Ajinkya Kumbhare

IL-10 induces an immune repressor pathway in sepsis by promoting S100A9 nuclear localization and MDSC development.

C. McCall, M. El Gazzar, Isatou Bah, 2018, Cellular immunology.

Nfia deletion in myeloid cells blocks expansion of myeloid-derived suppressor cells during sepsis

C. McCall, J. Dai, Z. Yao, 2017, Innate immunity.

S100A9 maintains myeloid-derived suppressor cells in chronic sepsis by inducing miR-21 and miR-181b.

C. McCall, M. El Gazzar, Z. Yao, 2019, Molecular immunology.

Intracellular S100A9 Promotes Myeloid-Derived Suppressor Cells during Late Sepsis

C. McCall, M. El Gazzar, J. Dai, 2017, Front. Immunol..

Long Non-Coding RNA Hotairm1 Promotes S100A9 Support of MDSC Expansion during Sepsis

C. McCall, Z. Yao, Isatou Bah, 2020, Journal of clinical & cellular immunology.

HuR promotes miRNA-mediated upregulation of NFI-A protein expression in MDSCs during murine sepsis.

C. McCall, M. El Gazzar, Z. Yao, 2020, Molecular immunology.

Expression of C/EBP&bgr; in myeloid progenitors during sepsis promotes immunosuppression

C. McCall, M. El Gazzar, J. Dai, 2017, Molecular immunology.

Corrigendum to "IL-10 induces an immune repressor pathway in sepsis by promoting S100A9 nuclear localization and MDSC development" [Cell Immunol. 332 (2018) 32-38].

C. McCall, M. El Gazzar, Isatou Bah, 2020, Cellular immunology.