M. Laorden

发表

The PKs PKA and ERK 1/2 are involved in phosphorylation of TH at Serine 40 and 31 during morphine withdrawal in rat hearts

P. Almela, M. Milanés, M. Laorden, 2008, British journal of pharmacology.

Protein kinase C phosphorylates the cAMP response element binding protein in the hypothalamic paraventricular nucleus during morphine withdrawal

M. Laorden, M. Milanés, M. Laorden, 2011, British journal of pharmacology.

Morphine withdrawal regulates phosphorylation of cAMP response element binding protein (CREB) through PKC in the nucleus tractus solitarius‐A2 catecholaminergic neurons

M. Laorden, M. Milanés, M. Laorden, 2009, Journal of neurochemistry.

Activation of stress‐related hypothalamic neuropeptide gene expression during morphine withdrawal

K. Kovács, M. Milanés, M. Laorden, 2007, Journal of neurochemistry.

Morphine administration modulates expression of Argonaute 2 and dopamine‐related transcription factors involved in midbrain dopaminergic neurons function

M. Milanés, M. Laorden, C. Núñez, 2013, British journal of pharmacology.

Glucocorticoid receptor but not mineralocorticoid receptor mediates the activation of ERK pathway and CREB during morphine withdrawal

J. Navarro-Zaragoza, M. Milanés, M. Laorden, 2017, Addiction biology.

Involvement of 3′,5′‐cyclic adenosine monophosphate‐dependent protein kinase in regulation of Fos expression and tyrosine hydroxylase levels during morphine withdrawal in the hypothalamic paraventricular nucleus and medulla oblongata catecholaminergic cell groups

M. Milanés, M. Laorden, M. Benavides, 2005, Journal of neurochemistry.

Regulation of tyrosine hydroxylase levels and activity and Fos expression during opioid withdrawal in the hypothalamic PVN and medulla oblongata catecholaminergic cell groups innervating the PVN

J. García-Borrón, M. Milanés, M. Laorden, 2003, The European journal of neuroscience.

Activation of c‐fos expression in the heart after morphine but not U‐50,488H withdrawal

A. González-Cuello, M. Milanés, M. Laorden, 2003, British journal of pharmacology.

Morphine withdrawal‐induced c‐fos expression in the hypothalamic paraventricular nucleus is dependent on the activation of catecholaminergic neurones

P. Almela, M. Milanés, M. Laorden, 2002, Journal of neurochemistry.

Effects of U‐50,488H withdrawal on catecholaminergic neurones of the rat ventricle

M. Laorden, M. Milanés, M. Laorden, 1998, British journal of pharmacology.

The involvement of CRF1 receptor within the basolateral amygdala and dentate gyrus in the naloxone-induced conditioned place aversion in morphine-dependent mice

P. Almela, M. Milanés, M. Laorden, 2018, Progress in Neuro-Psychopharmacology and Biological Psychiatry.

CRF2 mediates the increased noradrenergic activity in the hypothalamic paraventricular nucleus and the negative state of morphine withdrawal in rats

O. Valverde, J. Navarro-Zaragoza, M. Laorden, 2011, British journal of pharmacology.

Activation of the ERK signalling pathway contributes to the adaptive changes in rat hearts during naloxone‐induced morphine withdrawal

M. Laorden, P. Almela, M. Milanés, 2007, British journal of pharmacology.

Regulation of dopaminergic markers expression in response to acute and chronic morphine and to morphine withdrawal

M. Milanés, M. Laorden, C. Núñez, 2016, Addiction biology.

Corticotropin-releasing factor 1 receptor mediates the activity of the reward system evoked by morphine-induced conditioned place preference

M. Milanés, M. Laorden, C. Núñez, 2015, Neuropharmacology.

Regulation of extracellular signal-regulated kinases (ERKs) by naloxone-induced morphine withdrawal in the brain stress system

M. Laorden, M. Milanés, M. Laorden, 2008, Naunyn-Schmiedeberg's Archives of Pharmacology.

Glucocorticoid receptors participate in the opiate withdrawal‐induced stimulation of rats NTS noradrenergic activity and in the somatic signs of morphine withdrawal

J. Navarro-Zaragoza, M. Milanés, M. Laorden, 2012, British journal of pharmacology.

Elevated glucocorticoid levels are responsible for induction of tyrosine hydroxylase mRNA expression, phosphorylation, and enzyme activity in the nucleus of the solitary tract during morphine withdrawal.

K. Kovács, J. García-Borrón, M. Laorden, 2009, Endocrinology.

Quantitative Analysis of Corticotropin‐Releasing Factor and Arginine Vasopressin mRNA in the Hypothalamus During Chronic Morphine Treatment in Rats: An In Situ Hybridization Study

A. Burlet, M. Laorden, M. Milanés, 2003, Journal of neuroendocrinology.

Different contribution of glucocorticoids in the basolateral amygdala to the formation and expression of opiate withdrawal-associated memories

K. Kovács, S. Ferenczi, M. Laorden, 2016, Psychoneuroendocrinology.

Effects of chronic inflammation and morphine tolerance on the expression of phospho-ERK 1/2 and phospho-P38 in the injured tissue

P. García-Nogales, M. Laorden, M. Puig, 2009, Naunyn-Schmiedeberg's Archives of Pharmacology.

Inhibition of protein kinase C but not protein kinase A attenuates morphine withdrawal excitation of rat hypothalamus-pituitary-adrenal axis.

M. Laorden, M. Milanés, M. Laorden, 2002, European journal of pharmacology.

Effects of Corticotropin-Releasing Factor Receptor-1 Antagonists on the Brain Stress System Responses to Morphine Withdrawal

J. Navarro-Zaragoza, M. Laorden, M. Laorden, 2010, Molecular Pharmacology.