Octavia M. Peck

Differential regulation of cytokine and chemokine production in lipopolysaccharide-induced tolerance and priming.

J. Kalbfleisch, David L. Williams, G. Teti, 2004, Cytokine.

TOLL-LIKE RECEPTOR 4 COUPLED GI PROTEIN SIGNALING PATHWAYS REGULATE EXTRACELLULAR SIGNAL-REGULATED KINASE PHOSPHORYLATION AND AP-1 ACTIVATION INDEPENDENT OF NFκB ACTIVATION

P. Halushka, J. Cook, G. Tempel, 2004 .

Lipopolysaccharide- and gram-positive bacteria-induced cellular inflammatory responses: role of heterotrimeric Galpha(i) proteins.

L. Birnbaumer, B. Zingarelli, G. Teti, 2005, American journal of physiology. Cell physiology.

THE PHOSPHATIDYLINOSITOL 3 KINASE PATHWAY REGULATES TOLERANCE TO LIPOPOLYSACCHARIDE AND PRIMING RESPONSES TO STAPHYLOCOCCUS AUREUS AND LIPOPOLYSACCHARIDE

B. Zingarelli, G. Teti, P. Halushka, 2006, Shock.

Staphylococcus aureus and Lipopolysaccharide Induce Homologous Tolerance but Heterologous Priming: Role of Interferon-&ggr;

G. Teti, P. Halushka, J. Cook, 2004, Shock.

Toll-like receptor 4 coupled GI protein signaling pathways regulate extracellular signal-regulated kinase phosphorylation and AP-1 activation independent of NFkappaB activation.

P. Halushka, J. Cook, G. Tempel, 2004, Shock.