Qihang Zhang

发表

Increased cerebral oxygen consumption in Eker rats and effects of N‐methyl‐D‐aspartate blockade: Implications for autism

H. Weiss, O. Chi, Xia Liu, 2007, Journal of neuroscience research.

Cyclic GMP Reduces Myocardial Stunning Through Non-Cyclic GMP Protein Kinase Mechanisms

H. Weiss, Lin Yan, Qihang Zhang, 2004, Journal of cardiovascular pharmacology.

Hypoxia Inducible Factor-1 Improves the Actions of Nitric Oxide and Natriuretic Peptides after Simulated Ischemia-Reperfusion

H. Weiss, Jason A. Luciano, E. Huang, 2008, Cellular Physiology and Biochemistry.

Ethanol Reduces Cardiac Myocyte Function through Activation of the Nitric Oxide-Cyclic GMP Pathway

H. Weiss, Y. Vaks, Qihang Zhang, 2003, Pharmacology.

SERCA Inhibition Limits the Functional Effects of Cyclic GMP in Both Control and Hypertrophic Cardiac Myocytes

H. Weiss, Qihang Zhang, P. Scholz, 2009, Pharmacology.

Effects of natriuretic peptides on ventricular myocyte contraction and role of cyclic GMP signaling.

H. Weiss, J. Moalem, Qihang Zhang, 2005, European journal of pharmacology.

dog ventricular myocytes with hypertrophic failure Reduction in interaction between cGMP and cAMP in

H. Weiss, Qihang Zhang, P. Scholz, 2016 .

Reduction in interaction between cGMP and cAMP in dog ventricular myocytes with hypertrophic failure.

H. Weiss, Qihang Zhang, P. Scholz, 2005, American journal of physiology. Heart and circulatory physiology.

Brain Natriuretic Peptide Reverses the Effects of Myocardial Stunning in Rabbit Myocardium

H. Weiss, Qihang Zhang, P. Scholz, 2006, Pharmacology.

Cyclic GMP signaling and regulation of SERCA activity during cardiac myocyte contraction.

H. Weiss, Qihang Zhang, P. Scholz, 2005, Cell calcium.

Cyclic GMP-Induced Reduction in Cardiac Myocyte Function Is Partially Mediated by Activation of the Sarcoplasmic Reticulum Ca2+-ATPase

H. Weiss, Lin Yan, Qihang Zhang, 2002, Pharmacology.

Functional effects of C-type natriuretic peptide and nitric oxide are attenuated in hypertrophic myocytes from pressure-overloaded mouse hearts.

H. Weiss, J. Moalem, Qihang Zhang, 2005, American journal of physiology. Heart and circulatory physiology.

T4-induced cardiac hypertrophy disrupts cyclic GMP mediated responses to brain natriuretic peptide in rabbit myocardium

H. Weiss, Qihang Zhang, P. Scholz, 2006, Peptides.

Negative inotropic effects of C-type natriuretic peptide are attenuated in hypertrophied ventricular myocytes associated with reduced cyclic GMP production.

H. Weiss, G. Grover, J. Moalem, 2006, The Journal of surgical research.

Fluorescence Properties of Dibenzofluorescein in Aqueous Solution

Fushi Zhang, Qiang Liu, Fuqun Zhao, 2008, Journal of Fluorescence.

Photophysical properties of dibenzofluorescein and the presence of its tautomers or prototropic forms in organic solvents

Fushi Zhang, Qiang Liu, Fuqun Zhao, 2008, Photochemical & photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology.

Chronic nitrates blunt the effects of not only nitric oxide but also natriuretic peptides in cardiac myocytes.

H. Weiss, Qihang Zhang, P. Scholz, 2007, Pharmacological research.

Negative Functional Effects of Natriuretic Peptides Are Attenuated in Hypertrophic Cardiac Myocytes by Reduced Particulate Guanylyl Cyclase Activity

H. Weiss, K. Khurana, Qihang Zhang, 2007, Journal of cardiovascular pharmacology.

Synthesis of CsH and its effect on the hydrogen storage properties of the Mg(NH2)2-2LiH system

Lixian Sun, Jiaxun Zhang, H. Pan, 2016 .

Role of Phospholamban in Cyclic GMP Mediated Signaling in Cardiac Myocytes

H. Weiss, Qihang Zhang, P. Scholz, 2006, Cellular Physiology and Biochemistry.

Importance of ryanodine receptors in effects of cyclic GMP is reduced in thyroxine-induced cardiac hypertrophy.

H. Weiss, Qihang Zhang, P. Scholz, 2006, European journal of pharmacology.

Cyclic GMP protein kinase activity is reduced in thyroxine‐induced hypertrophic cardiac myocytes

H. Weiss, Lin Yan, Qihang Zhang, 2003, Clinical and experimental pharmacology & physiology.