. Postler, 2000, American journal of kidney diseases : the official journal of the National Kidney Foundation.

The chloride channel/transporter Slc26a9 regulates the systemic arterial pressure and renal chloride excretion

M. Soleimani, H. Amlal, K. Zahedi, 2013, Journal of Molecular Medicine.

The distinct roles of anion transporters Slc26a3 (DRA) and Slc26a6 (PAT-1) in fluid and electrolyte absorption in the murine small intestine

P. Song, W. Xia, U. Seidler, 2013, Pflügers Archiv - European Journal of Physiology.

Cation specificity and modes of the Na+:CO3(2-):HCO3- cotransporter in renal basolateral membrane vesicles.

P. Aronson, M. Soleimani, J. A. Bergman, 1991, The Journal of biological chemistry.

The switch of intestinal Slc26 exchangers from anion absorptive to HCO 3 (cid:1) secretory mode is dependent on CFTR anion channel function

O. Nylander, U. Seidler, M. Soleimani, 2010 .

The multiple roles of pendrin in the kidney.

M. Soleimani, 2015, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

SLC26A9-mediated chloride secretion prevents mucus obstruction in airway inflammation.

M. Kabesch, T. Thum, P. Anagnostopoulou, 2012, The Journal of clinical investigation.